CADASIL: a common form of hereditary arteriopathy causing brain infarcts and dementia.
about
Schimke immuno-osseous dysplasia: a clinicopathological correlationMicrovascular pathology and morphometrics of sporadic and hereditary small vessel diseases of the brainNOTCH3 variants and risk of ischemic stroke.Von Willebrand Factor permeates small vessels in CADASIL and inhibits smooth muscle gene expression.Distinct phenotypic and functional features of CADASIL mutations in the Notch3 ligand binding domainCerebral small vessel disease: insights and opportunities from mouse models of collagen IV-related small vessel disease and cerebral autosomal dominant arteriopathy with subcortical infarcts and leukoencephalopathy.Role of electron microscopy in the diagnosis of cadasil syndrome: a study of 32 patientsEmbryonic origins of human vascular smooth muscle cells: implications for in vitro modeling and clinical applicationLombardia GENS: a collaborative registry for monogenic diseases associated with stroke.Glial vascular degeneration in CADASIL.Genetics of Vascular Dementia.Cerebral autosomal dominant arteriopathy with subcortical infarcts and leukoencephalopathy in an Israeli family.VEGF: a critical player in neurodegeneration.Generation of human vascular smooth muscle subtypes provides insight into embryological origin-dependent disease susceptibilityThe minimum prevalence of CADASIL in northeast England.Grey matter volume alterations in CADASIL: a voxel-based morphometry studyA Report of Accelerated Coronary Artery Disease Associated with Cerebral Autosomal Dominant Arteriopathy with Subcortical Infarcts and Leukoencephalopathy.notch3 is essential for oligodendrocyte development and vascular integrity in zebrafish.Congruence between NOTCH3 mutations and GOM in 131 CADASIL patients.Neuropathological correlates of temporal pole white matter hyperintensities in CADASIL.Valproate activates the Notch3/c-FLIP signaling cascade: a strategy to attenuate white matter hyperintensities in bipolar disorder in late life?Identification of critical paralog groups with indispensable roles in the regulation of signaling flow.CADASIL: Ultrastructural insights into the morphology of granular osmiophilic material.The Cerebrovascular-Chronic Kidney Disease Connection: Perspectives and Mechanisms.Notch signaling in cerebrovascular diseases (Review)Signaling required for blood vessel maintenance: molecular basis and pathological manifestations.Small vessel disease and subcortical vascular dementia.Bidirectional encroachment of collagen into the tunica media in cerebral autosomal dominant arteriopathy with subcortical infarcts and leukoencephalopathyRetinal and Cortical Blood Flow Dynamics Following Systemic Blood-Neural Barrier DisruptionDifferences in proliferation rate between CADASIL and control vascular smooth muscle cells are related to increased TGFβ expression.Neuroimaging in Dementia.
P2860
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P2860
CADASIL: a common form of hereditary arteriopathy causing brain infarcts and dementia.
description
2002 nî lūn-bûn
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2002 թուականի Յուլիսին հրատարակուած գիտական յօդուած
@hyw
2002 թվականի հուլիսին հրատարակված գիտական հոդված
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2002年の論文
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2002年論文
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2002年論文
@zh-hant
2002年論文
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2002年論文
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2002年論文
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2002年论文
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name
CADASIL: a common form of hereditary arteriopathy causing brain infarcts and dementia.
@ast
CADASIL: a common form of hereditary arteriopathy causing brain infarcts and dementia.
@en
type
label
CADASIL: a common form of hereditary arteriopathy causing brain infarcts and dementia.
@ast
CADASIL: a common form of hereditary arteriopathy causing brain infarcts and dementia.
@en
prefLabel
CADASIL: a common form of hereditary arteriopathy causing brain infarcts and dementia.
@ast
CADASIL: a common form of hereditary arteriopathy causing brain infarcts and dementia.
@en
P2093
P1433
P1476
CADASIL: a common form of hereditary arteriopathy causing brain infarcts and dementia.
@en
P2093
Hannu Kalimo
Marie-Magdaleine Ruchoux
Matti Viitanen
Raj N Kalaria
P304
P577
2002-07-01T00:00:00Z