Overexpression of UCP2 protects thalamic neurons following global ischemia in the mouse. - Wikidata - awgldk - TriplyDB

Overexpression of UCP2 protects thalamic neurons following global ischemia in the mouse.

Overexpression of UCP2 protects thalamic neurons following global ischemia in the mouse.

http://www.wikidata.org/entity/Q33321139

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Amitriptyline improves motor function via enhanced neurotrophin signaling and mitochondrial functions in the murine N171-82Q Huntington disease model.UCP2 induced by natural birth regulates neuronal differentiation of the hippocampus and related adult behavior Resveratrol pretreatment protects rat brain from cerebral ischemic damage via a sirtuin 1-uncoupling protein 2 pathway Traditional Chinese medicine for modern treatment of Parkinson's disease.Both ischemic preconditioning and ghrelin administration protect hippocampus from ischemia/reperfusion and upregulate uncoupling protein-2 Mitochondrial Uncoupler Prodrug of 2,4-Dinitrophenol, MP201, Prevents Neuronal Damage and Preserves Vision in Experimental Optic Neuritis Potential therapeutic benefits of strategies directed to mitochondria.Deletion of mitochondrial uncoupling protein-2 increases ischemic brain damage after transient focal ischemia by altering gene expression patterns and enhancing inflammatory cytokines Mitochondrial approaches to protect against cardiac ischemia and reperfusion injury.Roles of oxidative stress, apoptosis, PGC-1α and mitochondrial biogenesis in cerebral ischemia.Peroxisome proliferator-activated receptors γ/mitochondrial uncoupling protein 2 signaling protects against seizure-induced neuronal cell death in the hippocampus following experimental status epilepticus.Sex-dependent mitochondrial respiratory impairment and oxidative stress in a rat model of neonatal hypoxic-ischemic encephalopathy.TIMP-3 and MMP-3 contribute to delayed inflammation and hippocampal neuronal death following global ischemia.Neuroprotective role of mitochondrial uncoupling protein 2 in cerebral stroke.UCP2 overexpression worsens mitochondrial dysfunction and accelerates disease progression in a mouse model of amyotrophic lateral sclerosis.Forced treadmill exercise can induce stress and increase neuronal damage in a mouse model of global cerebral ischemia.Signaling pathways leading to ischemic mitochondrial neuroprotection.Mitochondrial ROS signaling in organismal homeostasis.Protective effect of hydroxytyrosol in arsenic-induced mitochondrial dysfunction in rat brain.Sirt1: Role Under the Condition of Ischemia/Hypoxia.Mitochondrial Uncoupling Protein 2 (UCP2) Regulates Retinal Ganglion Cell Number and Survival.Uncoupling Protein, UCP-4 May Be Involved in Neuronal Defects During Aging and Resistance to Pathogens in Caenorhabditis elegans.Biochemical and Molecular Alterations Following Arsenic-Induced Oxidative Stress and Mitochondrial Dysfunction in Rat Brain.Putative roles of Ca(2+) -independent phospholipase A2 in respiratory chain-associated ROS production in brain mitochondria: influence of docosahexaenoic acid and bromoenol lactone.Endotoxemia induces lung-brain coupling and multi-organ injury following cerebral ischemia-reperfusion.Diverse roles of mitochondria in ischemic stroke.Protective Role of UCP2 in Oxidative Stress and Apoptosis during the Silent Phase of an Experimental Model of Epilepsy Induced by Pilocarpine

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P2860

Overexpression of UCP2 protects thalamic neurons following global ischemia in the mouse.

http://www.wikidata.org/entity/Q33321139

description

2008 nî lūn-bûn

@nan

2008 թուականի Փետրուարին հրատարակուած գիտական յօդուած

@hyw

2008 թվականի փետրվարին հրատարակված գիտական հոդված

@hy

2008年の論文

@ja

2008年学术文章

@wuu

2008年学术文章

@zh-cn

2008年学术文章

@zh-hans

2008年学术文章

@zh-my

2008年学术文章

@zh-sg

2008年學術文章

@yue

name

Overexpression of UCP2 protects thalamic neurons following global ischemia in the mouse.

@ast

Overexpression of UCP2 protects thalamic neurons following global ischemia in the mouse.

@en

Overexpression of UCP2 protects thalamic neurons following global ischemia in the mouse.

@nl

type

label

Overexpression of UCP2 protects thalamic neurons following global ischemia in the mouse.

@ast

Overexpression of UCP2 protects thalamic neurons following global ischemia in the mouse.

@en

Overexpression of UCP2 protects thalamic neurons following global ischemia in the mouse.

@nl

prefLabel

Overexpression of UCP2 protects thalamic neurons following global ischemia in the mouse.

@ast

Overexpression of UCP2 protects thalamic neurons following global ischemia in the mouse.

@en

Overexpression of UCP2 protects thalamic neurons following global ischemia in the mouse.

@nl

P1433

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P1433

Journal of Cerebral Blood Flow & Metabolism

P1476

Overexpression of UCP2 protects thalamic neurons following global ischemia in the mouse.

@en

P2093

Craig H Warden

http://www.w3.org/2001/XMLSchema#string

Gustav Mattiasson

http://www.w3.org/2001/XMLSchema#string

Sabrina Diano

http://www.w3.org/2001/XMLSchema#string

Tamas L Horvath

http://www.w3.org/2001/XMLSchema#string

Tomas Deierborg

http://www.w3.org/2001/XMLSchema#string

P2860

Persistent nuclear factor-kappa B activation in Ucp2-/- mice leads to enhanced nitric oxide and inflammatory cytokine production

Uncoupling proteins 2 and 3 are fundamental for mitochondrial Ca2+ uniport

Deletion of the adenosine A1 receptor gene does not alter neuronal damage following ischaemia in vivo or in vitro.

Uncoupling protein-2 prevents neuronal death and diminishes brain dysfunction after stroke and brain trauma.

Brain damage in a mouse model of global cerebral ischemia. Effect of NMDA receptor blockade.

Ischemic preconditioning and brain tolerance: temporal histological and functional outcomes, protein synthesis requirement, and interleukin-1 receptor antagonist and early gene expression.

Lack of neuroprotection by heat shock protein 70 overexpression in a mouse model of global cerebral ischemia.

Gene deletion of cystatin C aggravates brain damage following focal ischemia but mitigates the neuronal injury after global ischemia in the mouse.

Expression modification of uncoupling proteins and MnSOD in retinal endothelial cells and pericytes induced by high glucose: the role of reactive oxygen species in diabetic retinopathy.

Mitochondrial uncoupling protein-2 protects the immature brain from excitotoxic neuronal death.

P304

1186-1195

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scholarly article

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10.1038/JCBFM.2008.8

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6

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28

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Tadeusz Wieloch

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2008-02-27T00:00:00Z

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5551812

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18301432

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2642535

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