Streptococcus pneumoniae synergizes with nontypeable Haemophilus influenzae to induce inflammation via upregulating TLR2 - Wikidata - awgldk - TriplyDB

Streptococcus pneumoniae synergizes with nontypeable Haemophilus influenzae to induce inflammation via upregulating TLR2

Streptococcus pneumoniae synergizes with nontypeable Haemophilus influenzae to induce inflammation via upregulating TLR2

http://www.wikidata.org/entity/Q33355817

about

A Pseudomonas aeruginosa toxin that hijacks the host ubiquitin proteolytic system Innocent until proven guilty: mechanisms and roles of Streptococcus-Candida interactions in oral health and disease Panel 4: Recent advances in otitis media in molecular biology, biochemistry, genetics, and animal models.The toll-Like receptor adaptor TRIF contributes to otitis media pathogenesis and recovery.PKCθ synergizes with TLR-dependent TRAF6 signaling pathway to upregulate MUC5AC mucin via CARMA1.Co-colonization by Haemophilus influenzae with Streptococcus pneumoniae enhances pneumococcal-specific antibody response in young children.Update of cylindromatosis gene (CYLD) mutations in Brooke-Spiegler syndrome: novel insights into the role of deubiquitination in cell signaling Antiinflammatory role of MUC1 mucin during infection with nontypeable Haemophilus influenzae.Critical role of type 1 plasminogen activator inhibitor (PAI-1) in early host defense against nontypeable Haemophilus influenzae (NTHi) infection.Phosphodiesterase 4B mediates extracellular signal-regulated kinase-dependent up-regulation of mucin MUC5AC protein by Streptococcus pneumoniae by inhibiting cAMP-protein kinase A-dependent MKP-1 phosphatase pathway.Toll-like receptor 4 agonistic antibody promotes innate immunity against severe pneumonia induced by coinfection with influenza virus and Streptococcus pneumoniae.Differential regulation of Streptococcus pneumoniae-induced human MUC5AC mucin expression through distinct MAPK pathways.Synergistic and feedback signaling mechanisms in the regulation of inflammation in respiratory infections.Nuclear factor-κB: a key regulator in health and disease of lungs.The Lung Immune Response to Nontypeable Haemophilus influenzae (Lung Immunity to NTHi).Innate Immunity: Orchestrating Inflammation and Resolution of Otitis Media.Interaction Between Allergy and Middle Ear Infection.TBX21 participates in innate immune response by regulating Toll-like receptor 2 expression in Streptococcus pneumoniae infections.Regulation of murine sinonasal cilia function by microbial secreted factors.

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P2860

Streptococcus pneumoniae synergizes with nontypeable Haemophilus influenzae to induce inflammation via upregulating TLR2

http://www.wikidata.org/entity/Q33355817

description

2008 nî lūn-bûn

@nan

2008 թուականի Յուլիսին հրատարակուած գիտական յօդուած

@hyw

2008 թվականի հուլիսին հրատարակված գիտական հոդված

@hy

2008年の論文

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2008年論文

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2008年論文

@zh-hant

2008年論文

@zh-hk

2008年論文

@zh-mo

2008年論文

@zh-tw

2008年论文

@wuu

name

Streptococcus pneumoniae syner ...... ammation via upregulating TLR2

@ast

Streptococcus pneumoniae syner ...... ammation via upregulating TLR2

@en

type

label

Streptococcus pneumoniae syner ...... ammation via upregulating TLR2

@ast

Streptococcus pneumoniae syner ...... ammation via upregulating TLR2

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prefLabel

Streptococcus pneumoniae syner ...... ammation via upregulating TLR2

@ast

Streptococcus pneumoniae syner ...... ammation via upregulating TLR2

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Streptococcus pneumoniae syner ...... ammation via upregulating TLR2

@en

P2093

Akihiro Sakai

http://www.w3.org/2001/XMLSchema#string

Chang-Hoon Woo

http://www.w3.org/2001/XMLSchema#string

Haidong Xu

http://www.w3.org/2001/XMLSchema#string

Jae Hyang Lim

http://www.w3.org/2001/XMLSchema#string

Jian-Dong Li

http://www.w3.org/2001/XMLSchema#string

Soo-Mi Kweon

http://www.w3.org/2001/XMLSchema#string

Unhwan Ha

http://www.w3.org/2001/XMLSchema#string

P2860

Glucocorticoids synergize with IL-1beta to induce TLR2 expression via MAP Kinase Phosphatase-1-dependent dual Inhibition of MAPK JNK and p38 in epithelial cells

NF-kappaB is essential for induction of CYLD, the negative regulator of NF-kappaB: evidence for a novel inducible autoregulatory feedback pathway

Cyld inhibits tumor cell proliferation by blocking Bcl-3-dependent NF-kappaB signaling

Regulation of T cell development by the deubiquitinating enzyme CYLD

Tumor suppressor CYLD acts as a negative regulator for non-typeable Haemophilus influenza-induced inflammation in the middle ear and lung of mice.

Synergistic proinflammatory responses induced by polymicrobial colonization of epithelial surfaces

Role of genetic resistance in invasive pneumococcal infection: identification and study of susceptibility and resistance in inbred mouse strains.

The apoptotic response to pneumolysin is Toll-like receptor 4 dependent and protects against pneumococcal disease

Recognition of pneumolysin by Toll-like receptor 4 confers resistance to pneumococcal infection.

Impaired regulation of NF-kappaB and increased susceptibility to colitis-associated tumorigenesis in CYLD-deficient mice.

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40

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scholarly article

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10.1186/1471-2172-9-40

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9

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2008-07-29T00:00:00Z

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23134864

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1039265148

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18664270

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Toll-like receptor

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2515102

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