about
A Staphylococcus aureus small RNA is required for bacterial virulence and regulates the expression of an immune-evasion moleculeThe Staphylococcus aureus protein Sbi acts as a complement inhibitor and forms a tripartite complex with host complement Factor H and C3bDual RNA regulatory control of a Staphylococcus aureus virulence factorInteraction of human complement with Sbi, a staphylococcal immunoglobulin-binding protein: indications of a novel mechanism of complement evasion by Staphylococcus aureusStructure-function analysis of the C3 binding region of Staphylococcus aureus immune subversion protein SbiShotgun phage display — Selection for bacterial receptins or other exported proteinsEvasion of Neutrophil Killing by Staphylococcus aureusAntimicrobial Mechanisms of Macrophages and the Immune Evasion Strategies of Staphylococcus aureusStaphylococcal manipulation of host immune responsesNeutrophil-Mediated Phagocytosis of Staphylococcus aureusA mutation in the PP2C phosphatase gene in a Staphylococcus aureus USA300 clinical isolate with reduced susceptibility to vancomycin and daptomycin.Identification of novel adhesins from Group B streptococci by use of phage display reveals that C5a peptidase mediates fibronectin binding.Complement evasion by human pathogens.Global transcriptome responses including small RNAs during mixed-species interactions with methicillin-resistant Staphylococcus aureus and Pseudomonas aeruginosaEpic Immune Battles of History: Neutrophils vs. Staphylococcus aureus.Repair of global regulators in Staphylococcus aureus 8325 and comparative analysis with other clinical isolates.Adhesive polypeptides of Staphylococcus aureus identified using a novel secretion library technique in Escherichia coli.Synthetic effects of secG and secY2 mutations on exoproteome biogenesis in Staphylococcus aureusThe complement systemGlobal analysis of community-associated methicillin-resistant Staphylococcus aureus exoproteins reveals molecules produced in vitro and during infection.The Moraxella catarrhalis immunoglobulin D-binding protein MID has conserved sequences and is regulated by a mechanism corresponding to phase variationEmerging functions for the Staphylococcus aureus RNome.The Sbi protein is a multifunctional immune evasion factor of Staphylococcus aureusThe Sbi Protein Contributes to Staphylococcus aureus Inflammatory Response during Systemic Infection.The immune evasion protein Sbi of Staphylococcus aureus occurs both extracellularly and anchored to the cell envelope by binding lipoteichoic acid.The WalKR system controls major staphylococcal virulence genes and is involved in triggering the host inflammatory responseProtein A-specific monoclonal antibodies and prevention of Staphylococcus aureus disease in mice.Vaccination with a UV-irradiated genetically attenuated mutant of Staphylococcus aureus provides protection against subsequent systemic infection.18β-Glycyrrhetinic acid inhibits methicillin-resistant Staphylococcus aureus survival and attenuates virulence gene expression.Recurrent infections and immune evasion strategies of Staphylococcus aureus.Role of protein A in the evasion of host adaptive immune responses by Staphylococcus aureus.Release of protein A from the cell wall of Staphylococcus aureus.Parasitic scabies mites and associated bacteria joining forces against host complement defence.Mapping the transcription start points of the Staphylococcus aureus eap, emp, and vwb promoters reveals a conserved octanucleotide sequence that is essential for expression of these genes.Excretion of cytoplasmic proteins in Staphylococcus is most likely not due to cell lysis.Adaptive Immunity Against Staphylococcus aureus.Novel Evasion Mechanisms of the Classical Complement Pathway.Genetic variation among hospital isolates of methicillin-sensitive Staphylococcus aureus: evidence for horizontal transfer of virulence genes.Molecular analysis of the interaction between staphylococcal virulence factor Sbi-IV and complement C3d.Persistent Staphylococcus aureus isolates from two independent cases of bacteremia display increased bacterial fitness and novel immune evasion phenotypes.
P2860
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P2860
description
1998 nî lūn-bûn
@nan
1998 թուականի Ապրիլին հրատարակուած գիտական յօդուած
@hyw
1998 թվականի ապրիլին հրատարակված գիտական հոդված
@hy
1998年の論文
@ja
1998年論文
@yue
1998年論文
@zh-hant
1998年論文
@zh-hk
1998年論文
@zh-mo
1998年論文
@zh-tw
1998年论文
@wuu
name
A second IgG-binding protein in Staphylococcus aureus.
@ast
A second IgG-binding protein in Staphylococcus aureus.
@en
type
label
A second IgG-binding protein in Staphylococcus aureus.
@ast
A second IgG-binding protein in Staphylococcus aureus.
@en
prefLabel
A second IgG-binding protein in Staphylococcus aureus.
@ast
A second IgG-binding protein in Staphylococcus aureus.
@en
P2093
P1433
P1476
A second IgG-binding protein in Staphylococcus aureus.
@en
P2093
P304
P356
10.1099/00221287-144-4-985
P478
144 ( Pt 4)
P577
1998-04-01T00:00:00Z