Systematic identification of cellular signals reactivating Kaposi sarcoma-associated herpesvirus.
about
KSHV Genome Replication and MaintenancePhosphoproteomic analyses reveal signaling pathways that facilitate lytic gammaherpesvirus replicationActivation of PI3K/AKT and ERK MAPK signal pathways is required for the induction of lytic cycle replication of Kaposi's sarcoma-associated herpesvirus by herpes simplex virus type 1KSHV reactivation from latency requires Pim-1 and Pim-3 kinases to inactivate the latency-associated nuclear antigen LANAControl of Kaposi's sarcoma-associated herpesvirus reactivation induced by multiple signalsTelomere-independent Rap1 is an IKK adaptor and regulates NF-kappaB-dependent gene expressionProgressive accumulation of activated ERK2 within highly stable ORF45-containing nuclear complexes promotes lytic gammaherpesvirus infectionDistinct roles for extracellular signal-regulated kinase 1 (ERK1) and ERK2 in the structure and production of a primate gammaherpesvirusActivation of p90 ribosomal S6 kinase by ORF45 of Kaposi's sarcoma-associated herpesvirus and its role in viral lytic replicationHigh-resolution functional profiling of a gammaherpesvirus RTA locus in the context of the viral genomeInhibition of the phosphatidylinositol 3-kinase-Akt pathway enhances gamma-2 herpesvirus lytic replication and facilitates reactivation from latency.Viral latency and its regulation: lessons from the gamma-herpesviruses.Gene expression and transcription factor profiling reveal inhibition of transcription factor cAMP-response element-binding protein by gamma-herpesvirus replication and transcription activatorMicroRNAs encoded by Kaposi's sarcoma-associated herpesvirus regulate viral life cycle.Resveratrol inhibits KSHV reactivation by lowering the levels of cellular EGR-1Cell membrane-bound Kaposi's sarcoma-associated herpesvirus-encoded glycoprotein B promotes virus latency by regulating expression of cellular Egr-1KSHV miRNAs decrease expression of lytic genes in latently infected PEL and endothelial cells by targeting host transcription factors.Virus-encoded microRNAs.Mechanisms of Kaposi's Sarcoma-Associated Herpesvirus Latency and ReactivationMolecular biology of KSHV lytic reactivation.Molecular biology of Kaposi's sarcoma-associated herpesvirus and related oncogenesisExtracellular Hsp90 serves as a co-factor for MAPK activation and latent viral gene expression during de novo infection by KSHV.Cellular MicroRNA Let-7a Suppresses KSHV Replication through Targeting MAP4K4 Signaling Pathways.ORF45-Mediated Prolonged c-Fos Accumulation Accelerates Viral Transcription during the Late Stage of Lytic Replication of Kaposi's Sarcoma-Associated Herpesvirus.Varicella-Zoster virus ORF12 protein triggers phosphorylation of ERK1/2 and inhibits apoptosis.Oncogenic Herpesvirus Utilizes Stress-Induced Cell Cycle Checkpoints for Efficient Lytic ReplicationAn APE1 inhibitor reveals critical roles of the redox function of APE1 in KSHV replication and pathogenic phenotypesEts-1 is required for the activation of VEGFR3 during latent Kaposi's sarcoma-associated herpesvirus infection of endothelial cells.Kaposi's Sarcoma-Associated Herpesvirus MicroRNAs Target GADD45B To Protect Infected Cells from Cell Cycle Arrest and ApoptosisViral miRNAs and immune evasionX box binding protein XBP-1s transactivates the Kaposi's sarcoma-associated herpesvirus (KSHV) ORF50 promoter, linking plasma cell differentiation to KSHV reactivation from latency.The K1 Protein of Kaposi's Sarcoma-Associated Herpesvirus Augments Viral Lytic Replication.Kaposi sarcoma-associated herpesvirus: immunobiology, oncogenesis, and therapy.Reactivation and Lytic Replication of Kaposi's Sarcoma-Associated Herpesvirus: An Update.Cdk1 inhibition induces mutually inhibitory apoptosis and reactivation of Kaposi's sarcoma-associated herpesvirus.Myc is required for the maintenance of Kaposi's sarcoma-associated herpesvirus latencyVirus reactivation: a panoramic view in human infectionsIntegrative systems control approach for reactivating Kaposi's sarcoma-associated herpesvirus (KSHV) with combinatory drugs.Kaposi sarcoma-associated herpes virus (KSHV) G protein-coupled receptor (vGPCR) activates the ORF50 lytic switch promoter: a potential positive feedback loop for sustained ORF50 gene expressionB cell terminal differentiation factor XBP-1 induces reactivation of Kaposi's sarcoma-associated herpesvirus.
P2860
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P2860
Systematic identification of cellular signals reactivating Kaposi sarcoma-associated herpesvirus.
description
2007 nî lūn-bûn
@nan
2007 թուականի Մարտին հրատարակուած գիտական յօդուած
@hyw
2007 թվականի մարտին հրատարակված գիտական հոդված
@hy
2007年の論文
@ja
2007年論文
@yue
2007年論文
@zh-hant
2007年論文
@zh-hk
2007年論文
@zh-mo
2007年論文
@zh-tw
2007年论文
@wuu
name
Systematic identification of c ...... arcoma-associated herpesvirus.
@ast
Systematic identification of c ...... arcoma-associated herpesvirus.
@en
Systematic identification of c ...... arcoma-associated herpesvirus.
@nl
type
label
Systematic identification of c ...... arcoma-associated herpesvirus.
@ast
Systematic identification of c ...... arcoma-associated herpesvirus.
@en
Systematic identification of c ...... arcoma-associated herpesvirus.
@nl
prefLabel
Systematic identification of c ...... arcoma-associated herpesvirus.
@ast
Systematic identification of c ...... arcoma-associated herpesvirus.
@en
Systematic identification of c ...... arcoma-associated herpesvirus.
@nl
P2093
P2860
P1433
P1476
Systematic identification of c ...... arcoma-associated herpesvirus.
@en
P2093
Christian G Nelson
Fuyuhiko Tamanoi
Helen J Brown
Hongyu Deng
Jeffrey Vieira
Josephine N Harada
Juran Kato-Stankiewicz
Moon Jung Song
P2860
P356
10.1371/JOURNAL.PPAT.0030044
P577
2007-03-01T00:00:00Z