Appetite enhancement and weight gain by peripheral administration of TrkB agonists in non-human primates. - Wikidata - awgldk - TriplyDB

Appetite enhancement and weight gain by peripheral administration of TrkB agonists in non-human primates.

Appetite enhancement and weight gain by peripheral administration of TrkB agonists in non-human primates.

http://www.wikidata.org/entity/Q33326631

about

Understanding the control of ingestive behavior in primates Agonistic TAM-163 antibody targeting tyrosine kinase receptor-B: applying mechanistic modeling to enable preclinical to clinical translation and guide clinical trial design Sustained blockade of neurotrophin receptors TrkA, TrkB and TrkC reduces non-malignant skeletal pain but not the maintenance of sensory and sympathetic nerve fibers.Activation of TrkB with TAM-163 results in opposite effects on body weight in rodents and non-human primates.A monoclonal antibody TrkB receptor agonist as a potential therapeutic for Huntington's disease.The lighter side of BDNF.Molecular and neural bases underlying roles of BDNF in the control of body weight.Neurotrophic factor control of satiety and body weight.The skinny on brain-derived neurotrophic factor: evidence from animal models to GWAS.The central question of type 2 diabetes.Safety and Antitumor Activity of the Multitargeted Pan-TRK, ROS1, and ALK Inhibitor Entrectinib: Combined Results from Two Phase I Trials (ALKA-372-001 and STARTRK-1).Agonistic effect of polyunsaturated fatty acids (PUFAs) and its metabolites on brain-derived neurotrophic factor (BDNF) through molecular docking simulation.Use and Importance of Nonhuman Primates in Metabolic Disease Research: Current State of the Field.Molecular Therapy of Melanocortin-4-Receptor Obesity by an Autoregulatory BDNF Vector.Modulating Neurotrophin Receptor Signaling as a Therapeutic Strategy for Huntington's Disease.Glucose concentrations modulate brain-derived neurotrophic factor responsiveness of neurones in the paraventricular nucleus of the hypothalamus.Relationships between components of physical activity, cardiorespiratory fitness, cardiac autonomic health, and brain-derived neurotrophic factor.Hydrogel-delivered brain-derived neurotrophic factor promotes tissue repair and recovery after stroke.SORCS1 and SORCS3 control energy balance and orexigenic peptide production.

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P2860

Appetite enhancement and weight gain by peripheral administration of TrkB agonists in non-human primates.

http://www.wikidata.org/entity/Q33326631

description

2008 nî lūn-bûn

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2008 թուականի Ապրիլին հրատարակուած գիտական յօդուած

@hyw

2008 թվականի ապրիլին հրատարակված գիտական հոդված

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2008年の論文

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2008年論文

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2008年論文

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2008年論文

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2008年論文

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2008年論文

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2008年论文

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Appetite enhancement and weigh ...... gonists in non-human primates.

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Appetite enhancement and weigh ...... gonists in non-human primates.

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Appetite enhancement and weigh ...... gonists in non-human primates.

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Appetite enhancement and weigh ...... agonists in non-human primates

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Alison Forgie

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Anthony G Comuzzie

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Arnon Rosenthal

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Jeanette Dilley

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Jennifer Stratton

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P2860

A de novo mutation affecting human TrkB associated with severe obesity and developmental delay

Evolutionary studies of the nerve growth factor family reveal a novel member abundantly expressed in Xenopus ovary

Similarities and differences in the way neurotrophins interact with the Trk receptors in neuronal and nonneuronal cells

Brain-derived neurotrophic factor regulates energy balance downstream of melanocortin-4 receptor

Decreased cerebrospinal-fluid/serum leptin ratio in obesity: a possible mechanism for leptin resistance.

BDNF regulates eating behavior and locomotor activity in mice

Hyperphagia, severe obesity, impaired cognitive function, and hyperactivity associated with functional loss of one copy of the brain-derived neurotrophic factor (BDNF) gene.

Brain-derived neurotrophic factor-deficient mice develop aggressiveness and hyperphagia in conjunction with brain serotonergic abnormalities

Cerebrospinal fluid leptin levels: relationship to plasma levels and to adiposity in humans.

The Trk family of neurotrophin receptors.

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