Reduced microvascular thrombosis and improved outcome in acute murine stroke by inhibiting GP IIb/IIIa receptor-mediated platelet aggregation - Wikidata - awgldk - TriplyDB

Reduced microvascular thrombosis and improved outcome in acute murine stroke by inhibiting GP IIb/IIIa receptor-mediated platelet aggregation

Reduced microvascular thrombosis and improved outcome in acute murine stroke by inhibiting GP IIb/IIIa receptor-mediated platelet aggregation

http://www.wikidata.org/entity/Q32000831

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Site-specific phosphorylation of platelet focal adhesion kinase by low-density lipoprotein Dehydroascorbic acid, a blood-brain barrier transportable form of vitamin C, mediates potent cerebroprotection in experimental stroke Facing Time in Ischemic Stroke: An Alternative Hypothesis for Collateral Failure Combining antiplatelet and thrombolytic therapies for stroke Elucidation of the thromboregulatory role of CD39/ectoapyrase in the ischemic brain Targeting coagulation factor XII provides protection from pathological thrombosis in cerebral ischemia without interfering with hemostasis The calcium sensor STIM1 is an essential mediator of arterial thrombosis and ischemic brain infarction.Transient ischemia elicits a sustained enhancement of thrombus development in the cerebral microvasculature: effects of anti-thrombotic therapy.Targeted inhibition of intrinsic coagulation limits cerebral injury in stroke without increasing intracerebral hemorrhage Platelet adhesion receptors do not modulate infarct volume after a photochemically induced stroke in mice.Revisiting cerebral postischemic reperfusion injury: new insights in understanding reperfusion failure, hemorrhage, and edema.Engineered human soluble calcium-activated nucleotidase inhibits coagulation in vitro and thrombosis in vivo.Dissolution of arterial platelet thrombi in vivo with a bifunctional platelet GPIIIa49-66 ligand which specifically targets the platelet thrombus Hemostasis and alterations of the central nervous system Platelet recruitment to the inflamed glomerulus occurs via an alphaIIbbeta3/GPVI-dependent pathway.Neuroprotection by the Traditional Chinese Medicine, Tao-Hong-Si-Wu-Tang, against Middle Cerebral Artery Occlusion-Induced Cerebral Ischemia in Rats.SOD1 nanozyme salvages ischemic brain by locally protecting cerebral vasculature.Early changes of tissue perfusion after tissue plasminogen activator in hyperacute ischemic stroke.Glicoprotein (GP) IIb/IIIa inhibitors for acute stroke treatment.Neutrophil protein kinase Cdelta as a mediator of stroke-reperfusion injury.Safety of tirofiban for patients with acute ischemic stroke in routine clinical practice Venous system in acute brain injury: Mechanisms of pathophysiological change and function Are hemostasis and thrombosis two sides of the same coin?Acute vertebrobasilar occlusion: current treatment strategies.Platelet-derived HMGB1 is a critical mediator of thrombosis.Outcome after systemic thrombolysis is predicted by age and stroke severity: an open label experience with recombinant tissue plasminogen activator and tirofiban.Plasma L5 levels are elevated in ischemic stroke patients and enhance platelet aggregation.Tissue Plasminogen Activator Neurotoxicity is Neutralized by Recombinant ADAMTS 13 The combined approach to lysis utilizing eptifibatide and rt-PA in acute ischemic stroke: the CLEAR stroke trial Plasminogen activators and ischemic stroke: conditions for acute delivery.Virchow's triad: the vascular basis of cerebral injury Inflammation and the neurovascular unit in the setting of focal cerebral ischemia.Postischemic Long-Term Treatment with Qiangli Tianma Duzhong Capsule Improves Brain Functional Recovery via the Improvement of Hemorrheology and the Inhibition of Platelet Aggregation in a Rat Model of Focal Cerebral Ischemia.von Willebrand factor-cleaving protease ADAMTS13 reduces ischemic brain injury in experimental stroke.PEAR1 gene polymorphism in a Chinese pedigree with pulmonary thromboembolism.The neurovascular unit in the setting of stroke.Pericyte-mediated regulation of capillary diameter: a component of neurovascular coupling in health and disease Ischaemic stroke: a thrombo-inflammatory disease?Platelet adhesion and activation mechanisms in arterial thrombosis and ischaemic stroke.Blood coagulation factor XII--a neglected player in stroke pathophysiology.

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P2860

Reduced microvascular thrombosis and improved outcome in acute murine stroke by inhibiting GP IIb/IIIa receptor-mediated platelet aggregation

http://www.wikidata.org/entity/Q32000831

description

1998 nî lūn-bûn

@nan

1998 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած

@hyw

1998 թվականի հոտեմբերին հրատարակված գիտական հոդված

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1998年の論文

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1998年論文

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1998年論文

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1998年論文

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1998年論文

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1998年論文

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1998年论文

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name

Reduced microvascular thrombos ...... -mediated platelet aggregation

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Reduced microvascular thrombos ...... -mediated platelet aggregation

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Reduced microvascular thrombos ...... -mediated platelet aggregation

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Reduced microvascular thrombos ...... -mediated platelet aggregation

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B L Hoh

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C J Prestigiacomo

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D J Pinsky

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G Kottirsch

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H G Zerwes

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S C Kim

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T F Choudhri

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P2860

Antithrombotic effects of a platelet fibrinogen receptor antagonist in a canine model of carotid artery thrombosis

The bleeding time does not predict surgical bleeding.

Analyses of thrombocytopenia in idiopathic thrombocytopenic purpura-prone mice by platelet transfer experiments between (NZW x BXSB)F1 and normal mice.

Downregulation of human platelet reactivity by neutrophils. Participation of lipoxygenase derivatives and adhesive proteins.

Cardiac preservation is enhanced in a heterotopic rat transplant model by supplementing the nitric oxide pathway

Fibrinogen-independent platelet adhesion and thrombus formation on subendothelium mediated by glycoprotein IIb-IIIa complex at high shear rate.

Treatment of ischaemic stroke with prostacyclin.

Transient platelet accumulation in the rat brain after common carotid artery thrombosis. An 111In-labeled platelet study.

Cerebral protection in homozygous null ICAM-1 mice after middle cerebral artery occlusion. Role of neutrophil adhesion in the pathogenesis of stroke.

Exacerbation of cerebral injury in mice that express the P-selectin gene: identification of P-selectin blockade as a new target for the treatment of stroke.

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7

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102

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E Sander Connolly

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9769322

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