Comprehensive transcriptional profiling of prion infection in mouse models reveals networks of responsive genes - Wikidata - awgldk - TriplyDB

Comprehensive transcriptional profiling of prion infection in mouse models reveals networks of responsive genes

Comprehensive transcriptional profiling of prion infection in mouse models reveals networks of responsive genes

http://www.wikidata.org/entity/Q33322227

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A miRNA signature of prion induced neurodegeneration A systems approach to prion disease Regulation of GABA(A) and glutamate receptor expression, synaptic facilitation and long-term potentiation in the hippocampus of prion mutant mice Early mechanisms of pathobiology are revealed by transcriptional temporal dynamics in hippocampal CA1 neurons of prion infected mice Uncovering molecular biomarkers that correlate cognitive decline with the changes of hippocampus' gene expression profiles in Alzheimer's disease.Brain transcriptional stability upon prion protein-encoding gene invalidation in zygotic or adult mouse A 2cM genome-wide scan of European Holstein cattle affected by classical BSE.Application of "omics" to prion biomarker discovery.Early embryonic gene expression profiling of zebrafish prion protein (Prp2) morphants Gene expression profiling of brains from bovine spongiform encephalopathy (BSE)-infected cynomolgus macaques.Transcriptome analysis of the medulla tissue from cattle in response to bovine spongiform encephalopathy using digital gene expression tag profiling.Transcriptomic analysis brings new insight into the biological role of the prion protein during mouse embryogenesis Biological network inferences for a protection mechanism against familial Creutzfeldt-Jakob disease with E200K pathogenic mutation.Identification and structural analysis of C-terminally truncated collapsin response mediator protein-2 in a murine model of prion diseases.Gene regulatory network analysis supports inflammation as a key neurodegeneration process in prion disease.Proteomics analysis of amyloid and nonamyloid prion disease phenotypes reveals both common and divergent mechanisms of neuropathogenesis Development of kinomic analyses to identify dysregulated signaling pathways in cells expressing cytoplasmic PrP.Prion infection of mouse brain reveals multiple new upregulated genes involved in neuroinflammation or signal transduction.Expression of genes involved in the T cell signalling pathway in circulating immune cells of cattle 24 months following oral challenge with Bovine Amyloidotic Spongiform Encephalopathy (BASE).Identification of Major Signaling Pathways in Prion Disease Progression Using Network Analysis.Transcriptome Analysis Reveals Regulation of Gene Expression for Lipid Catabolism in Young Broilers by Butyrate Glycerides.Gene expression alterations in Rocky Mountain elk infected with chronic wasting disease.Distribution of Misfolded Prion Protein Seeding Activity Alone Does Not Predict Regions of Neurodegeneration.Transcriptomic Determinants of Scrapie Prion Propagation in Cultured Ovine Microglia.Differential molecular chaperone response associated with various mouse adapted scrapie strains.Identification and removal of proteins that co-purify with infectious prion protein improves the analysis of its secondary structure.Comparative profiling of highly enriched 22L and Chandler mouse scrapie prion protein preparations.Searching for MIND: microRNAs in neurodegenerative diseases.The Prion Disease Database: a comprehensive transcriptome resource for systems biology research in prion diseases.Proteomic profiling of cerebrospinal fluid in Creutzfeldt-Jakob disease.Gene expression profiling to identify druggable targets in prion diseases.Proteomic analysis of prion diseases: creating clarity or causing confusion?Prion disease and the innate immune system.Microdissection and transcriptional profiling: a window into the pathobiology of preclinical prion disease.Gene expression in the medulla following oral infection of cattle with bovine spongiform encephalopathy.microRNAs with different functions and roles in disease development and as potential biomarkers of diabetes: progress and challenges.Proteomics applications in prion biology and structure.Central nervous system gene expression changes in a transgenic mouse model for bovine spongiform encephalopathy.Functional genomics approach for identification of molecular processes underlying neurodegenerative disorders in prion diseases.Unraveling the neuroprotective mechanisms of PrP (C) in excitotoxicity.

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Comprehensive transcriptional profiling of prion infection in mouse models reveals networks of responsive genes

http://www.wikidata.org/entity/Q33322227

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Clark Phillipson

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Debra Parchaliuk

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P2860

Convergence of genes implicated in Alzheimer's disease on the cerebral cholesterol shuttle: APP, cholesterol, lipoproteins, and atherosclerosis

Induction of heparin-binding growth-associated molecule expression in reactive astrocytes following hippocampal neuronal injury

Apoptosis, axonal growth defects, and degeneration of peripheral neurons in mice lacking CREB

Gene expression alterations in brains of mice infected with three strains of scrapie

A comparative genomics approach to identifying the plasticity transcriptome.

Protein kinase C isozymes and the regulation of diverse cell responses.

Disruption of CREB function in brain leads to neurodegeneration.

Depleting neuronal PrP in prion infection prevents disease and reverses spongiosis.

The pleiotropin/midkine family of cytokines: role in glial-neuronal signalling.

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