Targeting the Nogo receptor to treat central nervous system injuries.
about
Sialidase enhances recovery from spinal cord contusion injuryNogo goes in the pure water: solution structure of Nogo-60 and design of the structured and buffer-soluble Nogo-54 for enhancing CNS regenerationStimulating neuroregeneration as a therapeutic drug approach for traumatic brain injury.Effect of combined treatment with methylprednisolone and soluble Nogo-66 receptor after rat spinal cord injury.Selective temporal and regional alterations of Nogo-A and small proline-rich repeat protein 1A (SPRR1A) but not Nogo-66 receptor (NgR) occur following traumatic brain injury in the ratDelayed Nogo receptor therapy improves recovery from spinal cord contusionThe N-terminal domain of Nogo-A inhibits cell adhesion and axonal outgrowth by an integrin-specific mechanism.The newt in usMyelin basic protein induces neuron-specific toxicity by directly damaging the neuronal plasma membrane.Human NgR-Fc decoy protein via lumbar intrathecal bolus administration enhances recovery from rat spinal cord contusion.Mst3b, a purine-sensitive Ste20-like protein kinase, regulates axon outgrowthSensitive periods in the development of the brain and behavior.Cognitive outcome following brain injury and treatment with an inhibitor of Nogo-A in association with an attenuated downregulation of hippocampal growth-associated protein-43 expression.Tissue sparing and functional recovery following experimental traumatic brain injury is provided by treatment with an anti-myelin-associated glycoprotein antibody.A re-assessment of the effects of a Nogo-66 receptor antagonist on regenerative growth of axons and locomotor recovery after spinal cord injury in mice.Functional MRI and other non-invasive imaging technologies: providing visual biomarkers for spinal cord structure and function after injury.Nogo-A and Nogo-66 receptor in amyotrophic lateral sclerosis.Sink hypothesis and therapeutic strategies for attenuating Abeta levels.Injury and repair in the neurovascular unit.Nogo/RTN4 isoforms and RTN3 expression protect SH-SY5Y cells against multiple death insults.Characterization of myelin ligand complexes with neuronal Nogo-66 receptor family members.The beta-amyloid protein of Alzheimer's disease increases neuronal CRMP-2 phosphorylation by a Rho-GTP mechanism.Treatment of penetrating brain injury in a rat model using collagen scaffolds incorporating soluble Nogo receptor.
P2860
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P2860
Targeting the Nogo receptor to treat central nervous system injuries.
description
2003 nî lūn-bûn
@nan
2003 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած
@hyw
2003 թվականի նոյեմբերին հրատարակված գիտական հոդված
@hy
2003年の論文
@ja
2003年論文
@yue
2003年論文
@zh-hant
2003年論文
@zh-hk
2003年論文
@zh-mo
2003年論文
@zh-tw
2003年论文
@wuu
name
Targeting the Nogo receptor to treat central nervous system injuries.
@ast
Targeting the Nogo receptor to treat central nervous system injuries.
@en
type
label
Targeting the Nogo receptor to treat central nervous system injuries.
@ast
Targeting the Nogo receptor to treat central nervous system injuries.
@en
prefLabel
Targeting the Nogo receptor to treat central nervous system injuries.
@ast
Targeting the Nogo receptor to treat central nervous system injuries.
@en
P2860
P356
P1476
Targeting the Nogo receptor to treat central nervous system injuries.
@en
P2093
Daniel H S Lee
Dinah W Y Sah
P2860
P2888
P304
P356
10.1038/NRD1228
P577
2003-11-01T00:00:00Z