Nod2 mediates susceptibility to Yersinia pseudotuberculosis in mice.
about
Crohn's disease and early exposure to domestic refrigerationFunction of Nod-like receptors in microbial recognition and host defenseHost genetics and environmental factors regulate ecological succession of the mouse colon tissue-associated microbiotaDirect bacterial killing in vitro by recombinant Nod2 is compromised by Crohn's disease-associated mutationsNOD2 is dispensable for ATG16L1 deficiency-mediated resistance to urinary tract infection.Immune responses upon Campylobacter jejuni infection of secondary abiotic mice lacking nucleotide-oligomerization-domain-2.MDP-NOD2 stimulation induces HNP-1 secretion, which contributes to NOD2 antibacterial functionColonization of cecum is important for development of persistent infection by Yersinia pseudotuberculosis.Nucleotide-binding oligomerization domain-containing protein 2 controls host response to Campylobacter jejuni in Il10-/- mice.NOD1 and NOD2 regulation of pulmonary innate immunity to Legionella pneumophila.In vivo-induced InvA-like autotransporters Ifp and InvC of Yersinia pseudotuberculosis promote interactions with intestinal epithelial cells and contribute to virulence.NOD2 mutations and anti-Saccharomyces cerevisiae antibodies are risk factors for Crohn's disease in African AmericansRespective Roles of Hematopoietic and Nonhematopoietic Nod2 on the Gut Microbiota and Mucosal Homeostasis.Nod2: The intestinal gate keeper.Campylobacter jejuni infection of conventionally colonized mice lacking nucleotide-oligomerization-domain-2.Microbiota Composition and Immune Responses During Campylobacter Jejuni Infection in Conventionally Colonized IL-10-/- Mice Lacking Nucleotide Oligomerization Domain 2.Expression and functional importance of innate immune receptors by intestinal epithelial cells.Absence of Nucleotide-Oligomerization-Domain-2 Is Associated with Less Distinct Disease in Campylobacter jejuni Infected Secondary Abiotic IL-10 Deficient Mice.Nucleotide-binding oligomerization domain 2 (Nod2) is dispensable for the innate immune responses of macrophages against Yersinia enterocolitica.Nucleotide-binding oligomerization domain 2 signaling promotes hyperresponsive macrophages and colitis in IL-10-deficient mice.Prevalence of Species in the Ileum of Crohn's Disease Patients and Controls
P2860
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P2860
Nod2 mediates susceptibility to Yersinia pseudotuberculosis in mice.
description
2008 nî lūn-bûn
@nan
2008 թուականի Յուլիսին հրատարակուած գիտական յօդուած
@hyw
2008 թվականի հուլիսին հրատարակված գիտական հոդված
@hy
2008年の論文
@ja
2008年論文
@yue
2008年論文
@zh-hant
2008年論文
@zh-hk
2008年論文
@zh-mo
2008年論文
@zh-tw
2008年论文
@wuu
name
Nod2 mediates susceptibility to Yersinia pseudotuberculosis in mice.
@ast
Nod2 mediates susceptibility to Yersinia pseudotuberculosis in mice.
@en
type
label
Nod2 mediates susceptibility to Yersinia pseudotuberculosis in mice.
@ast
Nod2 mediates susceptibility to Yersinia pseudotuberculosis in mice.
@en
prefLabel
Nod2 mediates susceptibility to Yersinia pseudotuberculosis in mice.
@ast
Nod2 mediates susceptibility to Yersinia pseudotuberculosis in mice.
@en
P2093
P2860
P50
P1433
P1476
Nod2 mediates susceptibility to Yersinia pseudotuberculosis in mice.
@en
P2093
Claude Villard
Corinne Alberti
Dominique Berrebi
Fabrice Chareyre
Frederick Barreau
Ghislaine Sterkers
Hans Wolf-Watz
Lars Eckmann
Marco Giovannini
Michel Peuchmaur
P2860
P356
10.1371/JOURNAL.PONE.0002769
P407
P577
2008-07-23T00:00:00Z