Glucose uptake and glycolysis reduce hypoxia-induced apoptosis in cultured neonatal rat cardiac myocytes. - Wikidata - awgldk - TriplyDB

Glucose uptake and glycolysis reduce hypoxia-induced apoptosis in cultured neonatal rat cardiac myocytes.

Glucose uptake and glycolysis reduce hypoxia-induced apoptosis in cultured neonatal rat cardiac myocytes.

http://www.wikidata.org/entity/Q33178140

about

Effect of hypoxia on the expression of αB-crystallin in head and neck squamous cell carcinoma Apoptosis in myocardial ischaemia and infarction Survival of cancer cells is maintained by EGFR independent of its kinase activity Hypoxia-inducible transcription factor-1alpha promotes hypoxia-induced A549 apoptosis via a mechanism that involves the glycolysis pathway Glucose Transporters in Cardiac Metabolism and Hypertrophy Induced inhibition of ischemic/hypoxic injury by APIP, a novel Apaf-1-interacting protein Phosphoinositide 3-kinase accelerates necrotic cell death during hypoxia GLUT-1 reduces hypoxia-induced apoptosis and JNK pathway activation Down-regulation of ARC contributes to vulnerability of hippocampal neurons to ischemia/hypoxia Aldosterone, through novel signaling proteins, is a fundamental molecular bridge between the genetic defect and the cardiac phenotype of hypertrophic cardiomyopathy Involvement of GADD153 and cardiac ankyrin repeat protein in hypoxia-induced apoptosis of H9c2 cells Involvement of GADD153 and cardiac ankyrin repeat protein in cardiac ischemia-reperfusion injury Activation of the prolyl-hydroxylase oxygen-sensing signal cascade leads to AMPK activation in cardiomyocytes Decreased glucose transporter expression triggers BAX-dependent apoptosis in the murine blastocyst Inhibition of hypoxia/reoxygenation-induced apoptosis in metallothionein-overexpressing cardiomyocytes.Hypoxia-inducible factor-1alpha is a critical mediator of hypoxia induced apoptosis in cardiac H9c2 and kidney epithelial HK-2 cells.Microtubular stability affects cardiomyocyte glycolysis by HIF-1alpha expression and endonuclear aggregation during early stages of hypoxia.OGFOD1, a member of the 2-oxoglutarate and iron dependent dioxygenase family, functions in ischemic signaling.β-adrenergic receptor-dependent alterations in murine cardiac transcript expression are differentially regulated by gefitinib in vivo Differential utilization of ketone bodies by neurons and glioma cell lines: a rationale for ketogenic diet as experimental glioma therapy TanshinoneIIA and cryptotanshinone protect against hypoxia-induced mitochondrial apoptosis in H9c2 cells.Redox signaling in cardiac myocytes.Adenovirus-mediated transfection with glucose transporter 3 suppresses PC12 cell apoptosis following ischemic injury.Cardiac tissue slice transplantation as a model to assess tissue-engineered graft thickness, survival, and function.GLUT1 enhances mTOR activity independently of TSC2 and AMPK.Causes and characteristics of diabetic cardiomyopathy.Implications of glucose transporter protein type 1 (GLUT1)-haplodeficiency in embryonic stem cells for their survival in response to hypoxic stress.The dynamic stress-induced "O-GlcNAc-ome" highlights functions for O-GlcNAc in regulating DNA damage/repair and other cellular pathways.The roles of O-linked β-N-acetylglucosamine in cardiovascular physiology and disease.α1A-Adrenergic receptor prevents cardiac ischemic damage through PKCδ/GLUT1/4-mediated glucose uptake Hypoxia inducible factor-1alpha inactivation unveils a link between tumor cell metabolism and hypoxia-induced cell death.Toll-like receptor signaling: a critical modulator of cell survival and ischemic injury in the heart HIF-1α knockdown by miRNA decreases survivin expression and inhibits A549 cell growth in vitro and in vivo Hypoxia-mediated up-regulation of Pim-1 contributes to solid tumor formation.AMP-activated protein kinase mediates ischemic glucose uptake and prevents postischemic cardiac dysfunction, apoptosis, and injury.Is cancer a disease of abnormal cellular metabolism? New angles on an old idea Progesterone stimulates mitochondrial activity with subsequent inhibition of apoptosis in MCF-10A benign breast epithelial cells.Functional consequences of a tissue-engineered myocardial patch for cardiac repair in a rat infarct model Enhancing the metabolic substrate: PPAR-alpha agonists in heart failure.Hypoxia enhances the expression of autocrine motility factor and the motility of human pancreatic cancer cells

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Glucose uptake and glycolysis reduce hypoxia-induced apoptosis in cultured neonatal rat cardiac myocytes.

http://www.wikidata.org/entity/Q33178140

description

1999 nî lūn-bûn

@nan

1999 թուականի Ապրիլին հրատարակուած գիտական յօդուած

@hyw

1999 թվականի ապրիլին հրատարակված գիտական հոդված

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1999年の論文

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1999年論文

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1999年論文

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1999年論文

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1999年論文

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1999年論文

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1999年论文

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name

Glucose uptake and glycolysis ...... neonatal rat cardiac myocytes.

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Glucose uptake and glycolysis ...... neonatal rat cardiac myocytes.

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type

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Glucose uptake and glycolysis ...... neonatal rat cardiac myocytes.

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Glucose uptake and glycolysis ...... neonatal rat cardiac myocytes.

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Glucose uptake and glycolysis ...... neonatal rat cardiac myocytes.

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Glucose uptake and glycolysis ...... neonatal rat cardiac myocytes.

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Glucose uptake and glycolysis ...... neonatal rat cardiac myocytes.

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Brosius FC 3rd

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Malhotra R

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P2860

ARC, an inhibitor of apoptosis expressed in skeletal muscle and heart that interacts selectively with caspases

Bcl-xL regulates the membrane potential and volume homeostasis of mitochondria

Inhibition of death receptor signals by cellular FLIP

Interaction of CED-4 with CED-3 and CED-9: a molecular framework for cell death

Apaf-1, a human protein homologous to C. elegans CED-4, participates in cytochrome c-dependent activation of caspase-3

Apoptosis in the pathogenesis and treatment of disease

Role of HIF-1alpha in hypoxia-mediated apoptosis, cell proliferation and tumour angiogenesis

Death receptors: signaling and modulation

Bax suppresses tumorigenesis and stimulates apoptosis in vivo

Bax directly induces release of cytochrome c from isolated mitochondria

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