Mechanisms underlying the impairment of ischemia-induced neovascularization in matrix metalloproteinase 2-deficient mice. - Wikidata - awgldk - TriplyDB

Mechanisms underlying the impairment of ischemia-induced neovascularization in matrix metalloproteinase 2-deficient mice.

Mechanisms underlying the impairment of ischemia-induced neovascularization in matrix metalloproteinase 2-deficient mice.

http://www.wikidata.org/entity/Q33275480

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Role of exercise in the prevention of cardiovascular disease: results, mechanisms, and new perspectives Effect of lumican on the migration of human mesenchymal stem cells and endothelial progenitor cells: involvement of matrix metalloproteinase-14 Endothelial progenitor cells: identity defined?OX40 ligand plays an important role in the development of atherosclerosis through vasa vasorum neovascularization Electrical Stimulation of Denervated Rat Skeletal Muscle Retards Capillary and Muscle Loss in Early Stages of Disuse Atrophy Tissue inhibitor of metalloproteinase-3 via oncolytic herpesvirus inhibits tumor growth and vascular progenitors Exercise restores muscle stem cell mobilization, regenerative capacity and muscle metabolic alterations via adiponectin/AdipoR1 activation in SAMP10 mice CD34+ cells represent highly functional endothelial progenitor cells in murine bone marrow.Mechanism of diastolic stiffening of the failing myocardium and its prevention by angiotensin receptor and calcium channel blockers.Bone marrow alterations and lower endothelial progenitor cell numbers in critical limb ischemia patients.Exercise training stimulates ischemia-induced neovascularization via phosphatidylinositol 3-kinase/Akt-dependent hypoxia-induced factor-1 alpha reactivation in mice of advanced age Multi-analyte profiling reveals matrix metalloproteinase-9 and monocyte chemotactic protein-1 as plasma biomarkers of cardiac aging.Cathepsin K-mediated Notch1 activation contributes to neovascularization in response to hypoxia.Increased Circulating Cathepsin K in Patients with Chronic Heart Failure Angiotensin type 1 receptor blocker reduces intimal neovascularization and plaque growth in apolipoprotein E-deficient mice Cysteinyl cathepsins: multifunctional enzymes in cardiovascular disease.Role of Matrix Metalloproteinase-2, Matrix Metalloproteinase-9, and Vascular Endothelial Growth Factor in the Development of Chronic Subdural Hematoma Circulating angiogenic precursors in idiopathic pulmonary arterial hypertension.Tissue kallikrein is essential for invasive capacity of circulating proangiogenic cells.Superoxide-dependent cathepsin activation is associated with hypertensive myocardial remodeling and represents a target for angiotensin II type 1 receptor blocker treatment.Thrombospondin-2 modulates extracellular matrix remodeling during physiological angiogenesis Matrix metalloproteinases: inflammatory regulators of cell behaviors in vascular formation and remodeling The angiogenic response is dictated by beta3 integrin on bone marrow-derived cells.Matrix metalloproteinase 13 is a new contributor to skeletal muscle regeneration and critical for myoblast migration.Exercise training and peripheral arterial disease.Endothelial colony-forming cells derived from pregnancies complicated by intrauterine growth restriction are fewer and have reduced vasculogenic capacity.Cell therapies for therapeutic angiogenesis: back to the bench.In Vivo Angiogenic Capacity of Stem Cells from Human Exfoliated Deciduous Teeth with Human Umbilical Vein Endothelial Cells.Intratracheal transplantation of endothelial progenitor cells attenuates smoking-induced COPD in mice.The Soluble VEGF Receptor sFlt-1 Contributes to Impaired Neovascularization in Aged Mice Progenitor Cells for Arterial Repair: Incremental Advancements towards Therapeutic Reality.Mitochondrial-Targeted Antioxidant Maintains Blood Flow, Mitochondrial Function, and Redox Balance in Old Mice Following Prolonged Limb Ischemia.Cathepsin K activity controls injury-related vascular repair in mice.Cleaved high molecular weight kininogen inhibits tube formation of endothelial progenitor cells via suppression of matrix metalloproteinase 2.Vasculogenic cytokines in wound healing.Tissue Inhibitor of Metalloproteinase 1 Influences Vascular Adaptations to Chronic Alterations in Blood Flow.Repeated phlebotomy augments angiogenesis to improve blood flow in murine ischemic legs.Chronic Psychological Stress Accelerates Vascular Senescence and Impairs Ischemia-Induced Neovascularization: The Role of Dipeptidyl Peptidase-4/Glucagon-Like Peptide-1-Adiponectin Axis.Cathepsin K activity controls cardiotoxin-induced skeletal muscle repair in mice.Relation of functional and morphological changes in mitochondria to myocardial contractile and relaxation reserves in asymptomatic to mildly symptomatic patients with hypertrophic cardiomyopathy.

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Mechanisms underlying the impairment of ischemia-induced neovascularization in matrix metalloproteinase 2-deficient mice.

http://www.wikidata.org/entity/Q33275480

description

2007 nî lūn-bûn

@nan

2007 թուականի Փետրուարին հրատարակուած գիտական յօդուած

@hyw

2007 թվականի փետրվարին հրատարակված գիտական հոդված

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2007年の論文

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2007年論文

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2007年論文

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2007年論文

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2007年論文

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2007年論文

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2007年论文

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name

Mechanisms underlying the impa ...... loproteinase 2-deficient mice.

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Mechanisms underlying the impa ...... loproteinase 2-deficient mice.

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Mechanisms underlying the impa ...... loproteinase 2-deficient mice.

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Mechanisms underlying the impa ...... loproteinase 2-deficient mice.

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Mechanisms underlying the impa ...... loproteinase 2-deficient mice.

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Mechanisms underlying the impa ...... loproteinase 2-deficient mice.

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Mechanisms underlying the impa ...... loproteinase 2-deficient mice.

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Mechanisms underlying the impa ...... loproteinase 2-deficient mice.

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Mechanisms underlying the impa ...... loproteinase 2-deficient mice.

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Circulation Research

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Mechanisms underlying the impa ...... loproteinase 2-deficient mice.

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Akihisa Iguchi

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Hai Jin

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Kae Nakamura

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Keiko Maeda

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Kenji Okumura

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Masafumi Kuzuya

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Michitaka Tsuzuki

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Mitsuhiro Yokota

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Natsuo Inoue

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Takahisa Kondo

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10.1161/01.RES.0000260801.12916.B5

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neovascularization