Inhibition of gap junction communication at ectopic Eph/ephrin boundaries underlies craniofrontonasal syndrome
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Eph/ephrin molecules--a hub for signaling and endocytosisNon-SH2/PDZ reverse signaling by ephrinsLine-scanning particle image velocimetry: an optical approach for quantifying a wide range of blood flow speeds in live animalsCartilage-specific deletion of ephrin-B2 in mice results in early developmental defects and an osteoarthritis-like phenotype during aging in vivo.Receptor tyrosine kinase signaling: regulating neural crest development one phosphate at a timeThe ephrin signaling pathway regulates morphology and adhesion of mouse granulosa cells in vitroSchlemm's canal is a unique vessel with a combination of blood vascular and lymphatic phenotypes that forms by a novel developmental processLoss of ephrin-A5 function disrupts lens fiber cell packing and leads to cataractBrain Vascular Imaging TechniquesThe role of GRIP1 and ephrin B3 in blood pressure control and vascular smooth muscle cell contractilityAn actomyosin-based barrier inhibits cell mixing at compartmental boundaries in Drosophila embryos.The impact of CFNS-causing EFNB1 mutations on ephrin-B1 functionEphrin B1 regulates bone marrow stromal cell differentiation and bone formation by influencing TAZ transactivation via complex formation with NHERF1.Clinical, genetic and imaging findings identify new causes for corpus callosum development syndromes.EphA4 and EfnB2a maintain rhombomere coherence by independently regulating intercalation of progenitor cells in the zebrafish neural keel.Regulation of lens gap junctions by Transforming Growth Factor beta.Phenotypes of craniofrontonasal syndrome in patients with a pathogenic mutation in EFNB1.EphrinB1 interacts with CNK1 and promotes cell migration through c-Jun N-terminal kinase (JNK) activationVessel arterial-venous plasticity in adult neovascularizationThe effect of conditional EFNB1 deletion in the T cell compartment on T cell development and function.Ephrin-B1 forward signaling regulates craniofacial morphogenesis by controlling cell proliferation across Eph-ephrin boundaries.Targeted disruption of ephrin B1 in cells of myeloid lineage increases osteoclast differentiation and bone resorption in mice.Ephrin reverse signaling in axon guidance and synaptogenesis.Cellular interference in craniofrontonasal syndrome: males mosaic for mutations in the X-linked EFNB1 gene are more severely affected than true hemizygotes.Molecular bioelectricity: how endogenous voltage potentials control cell behavior and instruct pattern regulation in vivo.Transgenic overexpression of ephrin b1 in bone cells promotes bone formation and an anabolic response to mechanical loading in mice.EphB/ephrinB signaling in cell adhesion and migration.Connexin37 and Connexin43 deficiencies in mice disrupt lymphatic valve development and result in lymphatic disorders including lymphedema and chylothoraxEndothelial ephrin-B2 is essential for arterial vasodilation in mice.Genistein attenuates glucocorticoid-induced bone deleterious effects through regulation Eph/ephrin expression in aged mice.Duplication of the EFNB1 gene in familial hypertelorism: imbalance in ephrin-B1 expression and abnormal phenotypes in humans and mice.Molecular mechanisms of cell segregation and boundary formation in development and tumorigenesis.Essential role of PDZ-RGS3 in the maintenance of neural progenitor cells.Early molecular responses of bone to estrogen deficiency induced by ovariectomy in rats.Receptor tyrosine kinase Ephb6 regulates vascular smooth muscle contractility and modulates blood pressure in concert with sex hormones.Efnb1 and Efnb2 proteins regulate thymocyte development, peripheral T cell differentiation, and antiviral immune responses and are essential for interleukin-6 (IL-6) signaling.Ephrin-B2 forward signaling regulates somite patterning and neural crest cell development.Possible role of Efnb1 protein, a ligand of Eph receptor tyrosine kinases, in modulating blood pressureCONNEXIN 43 AND BONE: NOT JUST A GAP JUNCTION PROTEIN.Bone cell interactions through Eph/ephrin: bone modeling, remodeling and associated diseases.
P2860
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P2860
Inhibition of gap junction communication at ectopic Eph/ephrin boundaries underlies craniofrontonasal syndrome
description
2006 nî lūn-bûn
@nan
2006 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2006 թվականի հոտեմբերին հրատարակված գիտական հոդված
@hy
2006年の論文
@ja
2006年論文
@yue
2006年論文
@zh-hant
2006年論文
@zh-hk
2006年論文
@zh-mo
2006年論文
@zh-tw
2006年论文
@wuu
name
Inhibition of gap junction com ...... ies craniofrontonasal syndrome
@ast
Inhibition of gap junction com ...... ies craniofrontonasal syndrome
@en
Inhibition of gap junction com ...... es craniofrontonasal syndrome.
@nl
type
label
Inhibition of gap junction com ...... ies craniofrontonasal syndrome
@ast
Inhibition of gap junction com ...... ies craniofrontonasal syndrome
@en
Inhibition of gap junction com ...... es craniofrontonasal syndrome.
@nl
prefLabel
Inhibition of gap junction com ...... ies craniofrontonasal syndrome
@ast
Inhibition of gap junction com ...... ies craniofrontonasal syndrome
@en
Inhibition of gap junction com ...... es craniofrontonasal syndrome.
@nl
P2860
P1433
P1476
Inhibition of gap junction com ...... ies craniofrontonasal syndrome
@en
P2093
Alice Davy
Jeffrey O Bush
P2860
P356
10.1371/JOURNAL.PBIO.0040315
P407
P577
2006-10-01T00:00:00Z