Epstein-Barr virus latency switch in human B-cells: a physico-chemical model. - Wikidata - awgldk - TriplyDB

Epstein-Barr virus latency switch in human B-cells: a physico-chemical model.

Epstein-Barr virus latency switch in human B-cells: a physico-chemical model.

http://www.wikidata.org/entity/Q33296308

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Diabetes, Epstein-Barr virus and extranodal natural killer/T-cell lymphoma in India: Unravelling the plausible nexus The B-cell specific transcription factor, Oct-2, promotes Epstein-Barr virus latency by inhibiting the viral immediate-early protein, BZLF1 Chromatin organization of gammaherpesvirus latent genomes.Cellular transcription factor Oct-1 interacts with the Epstein-Barr virus BRLF1 protein to promote disruption of viral latency Dynamics inside the cancer cell attractor reveal cell heterogeneity, limits of stability, and escape.Cooperative action in eukaryotic gene regulation: physical properties of a viral example.

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Epstein-Barr virus latency switch in human B-cells: a physico-chemical model.

http://www.wikidata.org/entity/Q33296308

description

2007 nî lūn-bûn

@nan

2007 թուականի Օգոստոսին հրատարակուած գիտական յօդուած

@hyw

2007 թվականի օգոստոսին հրատարակված գիտական հոդված

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2007年の論文

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2007年論文

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2007年論文

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2007年論文

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2007年論文

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2007年論文

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2007年论文

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name

Epstein-Barr virus latency switch in human B-cells: a physico-chemical model.

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Epstein-Barr virus latency switch in human B-cells: a physico-chemical model.

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Epstein-Barr virus latency switch in human B-cells: a physico-chemical model.

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type

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Epstein-Barr virus latency switch in human B-cells: a physico-chemical model.

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Epstein-Barr virus latency switch in human B-cells: a physico-chemical model.

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Epstein-Barr virus latency switch in human B-cells: a physico-chemical model.

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prefLabel

Epstein-Barr virus latency switch in human B-cells: a physico-chemical model.

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Epstein-Barr virus latency switch in human B-cells: a physico-chemical model.

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Epstein-Barr virus latency switch in human B-cells: a physico-chemical model.

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BMC Systems Biology

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Epstein-Barr virus latency switch in human B-cells: a physico-chemical model.

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Erik Aurell

http://www.w3.org/2001/XMLSchema#string

Ingemar Ernberg

http://www.w3.org/2001/XMLSchema#string

Jenny Almqvist

http://www.w3.org/2001/XMLSchema#string

Jiezhi Zou

http://www.w3.org/2001/XMLSchema#string

Maria Werner

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P2860

Interaction between the two ubiquitously expressed transcription factors NF-Y and Sp1

All Tcf HMG box transcription factors interact with Groucho-related co-repressors

DNA-dependent conversion of Oct-1 and Oct-2 into transcriptional repressors by Groucho/TLE

Reactivation of Epstein-Barr virus from latency

The molecular biology of the CCAAT-binding factor NF-Y

Epstein-Barr virus: 40 years on

In vivo dynamics of EBNA1-oriP interaction during latent and lytic replication of Epstein-Barr virus.

Redefining the Epstein-Barr virus-encoded nuclear antigen EBNA-1 gene promoter and transcription initiation site in group I Burkitt lymphoma cell lines

Repression of Epstein-Barr virus EBNA-1 gene transcription by pRb during restricted latency

EBNA-1: a protein pivotal to latent infection by Epstein-Barr virus.

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40

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10.1186/1752-0509-1-40

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1

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6074979

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17764547

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Epstein–Barr virus

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2164963

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