Intestinal bacteria condition dendritic cells to promote IgA production - Wikidata - awgldk - TriplyDB

Intestinal bacteria condition dendritic cells to promote IgA production

Intestinal bacteria condition dendritic cells to promote IgA production

http://www.wikidata.org/entity/Q33348616

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Intestinal antigen-presenting cells in mucosal immune homeostasis: crosstalk between dendritic cells, macrophages and B-cells The short isoform of the CEACAM1 receptor in intestinal T cells regulates mucosal immunity and homeostasis via Tfh cell induction Host response to probiotics determined by nutritional status of rotavirus-infected neonatal mice.Regulatory roles of the tumor necrosis factor receptor BCMA.Lactobacillus gasseri SBT2055 induces TGF-β expression in dendritic cells and activates TLR2 signal to produce IgA in the small intestine.M cell targeting by a Claudin 4 targeting peptide can enhance mucosal IgA responses Both systemic and mucosal LCMV immunization generate robust viral-specific IgG in mucosal secretions, but elicit poor LCMV-specific IgA.The mucosal adjuvant cholera toxin B instructs non-mucosal dendritic cells to promote IgA production via retinoic acid and TGF-β.Neural networks in intestinal immunoregulation Innate control of B cell responses.The microbiome and regulation of mucosal immunity.Host-microbial interactions and regulation of intestinal epithelial barrier function: From physiology to pathology.Vitamin A Deficiency Impairs Mucin Expression and Suppresses the Mucosal Immune Function of the Respiratory Tract in Chicks.Parasite Proximity Drives the Expansion of Regulatory T Cells in Peyer's Patches following Intestinal Helminth Infection.Epithelial expression of the cytosolic retinoid chaperone cellular retinol binding protein II is essential for in vivo imprinting of local gut dendritic cells by lumenal retinoids Lactobacilli and Bifidobacteria enhance mucosal B cell responses and differentially modulate systemic antibody responses to an oral human rotavirus vaccine in a neonatal gnotobiotic pig disease model.Comparison of probiotic lactobacilli and bifidobacteria effects, immune responses and rotavirus vaccines and infection in different host species Peyer's patches: organizing B-cell responses at the intestinal frontier Host and microbial factors in regulation of T cells in the intestine.Mucosal co-immunization with AIM2 enhances protective SIgA response and increases prophylactic efficacy of chitosan-DNA vaccine against coxsackievirus B3-induced myocarditis Role of retinoic acid in the imprinting of gut-homing IgA-secreting cells.Helminth products bypass the need for TSLP in Th2 immune responses by directly modulating dendritic cell function Plasticity is the differentiated state of CD4 T cells.Function of mucosa-associated lymphoid tissue in antibody formation.Vitamin A and immune regulation: role of retinoic acid in gut-associated dendritic cell education, immune protection and tolerance.Mononuclear phagocyte diversity in the intestine.New concepts in the generation and functions of IgA.Potential of immunoglobulin A to prevent allergic asthma.Identification and putative roles of distinct subtypes of intestinal dendritic cells in neuroimmune communication: what can be learned from other organ systems?Understanding the Molecular Mechanisms of the Interplay Between Herbal Medicines and Gut Microbiota.In situ proximity of CX3CR1-positive mononuclear phagocytes and VIP-ergic nerve fibers suggests VIP-ergic immunomodulation in the mouse ileum.Critical role of TSLP-responsive mucosal dendritic cells in the induction of nasal antigen-specific IgA response.The Gut's Little Brain in Control of Intestinal Immunity.Isolated Lymphoid Follicles are Dynamic Reservoirs for the Induction of Intestinal IgA.Effects of lactobacilli on cytokine expression by chicken spleen and cecal tonsil cells.Vitamin A metabolism and mucosal immune function are distinct between BALB/c and C57BL/6 mice.LRRK2: An Emerging New Molecule in the Enteric Neuronal System That Quantitatively Regulates Neuronal Peptides and IgA in the Gut.Retinoic acid, acting as a highly specific IgA isotype switch factor, cooperates with TGF-β1 to enhance the overall IgA response.

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P2860

Intestinal bacteria condition dendritic cells to promote IgA production

http://www.wikidata.org/entity/Q33348616

description

2008 nî lūn-bûn

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2008 թուականի Յուլիսին հրատարակուած գիտական յօդուած

@hyw

2008 թվականի հուլիսին հրատարակված գիտական հոդված

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2008年の論文

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2008年論文

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2008年論文

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2008年論文

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2008年論文

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2008年論文

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2008年论文

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name

Intestinal bacteria condition dendritic cells to promote IgA production

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Intestinal bacteria condition dendritic cells to promote IgA production

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type

label

Intestinal bacteria condition dendritic cells to promote IgA production

@ast

Intestinal bacteria condition dendritic cells to promote IgA production

@en

prefLabel

Intestinal bacteria condition dendritic cells to promote IgA production

@ast

Intestinal bacteria condition dendritic cells to promote IgA production

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JOURNAL.PONE.0002588

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Intestinal bacteria condition dendritic cells to promote IgA production

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Aubry Tardivel

http://www.w3.org/2001/XMLSchema#string

Bettina Ernst

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Joanna C Massacand

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Kurt Bürki

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Patrick Kaiser

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P2860

DCs induce CD40-independent immunoglobulin class switching through BLyS and APRIL

HIV-1 envelope triggers polyclonal Ig class switch recombination through a CD40-independent mechanism involving BAFF and C-type lectin receptors

A functionally specialized population of mucosal CD103+ DCs induces Foxp3+ regulatory T cells via a TGF-beta and retinoic acid-dependent mechanism

Interactions between commensal intestinal bacteria and the immune system

Nitric oxide and the immune response

Retinoic acid imprints gut-homing specificity on T cells

Selective generation of gut tropic T cells in gut-associated lymphoid tissue (GALT): requirement for GALT dendritic cells and adjuvant.

Epithelial cells secrete the chemokine interleukin-8 in response to bacterial entry.

Generation of gut-homing IgA-secreting B cells by intestinal dendritic cells.

Segmented filamentous bacteria are potent stimuli of a physiologically normal state of the murine gut mucosal immune system

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Pascal Schneider

Nicola L Harris

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5256309

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2432026

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