Activating mutations in c-KIT and PDGFRalpha are exclusively found in gastrointestinal stromal tumors and not in other tumors overexpressing these imatinib mesylate target genes.
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Immunohistochemical analysis of receptor tyrosine kinase signal transduction activity in chordomaImatinib in the treatment of solid tumours.Markers aiding the diagnosis of chondroid tumors: an immunohistochemical study including osteonectin, bcl-2, cox-2, actin, calponin, D2-40 (podoplanin), mdm-2, CD117 (c-kit), and YKL-40.Effects of AZD2171 and vandetanib (ZD6474, Zactima) on haemodynamic variables in an SW620 human colon tumour model: an investigation using dynamic contrast-enhanced MRI and the rapid clearance blood pool contrast agent, P792 (gadomelitol).Treatment of gastrointestinal stromal tumor: focus on imatinib mesylate.Process map proposal for the validation of genomic biomarkers.Endometrial stromal tumors: immunohistochemical and molecular analysis of potential targets of tyrosine kinase inhibitors.Molecular analysis of metaplastic breast carcinoma: high EGFR copy number via aneusomyThe use of human tumour cell lines in the discovery of new cancer chemotherapeutic drugs.Relationship between gene mutations and protein expressions of PDGFR α and C-kit in gastrointestinal stromal tumors.Coexistence of gastrointestinal stromal tumors and gastric adenocarcinomas.Positive immunohistochemical staining of KIT in solid-pseudopapillary neoplasms of the pancreas is not associated with KIT/PDGFRA mutations.Array comparative genomic hybridization reveals frequent alterations of G1/S checkpoint genes in undifferentiated pleomorphic sarcoma of bone.Receptor tyrosine kinases in sinonasal undifferentiated carcinomas--evaluation for EGFR, c-KIT, and HER2/neu expression.Platelet-derived growth factor receptor alpha mutational status and immunohistochemical expression in Merkel cell carcinoma: implications for treatment with imatinib mesylate.CD117/c-kit in Cancer Stem Cell-Mediated Progression and Therapeutic Resistance.Determining the profiles and parameters for gene amplification testing of growth factor receptors in lung cancer.PDGFRα: a new therapeutic target in the treatment of hepatocellular carcinoma?
P2860
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P2860
Activating mutations in c-KIT and PDGFRalpha are exclusively found in gastrointestinal stromal tumors and not in other tumors overexpressing these imatinib mesylate target genes.
description
2005 nî lūn-bûn
@nan
2005 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած
@hyw
2005 թվականի նոյեմբերին հրատարակված գիտական հոդված
@hy
2005年の論文
@ja
2005年論文
@yue
2005年論文
@zh-hant
2005年論文
@zh-hk
2005年論文
@zh-mo
2005年論文
@zh-tw
2005年论文
@wuu
name
Activating mutations in c-KIT ...... matinib mesylate target genes.
@ast
Activating mutations in c-KIT ...... matinib mesylate target genes.
@en
type
label
Activating mutations in c-KIT ...... matinib mesylate target genes.
@ast
Activating mutations in c-KIT ...... matinib mesylate target genes.
@en
prefLabel
Activating mutations in c-KIT ...... matinib mesylate target genes.
@ast
Activating mutations in c-KIT ...... matinib mesylate target genes.
@en
P2093
P356
P1476
Activating mutations in c-KIT ...... matinib mesylate target genes.
@en
P2093
Alex M de Bruin
Erik A C Wiemer
Erwin van der Harst
Hans P de Schipper
Hans van Tol
Harry F G M van den Ingh
Herman Burger
Johan M Kros
Kees Nooter
Michael A den Bakker
P304
P356
10.4161/CBT.4.11.2253
P577
2005-11-18T00:00:00Z