Reactive oxygen species mediate alpha-adrenergic receptor-stimulated hypertrophy in adult rat ventricular myocytes.
about
Monoamine oxidases (MAO) in the pathogenesis of heart failure and ischemia/reperfusion injuryThioredoxin and ventricular remodelingNovel Perspectives in Redox Biology and Pathophysiology of Failing Myocytes: Modulation of the Intramyocardial Redox Milieu for Therapeutic Interventions-A Review Article from the Working Group of Cardiac Cell Biology, Italian Society of CardiologyGRK2 in the heart: a GPCR kinase and beyondRegulation of signal transduction by reactive oxygen species in the cardiovascular systemCardiac myocyte gene expression profiling during H2O2-induced apoptosisMonoamine oxidase A-mediated enhanced catabolism of norepinephrine contributes to adverse remodeling and pump failure in hearts with pressure overload.Association between plasma homocysteine concentration and the risk of all-cause death in adults with diastolic dysfunction in a community: A 13-year cohort studyOxidant stress from nitric oxide synthase-3 uncoupling stimulates cardiac pathologic remodeling from chronic pressure load.Regulation and function of selenoproteins in human diseasePrimary carnitine deficiency and sudden death: in vivo evidence of myocardial lipid peroxidation and sulfonylation of sarcoendoplasmic reticulum calcium ATPase 2.Reactive oxygen species are involved in regulating alpha1-adrenoceptor-activated vascular smooth muscle contraction.Inhibition of endogenous thioredoxin in the heart increases oxidative stress and cardiac hypertrophy.The severe cardiorenal syndrome: 'Guyton revisited'.Pathophysiological mechanisms of catecholamine and cocaine-mediated cardiotoxicity.Foetal nicotine exposure causes PKCĪµ gene repression by promoter methylation in rat hearts.The cardiorenal syndrome: a reviewAlpha1-adrenergic receptors prevent a maladaptive cardiac response to pressure overload.Role of reactive oxygen species in hyperadrenergic hypertension: biochemical, physiological, and pharmacological evidence from targeted ablation of the chromogranin a (Chga) geneAdiponectin mediates cardioprotection in oxidative stress-induced cardiac myocyte remodeling.Cardiac hypertrophy is positively regulated by MicroRNA miR-23a.NADPH oxidase-derived reactive oxygen species in cardiac pathophysiologyRedox regulation of Ras and Rho GTPases: mechanism and function.Pivotal role of reactive oxygen species in differential regulation of lipopolysaccharide-induced prostaglandins production in macrophagesProlonged administration of a dithiol antioxidant protects against ventricular remodeling due to ischemia-reperfusion in mice.Identification of the Mtus1 Splice Variant as a Novel Inhibitory Factor Against Cardiac Hypertrophy.Cardiomyocyte-secreted acetylcholine is required for maintenance of homeostasis in the heartNongenomic effects of estrogen mediate the dose-related myocardial oxidative stress and dysfunction caused by acute ethanol in female rats.Hydrogen Sulfide Inhibits High-Salt Diet-Induced Myocardial Oxidative Stress and Myocardial Hypertrophy in Dahl Rats.Icariin attenuates angiotensin II-induced hypertrophy and apoptosis in H9c2 cardiomyocytes by inhibiting reactive oxygen species-dependent JNK and p38 pathways.Functional linkage as a direction for studies in oxidative stress: alpha-adrenergic receptors.Role of autocrine/paracrine mechanisms in response to myocardial strain.Pathogenesis of chronic cardiorenal syndrome: is there a role for oxidative stress?Comparative efficacy and the window of radioprotection for adrenergic and serotoninergic agents and aminothiols in experiments with small and large animals.Cardiac Hypertrophy: An Introduction to Molecular and Cellular Basis.Mouse models for the study of postnatal cardiac hypertrophy.The role of SUMO-1 in cardiac oxidative stress and hypertrophy.Regulation of angiotensin II-stimulated osteopontin expression in cardiac microvascular endothelial cells: role of p42/44 mitogen-activated protein kinase and reactive oxygen species.Sustained exposure to catecholamines affects cAMP/PKA compartmentalised signalling in adult rat ventricular myocytes.Direct enzymatic reduction of lucigenin decreases lucigenin-amplified chemiluminescence produced by superoxide ion.
P2860
Q24632770-B4C3E3B8-3FFA-4535-8206-AE5B0BFCFE51Q24683258-8AF7B7BF-96C2-4603-890B-D386266C5663Q26765793-7B4DF30A-6E60-496F-A12C-66990E08E15DQ26851548-9A7D33D1-7D9C-4556-ADE6-46771EAC2B85Q28391569-80775148-607F-4AFF-ACBE-719997E61E5DQ28568243-87E48A44-C9AA-4667-9607-A53BD45085F0Q33580858-74B39553-C265-4F1F-8E3F-8AD5475E9817Q33627168-C8DC3CAA-33FA-464C-9DD1-DB06BA770FE7Q33743649-879895DF-35EE-4CEC-A23D-D6F31163F75FQ34029071-35E26430-AAEB-42B0-9EA1-FDF3E93C50FFQ34083532-B41B1F88-E719-4931-98E3-AFB58BB641BEQ34097787-BE2322DE-E82A-4DD1-A7B8-7E37AC64F073Q34274405-661BEE6B-310E-4DE0-AB4D-9BFAB523EE5AQ34379161-09C146D7-82AF-4730-9CCD-7A9BA9F94002Q34396286-21D4DE43-9E15-4F72-A364-8455D83D6924Q34410283-5DC55E39-F752-4673-B9CD-08C4D006AD71Q34495307-37BEC4C8-EB7A-4FF8-BC0D-1A4E08223831Q34498911-8533EF5B-8C5E-47A9-B7AE-60311F38C3EBQ34642358-85B9CD1F-E231-4132-82C5-A0F01653E8D7Q35315318-39EAE298-B5E1-4D73-805D-2EA1339239A6Q35643799-BB4A0F21-7BDE-478C-85DB-7DD518E3E3EAQ36328374-193C797D-B296-4BE7-AB0D-D12BAC1F00F0Q36456556-848D8A90-82F3-4751-9E71-C4E1F1F33FE0Q36497800-9F8C9C3A-4793-4118-8E3F-19532C8A86E3Q36893405-BBFCAAF3-4176-4582-8EA9-228CF34AE064Q37240712-86562F6C-B174-497D-96E0-8F2934E3F9D3Q37327512-4B241197-F26E-45DC-92E1-EE9219385D14Q37656005-13DFE202-1B79-4252-8D83-CA66162A9094Q37703080-E3BDB5BA-73A8-45C4-B490-263481DF7306Q37710080-B630F073-4A52-48AD-B79D-B99C2C82D0EBQ37731337-21D3C76B-1C47-472F-9F97-20FA32227161Q37837614-BC264371-C153-4740-BC19-C961A303EEEFQ38165049-9217C4AF-2108-4F59-9103-C0F0FF2DC4E0Q38259871-C7840F17-A919-4ED8-ABA9-A0F5147506CBQ38906321-76E70593-A6D4-485A-ACE6-607A150030B1Q40963953-E4C08AD0-3599-4979-A95A-BDBDA73B0D13Q41961959-54C2E0D0-14B9-4796-BB45-3A4B2809EE5AQ42505155-66314314-5D25-4B14-A7A8-483B63FE1B1BQ42777336-9BBFB21A-9BB8-4CF5-9F3B-F0149E980EA1Q43759403-142B8479-532D-4C76-9431-06831E28755E
P2860
Reactive oxygen species mediate alpha-adrenergic receptor-stimulated hypertrophy in adult rat ventricular myocytes.
description
2001 nĆ® lÅ«n-bĆ»n
@nan
2001 Õ©ÕøÖÕ”ÕÆÕ”Õ¶Õ« Õ
ÕøÖÕ¶ÕøÖÕ”ÖÕ«Õ¶ Õ°ÖÕ”ÕæÕ”ÖÕ”ÕÆÕøÖÕ”Õ® Õ£Õ«ÕæÕ”ÕÆÕ”Õ¶ ÕµÖ
Õ¤ÕøÖÕ”Õ®
@hyw
2001 Õ©Õ¾Õ”ÕÆÕ”Õ¶Õ« Õ°ÕøÖÕ¶Õ¾Õ”ÖÕ«Õ¶ Õ°ÖÕ”ÕæÕ”ÖÕ”ÕÆÕ¾Õ”Õ® Õ£Õ«ÕæÕ”ÕÆÕ”Õ¶ Õ°ÕøÕ¤Õ¾Õ”Õ®
@hy
2001幓ć®č«ę
@ja
2001幓č«ę
@yue
2001幓č«ę
@zh-hant
2001幓č«ę
@zh-hk
2001幓č«ę
@zh-mo
2001幓č«ę
@zh-tw
2001幓č®ŗę
@wuu
name
Reactive oxygen species mediat ...... dult rat ventricular myocytes.
@ast
Reactive oxygen species mediat ...... dult rat ventricular myocytes.
@en
type
label
Reactive oxygen species mediat ...... dult rat ventricular myocytes.
@ast
Reactive oxygen species mediat ...... dult rat ventricular myocytes.
@en
prefLabel
Reactive oxygen species mediat ...... dult rat ventricular myocytes.
@ast
Reactive oxygen species mediat ...... dult rat ventricular myocytes.
@en
P2093
P356
P1476
Reactive oxygen species mediat ...... dult rat ventricular myocytes.
@en
P2093
Colucci WS
Pimental DR
P304
P356
10.1006/JMCC.2000.1285
P577
2001-01-01T00:00:00Z