Vav3 oncogene activates estrogen receptor and its overexpression may be involved in human breast cancer.
about
Inhibition of the Rho GTPase, Rac1, decreases estrogen receptor levels and is a novel therapeutic strategy in breast cancer.The use of knockout mice reveals a synergistic role of the Vav1 and Rasgrf2 gene deficiencies in lymphomagenesis and metastasisTherapeutic sensitivity to Rac GTPase inhibition requires consequential suppression of mTORC1, AKT, and MEK signaling in breast cancerPrediction of epigenetically regulated genes in breast cancer cell linesMinireview: Tipping the balance: ligand-independent activation of steroid receptors.The use of ultra-dense array CGH analysis for the discovery of micro-copy number alterations and gene fusions in the cancer genomeVAV3 mediates resistance to breast cancer endocrine therapy.Widespread alternative exon usage in clinically distinct subtypes of Invasive Ductal CarcinomaHigh nuclear level of Vav1 is a positive prognostic factor in early invasive breast tumors: a role in modulating genes related to the efficiency of metastatic processPCA3 noncoding RNA is involved in the control of prostate-cancer cell survival and modulates androgen receptor signaling.Identification of the Rac-GEF P-Rex1 as an essential mediator of ErbB signaling in breast cancer.VAV3 oncogene expression in colorectal cancer: clinical aspects and functional characterization.Genome-wide association study of susceptibility loci for breast cancer in Sardinian population.VAV3 Overexpressed in Cancer Stem Cells Is a Poor Prognostic Indicator in Ovarian Cancer PatientsRac signaling in breast cancer: a tale of GEFs and GAPs.PH motifs in PAR1&2 endow breast cancer growth.Overexpression of miR-499-5p inhibits non-small cell lung cancer proliferation and metastasis by targeting VAV3.Targeted estrogen delivery reverses the metabolic syndrome.How integrins control breast biology.The treatment of breast cancer using liposome technology.Inhibition of Vav3 gene can promote apoptosis of human gastric cancer cell line MGC803 by regulating ERK pathway.Inhibition of Vav3 could reverse the drug resistance of gastric cancer cells by downregulating JNK signaling pathway.Inhibition of gastric cancer cell growth and invasion through siRNA-mediated knockdown of guanine nucleotide exchange factor Vav3.Gankyrin promotes breast cancer cell metastasis by regulating Rac1 activity.Suppression of SPIN1-mediated PI3K-Akt pathway by miR-489 increases chemosensitivity in breast cancer.Truncated isoform Vav3.1 is highly expressed in ovarian cancer stem cells and clinically relevant in predicting prognosis and platinum-response.Expression of VAV1 in the tumour microenvironment of glioblastoma multiforme.The clinical value of Vav3 in peripheral blood for predicting lymphatic metastasis of gastric cancer.Evaluation of Vav3.1 as prognostic marker in endometrial cancer
P2860
Q30419413-9371FB35-4538-4A5B-B80E-0CBAF42096BAQ33518516-B26DE646-3D3C-4CF2-B73E-4CF021941182Q33591690-FB3C3D3B-137F-4AD4-9996-1173F194A4E6Q33595315-D6031849-2728-4930-A856-2102CAB75EE1Q33631775-B552BA5C-EEE1-4F68-8109-CB85CA3343D6Q33805565-443C5695-4B1D-44CC-805D-DD776AB472D5Q33827037-E0BF677B-2B19-472C-B7DE-7567E35ABE3AQ33911898-B944226C-A020-4C83-BB3C-40DDF1CFCA4BQ34100951-417057F3-6CAE-4D8B-B1AE-12D000BFAC7DQ34468270-50C740F7-56B2-470E-AC8B-BD63AD3EC6E0Q34571293-D162725C-2BAE-4AE7-8C2F-4D0F66ECA730Q35198158-BEC9F850-640A-4E6D-AEEB-DBC443A46D61Q35615396-D9CE8C23-593A-43A4-8AC0-665BB0C324EAQ35844366-3C316EE5-E060-4FA4-978D-7F14BE50E8EEQ35854151-D13364C6-7DF5-4946-8370-10D445AB2545Q36357012-53FCD2CA-6E4F-4864-A911-19E75138AA91Q36682155-D122BE0B-0E52-402C-B63F-AB564D7692DBQ37134063-4555EECD-8CFF-4D34-B333-E90096CDE6ECQ37254238-FF0D2213-03D8-4A01-B83A-8D2C9A56E188Q38002854-61B14A06-8A21-4C70-916B-27BB33934E3DQ38809679-F2D436A0-3A9E-464E-87FD-606DDB7225E6Q38937276-83AD4BF1-2E80-420E-B120-FA31CFAC0D26Q39090736-A42662A4-DD23-404D-A91A-C81FC9DAE134Q39298025-BCE80F3D-FEC3-44B3-80DF-DE48FD289471Q39772391-1261D574-A670-4FD9-8C85-FEAA7E7B63B4Q48197803-0BEC0A01-5ED1-40D5-8A67-50849B96BF44Q48419123-5543456C-F3AF-4528-8FF6-7C98D453BB08Q50997022-5F39A488-5E1B-4490-9A6B-2F13F9A1B2ABQ57141071-DA309C2A-DFB7-4C8C-8AA6-26DE8A84B005
P2860
Vav3 oncogene activates estrogen receptor and its overexpression may be involved in human breast cancer.
description
2008 nî lūn-bûn
@nan
2008 թուականի Յունիսին հրատարակուած գիտական յօդուած
@hyw
2008 թվականի հունիսին հրատարակված գիտական հոդված
@hy
2008年の論文
@ja
2008年論文
@yue
2008年論文
@zh-hant
2008年論文
@zh-hk
2008年論文
@zh-mo
2008年論文
@zh-tw
2008年论文
@wuu
name
Vav3 oncogene activates estrog ...... volved in human breast cancer.
@ast
Vav3 oncogene activates estrog ...... volved in human breast cancer.
@en
Vav3 oncogene activates estrog ...... volved in human breast cancer.
@nl
type
label
Vav3 oncogene activates estrog ...... volved in human breast cancer.
@ast
Vav3 oncogene activates estrog ...... volved in human breast cancer.
@en
Vav3 oncogene activates estrog ...... volved in human breast cancer.
@nl
prefLabel
Vav3 oncogene activates estrog ...... volved in human breast cancer.
@ast
Vav3 oncogene activates estrog ...... volved in human breast cancer.
@en
Vav3 oncogene activates estrog ...... volved in human breast cancer.
@nl
P2093
P2860
P356
P1433
P1476
Vav3 oncogene activates estrog ...... volved in human breast cancer.
@en
P2093
P2860
P2888
P356
10.1186/1471-2407-8-158
P407
P577
2008-06-02T00:00:00Z
P5875
P6179
1047118532