Striatal proteomic analysis suggests that first L-dopa dose equates to chronic exposure.
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Morphological changes of glutamatergic synapses in animal models of Parkinson's diseaseNeuroanatomical study of the A11 diencephalospinal pathway in the non-human primateDifferential structural plasticity of corticostriatal and thalamostriatal axo-spinous synapses in MPTP-treated Parkinsonian monkeys.L-DOPA-induced dyskinesia is associated with regional increase of striatal dynorphin peptides as elucidated by imaging mass spectrometry.Priming for l-dopa-induced dyskinesia in Parkinson's disease: a feature inherent to the treatment or the disease?Basal Ganglia circuits underlying the pathophysiology of levodopa-induced dyskinesia.Astrocytosis in parkinsonism: considering tripartite striatal synapses in physiopathology?Striatal spine plasticity in Parkinson's diseaseSubthalamic nucleus high-frequency stimulation restores altered electrophysiological properties of cortical neurons in parkinsonian ratNeuroglial plasticity at striatal glutamatergic synapses in Parkinson's diseaseSerotonin hyperinnervation and upregulated 5-HT2A receptor expression and motor-stimulating function in nigrostriatal dopamine-deficient Pitx3 mutant mice.Differential striatal spine pathology in Parkinson's disease and cocaine addiction: a key role of dopamine?Role of movement in long-term basal ganglia changes: implications for abnormal motor responses.Exploiting the potential of molecular profiling in Parkinson's disease: current practice and future probabilities.Anatomo-proteomic characterization of human basal ganglia: focus on striatum and globus pallidus.Loss and remodeling of striatal dendritic spines in Parkinson's disease: from homeostasis to maladaptive plasticity?Chronic but not acute dopaminergic transmission interruption promotes a progressive increase in cortical beta frequency synchronization: relationships to vigilance state and akinesia.Exercise-Induced Neuroprotection of the Nigrostriatal Dopamine System in Parkinson's Disease.Levodopa treatment and dendritic spine pathology.Proteomic analysis of striatum from MPTP-treated marmosets (Callithrix jacchus) with L-DOPA-induced dyskinesia of differing severity.Presynaptic dopamine depletion determines the timing of levodopa-induced dyskinesia onset in Parkinson's disease.
P2860
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P2860
Striatal proteomic analysis suggests that first L-dopa dose equates to chronic exposure.
description
2008 nî lūn-bûn
@nan
2008 թուականի Փետրուարին հրատարակուած գիտական յօդուած
@hyw
2008 թվականի փետրվարին հրատարակված գիտական հոդված
@hy
2008年の論文
@ja
2008年論文
@yue
2008年論文
@zh-hant
2008年論文
@zh-hk
2008年論文
@zh-mo
2008年論文
@zh-tw
2008年论文
@wuu
name
Striatal proteomic analysis suggests that first L-dopa dose equates to chronic exposure.
@ast
Striatal proteomic analysis suggests that first L-dopa dose equates to chronic exposure.
@en
Striatal proteomic analysis suggests that first L-dopa dose equates to chronic exposure.
@nl
type
label
Striatal proteomic analysis suggests that first L-dopa dose equates to chronic exposure.
@ast
Striatal proteomic analysis suggests that first L-dopa dose equates to chronic exposure.
@en
Striatal proteomic analysis suggests that first L-dopa dose equates to chronic exposure.
@nl
prefLabel
Striatal proteomic analysis suggests that first L-dopa dose equates to chronic exposure.
@ast
Striatal proteomic analysis suggests that first L-dopa dose equates to chronic exposure.
@en
Striatal proteomic analysis suggests that first L-dopa dose equates to chronic exposure.
@nl
P2093
P2860
P50
P1433
P1476
Striatal proteomic analysis suggests that first L-dopa dose equates to chronic exposure
@en
P2093
Alan R Crossman
Céline Guigoni
Karl Sköld
Marcus Svensson
Maria Fälth
Per E Andrén
P2860
P356
10.1371/JOURNAL.PONE.0001589
P407
P577
2008-02-13T00:00:00Z