Angiotensin, inflammation, hypertension, and cardiovascular disease. - Wikidata - awgldk - TriplyDB

Angiotensin, inflammation, hypertension, and cardiovascular disease.

Angiotensin, inflammation, hypertension, and cardiovascular disease.

http://www.wikidata.org/entity/Q31935145

about

Role and therapeutic potential of G-protein coupled receptors in breast cancer progression and metastases Pentosan polysulfate treatment preserves renal autoregulation in ANG II-infused hypertensive rats via normalization of P2X1 receptor activation.Critical appraisal of the differential effects of antihypertensive agents on arterial stiffness.The effect of an Angiotensin receptor blocker on arterial stiffness in type 2 diabetes mellitus patients with hypertension.Comparative effects of angiotensin-converting enzyme inhibition and angiotensin-receptor blockade on inflammation during hemodialysis.Upregulation of junctional adhesion molecule-A is a putative prognostic marker of hypertension Calcium-channel modulators for cardiovascular disease.Mast cell renin and a local renin-angiotensin system in the airway: role in bronchoconstriction Positioning of inflammatory biomarkers in the heart failure landscape.LDL cholesterol angiotensin II interactions in atherosclerosis.Quercetin and Vitamin C Mitigate Cobalt Chloride-Induced Hypertension through Reduction in Oxidative Stress and Nuclear Factor Kappa Beta (NF-Kb) Expression in Experimental Rat Model.Angiotensin-converting enzyme inhibition attenuates myonuclear addition in overloaded slow-twitch skeletal muscle.High-fat diet- and angiotensin II-induced aneurysm concurrently elicits splenic hypertrophy.Angiotensin II-induced Hypertension is Reduced by Deficiency of P-selectin Glycoprotein Ligand-1.Antihypertensive treatment decreases arterial stiffness at night but not during the day. Results from the Hypertension in the Very Elderly Trial.A Brief Introduction into the Renin-Angiotensin-Aldosterone System: New and Old Techniques.

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Angiotensin, inflammation, hypertension, and cardiovascular disease.

http://www.wikidata.org/entity/Q31935145

description

2001 nî lūn-bûn

@nan

2001 թուականի Փետրուարին հրատարակուած գիտական յօդուած

@hyw

2001 թվականի փետրվարին հրատարակված գիտական հոդված

@hy

2001年の論文

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2001年学术文章

@wuu

2001年学术文章

@zh-cn

2001年学术文章

@zh-hans

2001年学术文章

@zh-my

2001年学术文章

@zh-sg

2001年學術文章

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name

Angiotensin, inflammation, hypertension, and cardiovascular disease.

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Angiotensin, inflammation, hypertension, and cardiovascular disease.

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type

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Angiotensin, inflammation, hypertension, and cardiovascular disease.

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Angiotensin, inflammation, hypertension, and cardiovascular disease.

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Angiotensin, inflammation, hypertension, and cardiovascular disease.

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Angiotensin, inflammation, hypertension, and cardiovascular disease.

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S11906-001-0082-Y

P1433

Current Hypertension Reports

P1476

Angiotensin, inflammation, hypertension, and cardiovascular disease

@en

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Luft FC

http://www.w3.org/2001/XMLSchema#string

P2860

Angiotensin II stimulates endothelial vascular cell adhesion molecule-1 via nuclear factor-kappaB activation induced by intracellular oxidative stress

State-of-the-Art lecture. Statins and blockers of the renin-angiotensin system: vascular protection beyond their primary mode of action.

Oxidant signaling in vascular cell growth, death, and survival : a review of the roles of reactive oxygen species in smooth muscle and endothelial cell mitogenic and apoptotic signaling.

Structure and mechanical properties of resistance arteries in hypertension: role of adhesion molecules and extracellular matrix determinants.

Tumor necrosis factor activates angiotensinogen gene expression by the Rel A transactivator.

Theodore Cooper Memorial Lecture. Hypertension and the pathogenesis of atherosclerosis. Oxidative stress and the mediation of arterial inflammatory response: a new perspective.

Angiotensin II (AT(1)) receptor blockade reduces vascular tissue factor in angiotensin II-induced cardiac vasculopathy.

Fluvastatin inhibits matrix metalloproteinase-1 expression in human vascular endothelial cells.

ACE inhibitor quinapril reduces the arterial expression of NF-kappaB-dependent proinflammatory factors but not of collagen I in a rabbit model of atherosclerosis.

High plasma level of N-acetyl-seryl-aspartyl-lysyl-proline: a new marker of chronic angiotensin-converting enzyme inhibition.

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s11906-001-0082-y

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61-67

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scholarly article

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10.1007/S11906-001-0082-Y

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1

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Friedrich C. Luft

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2001-02-01T00:00:00Z

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1026341832

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P921

cardiovascular disease

arterial hypertension