Chronic parasympathetic sectioning decreases regional cerebral blood flow during hemorrhagic hypotension and increases infarct size after middle cerebral artery occlusion in spontaneously hypertensive rats.

Chronic parasympathetic sectioning decreases regional cerebral blood flow during hemorrhagic hypotension and increases infarct size after middle cerebral artery occlusion in spontaneously hypertensive rats.

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http://www.wikidata.org/entity/Q33224089

Q33224089

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Neuronal nitric oxide mediates cerebral vasodilatation during acute hypertension Projections from the hypothalamic paraventricular nucleus and the nucleus of the solitary tract to prechoroidal neurons in the superior salivatory nucleus: Pathways controlling rodent choroidal blood flow L-arginine dilates rat pial arterioles by nitric oxide-dependent mechanisms and increases blood flow during focal cerebral ischaemia Choroidal blood flow compensation in rats for arterial blood pressure decreases is neuronal nitric oxide-dependent but compensation for arterial blood pressure increases is not.Altered heart rhythm dynamics in very low birth weight infants with impending intraventricular hemorrhage Flow Augmentation in Acute Ischemic Stroke.The identification and neurochemical characterization of central neurons that target parasympathetic preganglionic neurons involved in the regulation of choroidal blood flow in the rat eye using pseudorabies virus, immunolabeling and conventional pa Stimulation of Baroresponsive Parts of the Nucleus of the Solitary Tract Produces Nitric Oxide-mediated Choroidal Vasodilation in Rat Eye.Influence of cerebrovascular sympathetic, parasympathetic, and sensory nerves on autoregulation and spontaneous vasomotion.Inhibition of nitric oxide synthase attenuates the cerebral blood flow response to stimulation of postganglionic parasympathetic nerves in the rat.Contribution of cerebrovascular parasympathetic and sensory innervation to the short-term control of blood flow in rat cerebral cortex.Ablation of the sphenopalatine ganglion does not attenuate the infarct reducing effect of vagus nerve stimulation.Intravenous infusion of adrenomedullin and increase in regional cerebral blood flow and prevention of ischemic brain injury after middle cerebral artery occlusion in rats.Effects of Stroke on the Autonomic Nervous System.Prediction of intraventricular haemorrhage in preterm infants using time series analysis of blood pressure and respiratory signals.Parasympathetic innervation of vertebrobasilar arteries: is this a potential clinical target?Late stimulation of the sphenopalatine-ganglion in ischemic rats: improvement in N-acetyl-aspartate levels and diffusion weighted imaging characteristics as seen by MR.Parasympathetic tonic dilatory influences on cerebral vessels.Detrended fluctuation analysis of blood pressure in preterm infants with intraventricular hemorrhage.Direct projections from the cardiovascular nucleus tractus solitarii to pontine preganglionic parasympathetic neurons: a link to cerebrovascular regulation.Implant for augmentation of cerebral blood flow trial 1: a pilot study evaluating the safety and effectiveness of the Ischaemic Stroke System for treatment of acute ischaemic stroke.

P2860

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P2860

Chronic parasympathetic sectioning decreases regional cerebral blood flow during hemorrhagic hypotension and increases infarct size after middle cerebral artery occlusion in spontaneously hypertensive rats.

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http://www.wikidata.org/entity/Q33224089

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1992 nî lūn-bûn

@nan

1992 թուականի Յուլիսին հրատարակուած գիտական յօդուած

@hyw

1992 թվականի հուլիսին հրատարակված գիտական հոդված

@hy

1992年の論文

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1992年論文

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1992年論文

@zh-hant

1992年論文

@zh-hk

1992年論文

@zh-mo

1992年論文

@zh-tw

1992年论文

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