No widespread induction of cell death genes occurs in pure motoneurons in an amyotrophic lateral sclerosis mouse model. - Wikidata - awgldk - TriplyDB

No widespread induction of cell death genes occurs in pure motoneurons in an amyotrophic lateral sclerosis mouse model.

No widespread induction of cell death genes occurs in pure motoneurons in an amyotrophic lateral sclerosis mouse model.

http://www.wikidata.org/entity/Q33224176

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Oxr1 is essential for protection against oxidative stress-induced neurodegeneration Gene expression profiling for human iPS-derived motor neurons from sporadic ALS patients reveals a strong association between mitochondrial functions and neurodegeneration.Electroconvulsive seizure-induced gene expression profile of the hippocampus dentate gyrus granule cell layer.Axonal involvement in the Wlds neuroprotective effect: analysis of pure motoneurons in a mouse model protected from motor neuron disease at a pre-symptomatic age.Toxicity from different SOD1 mutants dysregulates the complement system and the neuronal regenerative response in ALS motor neurons.Weighted gene co-expression network analysis of the peripheral blood from Amyotrophic Lateral Sclerosis patients Deregulated expression of cytoskeleton related genes in the spinal cord and sciatic nerve of presymptomatic SOD1(G93A) Amyotrophic Lateral Sclerosis mouse model.Axotomy-induced target disconnection promotes an additional death mechanism involved in motoneuron degeneration in amyotrophic lateral sclerosis transgenic mice.Inhibition of β-Glucocerebrosidase Activity Preserves Motor Unit Integrity in a Mouse Model of Amyotrophic Lateral Sclerosis.The emerging use of in vivo optical imaging in the study of neurodegenerative diseases.Early gene expression changes in spinal cord from SOD1(G93A) Amyotrophic Lateral Sclerosis animal model.Neuron-specific antioxidant OXR1 extends survival of a mouse model of amyotrophic lateral sclerosis Gene expression changes in spinal motoneurons of the SOD1(G93A) transgenic model for ALS after treatment with G-CSF.Brca1 is expressed in human microglia and is dysregulated in human and animal model of ALS.Dysregulated expression of death, stress and mitochondrion related genes in the sciatic nerve of presymptomatic SOD1(G93A) mouse model of Amyotrophic Lateral Sclerosis.Type I Vs. Type II Cytokine Levels as a Function of SOD1 G93A Mouse Amyotrophic Lateral Sclerosis Disease Progression.Translational profiling identifies a cascade of damage initiated in motor neurons and spreading to glia in mutant SOD1-mediated ALS.An indicator cell assay for blood-based diagnostics.Neurofilaments at a glance.Current and future directions in genomics of amyotrophic lateral sclerosis.Stathmin 1/2-triggered microtubule loss mediates Golgi fragmentation in mutant SOD1 motor neurons.C1q induction and global complement pathway activation do not contribute to ALS toxicity in mutant SOD1 mice.Investigating cell death mechanisms in amyotrophic lateral sclerosis using transcriptomics.Can transcriptomics cut the gordian knot of amyotrophic lateral sclerosis?Differential effect of oxidative or excitotoxic stress on the transcriptional profile of amyotrophic lateral sclerosis-linked mutant SOD1 cultured neurons.Microglia Responses in Acute and Chronic Neurological Diseases: What Microglia-Specific Transcriptomic Studies Taught (and did Not Teach) Us.Common and Divergent Mechanisms in Developmental Neuronal Remodeling and Dying Back Neurodegeneration.Gene expression profile of SOD1-G93A mouse spinal cord, blood and muscle.Gene Expression Profiling of Cutaneous Injured and Non-Injured Nociceptors in SNI Animal Model of Neuropathic Pain.Adeno-associated virus-mediated delivery of a recombinant single-chain antibody against misfolded superoxide dismutase for treatment of amyotrophic lateral sclerosis.Unraveling gene expression profiles in peripheral motor nerve from amyotrophic lateral sclerosis patients: insights into pathogenesis.Meta-analysis of gene expression profiling in amyotrophic lateral sclerosis: a comparison between transgenic mouse models and human patients.Superoxide dismutase-1 and other proteins in inclusions from transgenic amyotrophic lateral sclerosis model mice.RNA-Seq Analysis of Spinal Cord Tissues from hPFN1 Transgenic Mouse Model of ALS at Pre-symptomatic and End-Stages of Disease

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P2860

No widespread induction of cell death genes occurs in pure motoneurons in an amyotrophic lateral sclerosis mouse model.

http://www.wikidata.org/entity/Q33224176

description

2005 nî lūn-bûn

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2005 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած

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2005 թվականի սեպտեմբերին հրատարակված գիտական հոդված

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2005年の論文

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2005年学术文章

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2005年学术文章

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2005年学术文章

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2005年学术文章

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2005年学术文章

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2005年學術文章

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name

No widespread induction of cel ...... lateral sclerosis mouse model.

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No widespread induction of cel ...... lateral sclerosis mouse model.

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Human Molecular Genetics

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No widespread induction of cel ...... lateral sclerosis mouse model

@en

P2093

Ann C Kato

http://www.w3.org/2001/XMLSchema#string

Gaelle Boisset

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Mylene Docquier

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Olivier Schaad

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Patrick Descombes

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P2860

Axonal plasticity and functional recovery after spinal cord injury in mice deficient in both glial fibrillary acidic protein and vimentin genes.

Matrix metalloproteinases

14-3-3 Interacts directly with and negatively regulates pro-apoptotic Bax

Aggresomes: a cellular response to misfolded proteins

Unraveling the mechanisms involved in motor neuron degeneration in ALS

Protofibrils, pores, fibrils, and neurodegeneration: separating the responsible protein aggregates from the innocent bystanders

Differential expression of 14 genes in amyotrophic lateral sclerosis spinal cord detected using gridded cDNA arrays.

Gene expression profile of spinal motor neurons in sporadic amyotrophic lateral sclerosis.

IAP family proteins delay motoneuron cell death in vivo.

Metalloproteinases in biology and pathology of the nervous system.

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3309-3320

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10.1093/HMG/DDI357

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21

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14

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Florence E Perrin

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2005-09-28T00:00:00Z

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7572822

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amyotrophic lateral sclerosis