Mandatory role of proteinase-activated receptor 1 in experimental bladder inflammation - Wikidata - awgldk - TriplyDB

Mandatory role of proteinase-activated receptor 1 in experimental bladder inflammation

Mandatory role of proteinase-activated receptor 1 in experimental bladder inflammation

http://www.wikidata.org/entity/Q33280660

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Regulatory network of inflammation downstream of proteinase-activated receptors Transcription factor network downstream of protease activated receptors (PARs) modulating mouse bladder inflammation.Molecular networks discriminating mouse bladder responses to intravesical bacillus Calmette-Guerin (BCG), LPS, and TNF-alpha.Loss of prostaglandin E2 release from immortalized urothelial cells obtained from interstitial cystitis patient bladders Thrombin induces macrophage migration inhibitory factor release and upregulation in urothelium: a possible contribution to bladder inflammation.VEGF signaling mediates bladder neuroplasticity and inflammation in response to BCG VEGF induces sensory and motor peripheral plasticity, alters bladder function, and promotes visceral sensitivity.Par genes: molecular probes to pathological assessment in breast cancer progression Macrophage Migration Inhibitory Factor Mediates PAR-Induced Bladder Pain.The interplay of extracellular matrix and microbiome in urothelial bladder cancer.VEGF receptors and neuropilins are expressed in the urothelial and neuronal cells in normal mouse urinary bladder and are upregulated in inflammation.The mast cell integrates the splanchnic and systemic inflammatory response in portal hypertension.Role of neurogenic inflammation in local communication in the visceral mucosa.

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P2860

Mandatory role of proteinase-activated receptor 1 in experimental bladder inflammation

http://www.wikidata.org/entity/Q33280660

description

2007 nî lūn-bûn

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2007 թուականի Մարտին հրատարակուած գիտական յօդուած

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2007 թվականի մարտին հրատարակված գիտական հոդված

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2007年の論文

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2007年論文

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2007年論文

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2007年論文

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2007年論文

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2007年論文

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2007年论文

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name

Mandatory role of proteinase-activated receptor 1 in experimental bladder inflammation

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Mandatory role of proteinase-activated receptor 1 in experimental bladder inflammation

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label

Mandatory role of proteinase-activated receptor 1 in experimental bladder inflammation

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Mandatory role of proteinase-activated receptor 1 in experimental bladder inflammation

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prefLabel

Mandatory role of proteinase-activated receptor 1 in experimental bladder inflammation

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Mandatory role of proteinase-activated receptor 1 in experimental bladder inflammation

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Mandatory role of proteinase-activated receptor 1 in experimental bladder inflammation

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Carole A Davis

http://www.w3.org/2001/XMLSchema#string

Cindy Simpson

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Claudia K Derian

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Igor Dozmorov

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Marcia R Saban

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Michael A Ihnat

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Michael R D'Andrea

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Patricia Andrade-Gordon

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Ricardo Saban

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P2860

Proteinase-activated receptors, targets for kallikrein signaling

Jab1, a novel protease-activated receptor-2 (PAR-2)-interacting protein, is involved in PAR-2-induced activation of activator protein-1

Activation of protease-activated receptor (PAR)-1, PAR-2, and PAR-4 stimulates IL-6, IL-8, and prostaglandin E2 release from human respiratory epithelial cells

Agonists of proteinase-activated receptor 1 induce plasma extravasation by a neurogenic mechanism

Cardiovascular responses mediated by protease-activated receptor-2 (PAR-2) and thrombin receptor (PAR-1) are distinguished in mice deficient in PAR-2 or PAR-1

Possible involvement of Ca2+-independent phospholipase A2 in protease-activated receptor-2-mediated contraction of rat urinary bladder

Mouse chromosome 17A3.3 contains 13 genes that encode functional tryptic-like serine proteases with distinct tissue and cell expression patterns

Delayed onset of inflammation in protease-activated receptor-2-deficient mice.

Proteinase-activated receptors: novel mechanisms of signaling by serine proteases.

Gene expression profiling of inflammatory bladder disorders.

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4

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scholarly article

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10.1186/1472-6793-7-4

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7

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Robert E. Hurst

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2007-03-30T00:00:00Z

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51380749

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1006705443

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