A model of atypical absence seizures: EEG, pharmacology, and developmental characterization.
about
Mutation I810N in the alpha3 isoform of Na+,K+-ATPase causes impairments in the sodium pump and hyperexcitability in the CNS.Pathogenesis and new candidate treatments for infantile spasms and early life epileptic encephalopathies: A view from preclinical studiesGABA receptor proteins within lipid rafts in the AY-9944 model of atypical absence seizuresSeizures and epilepsy: an overview for neuroscientistsTypical versus atypical absence seizures: network mechanisms of the spread of paroxysms.Cellular and network mechanisms of genetically-determined absence seizures.Gamma-hydroxybutyric acid: neurobiology and toxicology of a recreational drug.Clinical and neurophysiologic spectrum associated with atypical absence seizures in children with intractable epilepsy.Neurochemical and behavioral features in genetic absence epilepsy and in acutely induced absence seizures.Choosing the correct antiepileptic drugs: from animal studies to the clinic.Gender and age differences in expression of GABAA receptor subunits in rat somatosensory thalamus and cortex in an absence epilepsy modelUpdate on the management of Lennox-Gastaut syndrome with a focus on rufinamide.Spike-wave discharges in adult Sprague-Dawley rats and their implications for animal models of temporal lobe epilepsy.Treatment-resistant Lennox-Gastaut syndrome: therapeutic trends, challenges and future directions.Differential molecular and behavioural alterations in mouse models of GABRG2 haploinsufficiency versus dominant negative mutations associated with human epilepsySystemic availability of guanidinoacetate affects GABAA receptor function and seizure threshold in GAMT deficient mice.Hormonal regulation of atypical absence seizures.Brain sterols in the AY-9944 rat model of atypical absence seizures.Environmental enrichment improves behavioral outcome in the AY-9944 model of childhood atypical absence epilepsy.Lovastatin exacerbates atypical absence seizures with only minimal effects on brain sterols.Monoamine variability in the chronic model of atypical absence seizures.Role of gap junctional coupling in astrocytic networks in the determination of global ischaemia-induced oxidative stress and hippocampal damage.Functional contribution of specific brain areas to absence seizures: role of thalamic gap-junctional coupling.Serotonin depletion attenuates AY-9944-mediated atypical absence seizures.EEG recording in rodents, with a focus on epilepsy.Seizure aggravation by antiepileptic drugs.
P2860
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P2860
A model of atypical absence seizures: EEG, pharmacology, and developmental characterization.
description
2001 nî lūn-bûn
@nan
2001 թուականի Փետրուարին հրատարակուած գիտական յօդուած
@hyw
2001 թվականի փետրվարին հրատարակված գիտական հոդված
@hy
2001年の論文
@ja
2001年論文
@yue
2001年論文
@zh-hant
2001年論文
@zh-hk
2001年論文
@zh-mo
2001年論文
@zh-tw
2001年论文
@wuu
name
A model of atypical absence se ...... evelopmental characterization.
@ast
A model of atypical absence se ...... evelopmental characterization.
@en
type
label
A model of atypical absence se ...... evelopmental characterization.
@ast
A model of atypical absence se ...... evelopmental characterization.
@en
prefLabel
A model of atypical absence se ...... evelopmental characterization.
@ast
A model of atypical absence se ...... evelopmental characterization.
@en
P2093
P356
P1433
P1476
A model of atypical absence se ...... evelopmental characterization.
@en
P2093
McKerlie C
Snead OC 3rd
P304
P356
10.1212/WNL.56.3.341
P407
P577
2001-02-01T00:00:00Z