Colony-stimulating factor-1-dependent macrophages are responsible for IVIG protection in antibody-induced autoimmune disease.
about
Immune complexes as therapy for autoimmunityMonoclonal antibodies capable of discriminating the human inhibitory Fcgamma-receptor IIB (CD32B) from the activating Fcgamma-receptor IIA (CD32A): biochemical, biological and functional characterizationThe antiinflammatory activity of IgG: the intravenous IgG paradoxThe role of the complement and the Fc gamma R system in the pathogenesis of arthritisK/BxN Serum-Transfer Arthritis as a Model for Human Inflammatory ArthritisGlycans in the immune system and The Altered Glycan Theory of Autoimmunity: a critical reviewTargeting the Fc receptor in autoimmune diseaseCrystal Structure of Fc Receptor I and Its Implication in High Affinity -Immunoglobulin BindingToll-like receptor-mediated IRE1α activation as a therapeutic target for inflammatory arthritisNeutrophils in a mouse model of autoantibody-mediated arthritis: critical producers of Fc receptor gamma, the receptor for C5a, and lymphocyte function-associated antigen 1Different glycosylation pattern of human IgG1 and IgG3 antibodies isolated from transiently as well as permanently transfected cell lines.Intravenous immunoglobulins modulate neutrophil activation and vascular injury through FcγRIII and SHP-1.Tetramolecular immune complexes are more efficient than IVIg to prevent antibody-dependent in vitro and in vivo phagocytosis of blood cells.Intravenous immunoglobulin ameliorates ITP via activating Fc gamma receptors on dendritic cells.Absence of cytokine modulation following therapeutic infusion of intravenous immunoglobulin or anti-red blood cell antibodies in a mouse model of immune thrombocytopenic purpura.Recapitulation of IVIG anti-inflammatory activity with a recombinant IgG Fc.Mechanisms of action of intravenous immunoglobulins.IVIg-mediated amelioration of ITP in mice is dependent on sialic acid and SIGNR1.B cells and CD22 are dispensable for the immediate antiinflammatory activity of intravenous immunoglobulins in vivo.High-dose methylprednisolone pulse therapy upregulated FcγRIIb expression on B cells in primary Sjögren's syndrome patients with thrombocytopenia.CD44 antibody-mediated amelioration of murine immune thrombocytopenia (ITP): mouse background determines the effect of FcγRIIb genetic disruption.Immunoglobulin G galactosylation and sialylation are associated with pregnancy-induced improvement of rheumatoid arthritis and the postpartum flare: results from a large prospective cohort studyPassively administered pooled human immunoglobulins exert IL-10 dependent anti-inflammatory effects that protect against fatal HSV encephalitis.Therapeutic efficacy of high-dose intravenous immunoglobulin in Mycobacterium tuberculosis infection in miceAnti-inflammatory IgG production requires functional P1 promoter in β-galactoside α2,6-sialyltransferase 1 (ST6Gal-1) geneGenetic deficiency of Syk protects mice from autoantibody-induced arthritis.Essential role of NK cells in IgG therapy for experimental autoimmune encephalomyelitisA novel anti-inflammatory role for secretory phospholipase A2 in immune complex-mediated arthritis.FcgammaRIII engagement provides activating signals to NKT cells in antibody-induced joint inflammation.Regulation of human neutrophil Fcγ receptor IIa by C5a receptor promotes inflammatory arthritis in mice.Emerging antibody products and Nicotiana manufacturing.Platelets participate in synovitis via Cox-1-dependent synthesis of prostacyclin independently of microparticle generation.Gab2 promotes colony-stimulating factor 1-regulated macrophage expansion via alternate effectors at different stages of development.Multiple mechanisms of immune suppression by B lymphocytesMast cells contribute to initiation of autoantibody-mediated arthritis via IL-1Deregulated Fcγ receptor expression in patients with CIDP.Fc receptor targeting in the treatment of allergy, autoimmune diseases and cancer.B cells as a therapeutic target in autoimmune diseases.Antibody isotype-specific engagement of Fcgamma receptors regulates B lymphocyte depletion during CD20 immunotherapy.Pathology and protection in nephrotoxic nephritis is determined by selective engagement of specific Fc receptors.
P2860
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P2860
Colony-stimulating factor-1-dependent macrophages are responsible for IVIG protection in antibody-induced autoimmune disease.
description
2003 nî lūn-bûn
@nan
2003 թուականի Ապրիլին հրատարակուած գիտական յօդուած
@hyw
2003 թվականի ապրիլին հրատարակված գիտական հոդված
@hy
2003年の論文
@ja
2003年学术文章
@wuu
2003年学术文章
@zh-cn
2003年学术文章
@zh-hans
2003年学术文章
@zh-my
2003年学术文章
@zh-sg
2003年學術文章
@yue
name
Colony-stimulating factor-1-de ...... dy-induced autoimmune disease.
@ast
Colony-stimulating factor-1-de ...... dy-induced autoimmune disease.
@en
type
label
Colony-stimulating factor-1-de ...... dy-induced autoimmune disease.
@ast
Colony-stimulating factor-1-de ...... dy-induced autoimmune disease.
@en
prefLabel
Colony-stimulating factor-1-de ...... dy-induced autoimmune disease.
@ast
Colony-stimulating factor-1-de ...... dy-induced autoimmune disease.
@en
P2093
P1433
P1476
Colony-stimulating factor-1-de ...... dy-induced autoimmune disease.
@en
P2093
Astrid Samuelsson
Jeffrey V Ravetch
Jeffrey W Pollard
P304
P356
10.1016/S1074-7613(03)00080-3
P407
P577
2003-04-01T00:00:00Z