Ex vivo gene delivery of GDNF using primary astrocytes transduced with a lentiviral vector provides neuroprotection in a rat model of Parkinson's disease.
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Inhibition of the leucine-rich repeat protein LINGO-1 enhances survival, structure, and function of dopaminergic neurons in Parkinson's disease modelsTransplantation of stem cell-derived astrocytes for the treatment of amyotrophic lateral sclerosis and spinal cord injuryThe nigrostriatal dopamine system of aging GFRalpha-1 heterozygous mice: neurochemistry, morphology and behaviorNeuroregeneration in neurodegenerative disorders.Blocking LINGO-1 as a therapy to promote CNS repair: from concept to the clinic.Anti-tat Hutat2:Fc mediated protection against tat-induced neurotoxicity and HIV-1 replication in human monocyte-derived macrophagesAdvances in gene therapy for movement disorders.Regulated expression of lentivirus-mediated GDNF in human bone marrow-derived mesenchymal stem cells and its neuroprotection on dopaminergic cells in vitro.Potential sources of stem cells as a regenerative therapy for Parkinson's disease.Transplantation of subventricular zone neural precursors induces an endogenous precursor cell response in a rat model of Parkinson's disease.Delayed transplantation of precursor cell-derived astrocytes provides multiple benefits in a rat model of Parkinsons.Stem and Progenitor Cell-Derived Astroglia Therapies for Neurological Diseases.Stem cells therapy for ALS.Ret is essential to mediate GDNF's neuroprotective and neuroregenerative effect in a Parkinson disease mouse model.Glutathione transferase-M2-2 secreted from glioblastoma cell protects SH-SY5Y cells from aminochrome neurotoxicity.Glutathione transferase mu 2 protects glioblastoma cells against aminochrome toxicity by preventing autophagy and lysosome dysfunction.Adeno-associated virus type 2 vector-mediated glial cell line-derived neurotrophic factor gene transfer induces neuroprotection and neuroregeneration in a ubiquitin-proteasome system impairment animal model of Parkinson's disease.Overexpression of serum response factor restores ocular dominance plasticity in a model of fetal alcohol spectrum disorders.Optimized quantities of GDNF overexpressed by engineered astrocytes are critical for protection of neuroblastoma cells against 6-OHDA toxicity.BDNF regulation under GFAP promoter provides engineered astrocytes as a new approach for long-term protection in Huntington's disease.Overexpression of Serum Response Factor in Neurons Restores Ocular Dominance Plasticity in a Model of Fetal Alcohol Spectrum DisordersOverexpression of serum response factor in astrocytes improves neuronal plasticity in a model of early alcohol exposure.
P2860
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P2860
Ex vivo gene delivery of GDNF using primary astrocytes transduced with a lentiviral vector provides neuroprotection in a rat model of Parkinson's disease.
description
2005 nî lūn-bûn
@nan
2005 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2005 թվականի դեկտեմբերին հրատարակված գիտական հոդված
@hy
2005年の論文
@ja
2005年論文
@yue
2005年論文
@zh-hant
2005年論文
@zh-hk
2005年論文
@zh-mo
2005年論文
@zh-tw
2005年论文
@wuu
name
Ex vivo gene delivery of GDNF ...... model of Parkinson's disease.
@ast
Ex vivo gene delivery of GDNF ...... model of Parkinson's disease.
@en
type
label
Ex vivo gene delivery of GDNF ...... model of Parkinson's disease.
@ast
Ex vivo gene delivery of GDNF ...... model of Parkinson's disease.
@en
prefLabel
Ex vivo gene delivery of GDNF ...... model of Parkinson's disease.
@ast
Ex vivo gene delivery of GDNF ...... model of Parkinson's disease.
@en
P2093
P921
P1476
Ex vivo gene delivery of GDNF ...... model of Parkinson's disease.
@en
P2093
Biljana Georgievska
Cecilia Ericson
Cecilia Lundberg
P304
P356
10.1111/J.1460-9568.2005.04503.X
P407
P577
2005-12-01T00:00:00Z