Normobaric hyperoxia improves cerebral blood flow and oxygenation, and inhibits peri-infarct depolarizations in experimental focal ischaemia. - Wikidata - awgldk - TriplyDB

Normobaric hyperoxia improves cerebral blood flow and oxygenation, and inhibits peri-infarct depolarizations in experimental focal ischaemia.

Normobaric hyperoxia improves cerebral blood flow and oxygenation, and inhibits peri-infarct depolarizations in experimental focal ischaemia.

http://www.wikidata.org/entity/Q33283163

about

Protection against focal ischemic injury to the brain by trans-sodium crocetinate. Laboratory investigation.Non-pharmaceutical therapies for stroke: mechanisms and clinical implications.Cocktail treatment, a promising strategy to treat acute cerebral ischemic stroke?Spreading Depression, Spreading Depolarizations, and the Cerebral Vasculature Distinct spatiotemporal patterns of spreading depolarizations during early infarct evolution: evidence from real-time imaging.Chronic imaging of cortical blood flow using Multi-Exposure Speckle Imaging Mild induced hypertension improves blood flow and oxygen metabolism in transient focal cerebral ischemia.Simultaneous imaging of cerebral partial pressure of oxygen and blood flow during functional activation and cortical spreading depression.Normobaric hyperoxia therapy for traumatic brain injury and stroke: a review.Normobaric hyperoxia combined with minocycline provides greater neuroprotection than either alone in transient focal cerebral ischemia.Combination therapy with normobaric oxygen (NBO) plus thrombolysis in experimental ischemic stroke.Laser speckle contrast imaging in biomedical optics.Cortical spreading depression-new insights and persistent questions.Laser speckle contrast imaging of cerebral blood flow.Endothelial dysfunction abrogates the efficacy of normobaric hyperoxia in stroke.In vivo monitoring of neuronal loss in traumatic brain injury: a microdialysis study.Advances in emerging nondrug therapies for acute stroke 2007.Inhibition of gp91(phox) contributes towards normobaric hyperoxia afforded neuroprotection in focal cerebral ischemia.Effects of normobaric oxygen on the progression of focal cerebral ischemia in rats Rho-kinase inhibition improves ischemic perfusion deficit in hyperlipidemic mice.Supply-demand mismatch transients in susceptible peri-infarct hot zones explain the origins of spreading injury depolarizations.Effects of normobaric oxygen and melatonin on reperfusion injury: role of cerebral microcirculation Does normobaric hyperoxia increase oxidative stress in acute ischemic stroke? A critical review of the literature.Evaluating effects of normobaric oxygen therapy in acute stroke with MRI-based predictive models.Supplemental oxygen delivery to suspected stroke patients in pre hospital and emergency department settings.Reduction of zinc accumulation in mitochondria contributes to decreased cerebral ischemic injury by normobaric hyperoxia treatment in an experimental stroke model.Dynamic functional cerebral blood volume responses to normobaric hyperoxia in acute ischemic stroke.Expanding applications, accuracy, and interpretation of laser speckle contrast imaging of cerebral blood flow.Pathophysiology of ischaemic stroke: insights from imaging, and implications for therapy and drug discovery.Experimental models, neurovascular mechanisms and translational issues in stroke research.Normobaric hyperoxia-based neuroprotective therapies in ischemic stroke.The impact of anesthetics and hyperoxia on cortical spreading depression.Hypothermia-induced neuroprotection is associated with reduced mitochondrial membrane permeability in a swine model of cardiac arrest.Normobaric hyperoxia inhibits NADPH oxidase-mediated matrix metalloproteinase-9 induction in cerebral microvessels in experimental stroke.Methylene blue and normobaric hyperoxia combination therapy in experimental ischemic stroke.Normobaric hyperoxia attenuates early blood-brain barrier disruption by inhibiting MMP-9-mediated occludin degradation in focal cerebral ischemia.Wound healing essentials: let there be oxygen Normobaric hyperoxia reduces the neurovascular complications associated with delayed tissue plasminogen activator treatment in a rat model of focal cerebral ischemia.Spreading depolarization: a possible new culprit in the delayed cerebral ischemia of subarachnoid hemorrhage.Influence of 100% and 40% oxygen on penumbral blood flow, oxygen level, and T2*-weighted MRI in a rat stroke model.

P2860

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P2860

Normobaric hyperoxia improves cerebral blood flow and oxygenation, and inhibits peri-infarct depolarizations in experimental focal ischaemia.

http://www.wikidata.org/entity/Q33283163

description

2007 nî lūn-bûn

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2007 թուականի Ապրիլին հրատարակուած գիտական յօդուած

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2007 թվականի ապրիլին հրատարակված գիտական հոդված

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2007年の論文

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Normobaric hyperoxia improves ...... experimental focal ischaemia.

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Normobaric hyperoxia improves ...... experimental focal ischaemia.

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Normobaric hyperoxia improves ...... experimental focal ischaemia.

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Normobaric hyperoxia improves ...... experimental focal ischaemia.

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Normobaric hyperoxia improves ...... experimental focal ischaemia.

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Normobaric hyperoxia improves ...... experimental focal ischaemia.

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Normobaric hyperoxia improves ...... experimental focal ischaemia.

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Normobaric hyperoxia improves ...... experimental focal ischaemia.

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Normobaric hyperoxia improves ...... experimental focal ischaemia.

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Andrew K Dunn

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Eng H Lo

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Hwa Kyoung Shin

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Michael A Moskowitz

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Phillip B Jones

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P2860

Dynamic imaging of cerebral blood flow using laser speckle.

Modulation of evoked cerebral blood flow under excessive blood supply and hyperoxic conditions.

Simultaneous imaging of total cerebral hemoglobin concentration, oxygenation, and blood flow during functional activation.

Products of hemolysis in the subarachnoid space inducing spreading ischemia in the cortex and focal necrosis in rats: a model for delayed ischemic neurological deficits after subarachnoid hemorrhage?

Laser Doppler, speckle and related techniques for blood perfusion mapping and imaging.

Laser speckle flowmetry for the study of cerebrovascular physiology in normal and ischemic mouse cortex.

Evaluation of laser speckle flowmetry for imaging cortical perfusion in experimental stroke studies: quantitation of perfusion and detection of peri-infarct depolarisations.

Eubaric hyperoxemia and experimental cerebral infarction.

Pulmonary and circulatory changes in conscious sheep exposed to 100% O2 at 1 ATA.

Hyperoxia and lung disease.

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scholarly article

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10.1093/BRAIN/AWM071

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Pt 6

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130

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17468117

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cerebral blood flow

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3023418

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