Microbial patterns signaling via Toll-like receptors 2 and 5 contribute to epithelial repair, growth and survival. - Wikidata - awgldk - TriplyDB

Microbial patterns signaling via Toll-like receptors 2 and 5 contribute to epithelial repair, growth and survival.

Microbial patterns signaling via Toll-like receptors 2 and 5 contribute to epithelial repair, growth and survival.

http://www.wikidata.org/entity/Q33312949

about

Identification of the cathelicidin peptide LL-37 as agonist for the type I insulin-like growth factor receptor TLR5 risk-associated haplotype for canine inflammatory bowel disease confers hyper-responsiveness to flagellin Flagellin-induced corneal antimicrobial peptide production and wound repair involve a novel NF-kappaB-independent and EGFR-dependent pathway.Multi-factorial modulation of colorectal carcinoma cells motility - partial coordination by the tetraspanin Co-029/tspan8 The role of toll-like receptor 4 in corneal epithelial wound healing Interactions of Candida albicans with host epithelial surfaces Gut microbial colonization orchestrates TLR2 expression, signaling and epithelial proliferation in the small intestinal mucosa Checks and balances: the ocular response to infection.Response of corneal epithelial cells to Staphylococcus aureus.Intestinal epithelial suppressor of cytokine signaling 3 enhances microbial-induced inflammatory tumor necrosis factor-α, contributing to epithelial barrier dysfunction.TLR2 expression is increased in rosacea and stimulates enhanced serine protease production by keratinocytes.β-Catenin and NF-κB co-activation triggered by TLR3 stimulation facilitates stem cell-like phenotypes in breast cancer Antimicrobial peptides and gut microbiota in homeostasis and pathology.Contribution of toll-like receptor 2 to the innate response against Staphylococcus aureus infection in mice.Ovarian cancer stem cells and inflammation Mucosal immunity and Candida albicans infection.Epithelial cell innate response to Candida albicans TLR5 expression in the small intestine depends on the adaptors MyD88 and TRIF, but is independent of the enteric microbiota.Staphylococcus aureus: an introduction The role of pattern recognition receptors in the innate recognition of Candida albicans.New approaches in the development of a vaccine for mucosal candidiasis: progress and challenges Multitasking basal cells: combining stem cell and innate immune duties.Immunopathogenesis of Staphylococcus aureus pulmonary infection.Alterations of splenic architecture in malaria are induced independently of Toll-like receptors 2, 4, and 9 or MyD88 and may affect antibody affinity Transforming growth factor β1 genotypes in relation to TGFβ1, interleukin-8, and tumor necrosis factor alpha in induced sputum and blood in cystic fibrosis.The immunobiology of cholangiocytes.Respiratory tract epithelial cells express retinaldehyde dehydrogenase ALDH1A and enhance IgA production by stimulated B cells in the presence of vitamin A.Wound repair at a glance.Lower nasopharyngeal epithelial cell repair and diminished innate inflammation responses contribute to the onset of acute otitis media in otitis-prone children.Innate immune recognition in infectious and noninfectious diseases of the lung.Endogenous toll-like receptor ligands and their biological significance.Innate immunity in the respiratory epithelium.The clinical implications of antitumor immunity in head and neck cancer.Helicobacter pylori-Induced Signaling Pathways Contribute to Intestinal Metaplasia and Gastric Carcinogenesis.Toll-Like Receptors and Tissue Remodeling: The Pro/Cons Recent Findings.STAT3: a critical component in the response to Helicobacter pylori infection.Airway structural cells regulate TLR5-mediated mucosal adjuvant activity.Immunohistochemical detection of Helicobacter pylori without association of TLR5 expression in oral squamous cell carcinoma.Neisseria gonorrhoeae breaches the apical junction of polarized epithelial cells for transmigration by activating EGFR [Microbiota and probiotics: effects on human health].

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P2860

Microbial patterns signaling via Toll-like receptors 2 and 5 contribute to epithelial repair, growth and survival.

http://www.wikidata.org/entity/Q33312949

description

2008 nî lūn-bûn

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2008 թուականի Յունուարին հրատարակուած գիտական յօդուած

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2008 թվականի հունվարին հրատարակված գիտական հոդված

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2008年の論文

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2008年論文

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Microbial patterns signaling v ...... l repair, growth and survival.

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Microbial patterns signaling v ...... l repair, growth and survival.

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Jürgen Behr

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Renat Shaykhiev

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Robert Bals

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P2860

Cutting edge: recognition of Gram-positive bacterial cell wall components by the innate immune system occurs via Toll-like receptor 2

Segregation of receptor and ligand regulates activation of epithelial growth factor receptor

Recognition of Staphylococcus aureus by the innate immune system.

Innate immune recognition

Pathogen recognition and innate immunity

Staphylococcus aureus infections

The innate immune response to bacterial flagellin is mediated by Toll-like receptor 5

Platelet-activating factor receptor and ADAM10 mediate responses to Staphylococcus aureus in epithelial cells

IL-8 promotes cell proliferation and migration through metalloproteinase-cleavage proHB-EGF in human colon carcinoma cells

Toll-like receptor 2 enhances ZO-1-associated intestinal epithelial barrier integrity via protein kinase C

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