Production of IFN-gamma by CD4 T cells is essential for resolving ehrlichia infection.
about
TLR2 and Nod2 mediate resistance or susceptibility to fatal intracellular Ehrlichia infection in murine models of ehrlichiosis.Immune mediators of protective and pathogenic immune responses in patients with mild and fatal human monocytotropic ehrlichiosis.MyD88-dependent signaling contributes to host defense against ehrlichial infection.Human ehrlichiosis and anaplasmosis.Natural killer cells promote tissue injury and systemic inflammatory responses during fatal Ehrlichia-induced toxic shock-like syndrome.Structure-based vaccines provide protection in a mouse model of ehrlichiosis.Progress and obstacles in vaccine development for the ehrlichiosesIdentification of novel antigenic proteins in a complex Anaplasma marginale outer membrane immunogen by mass spectrometry and genomic mapping.Early induction of interleukin-10 limits antigen-specific CD4⁺ T cell expansion, function, and secondary recall responses during persistent phagosomal infection.Role of tumor necrosis factor alpha (TNF-alpha) and interleukin-10 in the pathogenesis of severe murine monocytotropic ehrlichiosis: increased resistance of TNF receptor p55- and p75-deficient mice to fatal ehrlichial infection.An intradermal environment promotes a protective type-1 response against lethal systemic monocytotropic ehrlichial infectionThe interaction between IL-18 and IL-18 receptor limits the magnitude of protective immunity and enhances pathogenic responses following infection with intracellular bacteriaImmunization with Ehrlichia P28 outer membrane proteins confers protection in a mouse model of ehrlichiosis.Differential clearance and immune responses to tick cell-derived versus macrophage culture-derived Ehrlichia chaffeensis in mice.Vaccination with an Attenuated Mutant of Ehrlichia chaffeensis Induces Pathogen-Specific CD4+ T Cell Immunity and Protection from Tick-Transmitted Wild-Type Challenge in the Canine HostRelative importance of T-cell subsets in monocytotropic ehrlichiosis: a novel effector mechanism involved in Ehrlichia-induced immunopathology in murine ehrlichiosis.NK Cell-Mediated Regulation of Protective Memory Responses against Intracellular Ehrlichial Pathogens.T-Cell-independent humoral immunity is sufficient for protection against fatal intracellular ehrlichia infectionRegulatory roles of CD1d-restricted NKT cells in the induction of toxic shock-like syndrome in an animal model of fatal ehrlichiosis.Protective heterologous immunity against fatal ehrlichiosis and lack of protection following homologous challengeIncreased cytokine and nitric oxide levels in serum of dogs experimentally infected with Rangelia vitaliiProteomic analysis of and immune responses to Ehrlichia chaffeensis lipoproteinsDefining the immune response to Ehrlichia species using murine models.Economic Game Theory to Model the Attenuation of Virulence of an Obligate Intracellular Bacterium.Persistent infection contributes to heterologous protective immunity against fatal ehrlichiosis.Recombinant Ehrlichia P29 protein induces a protective immune response in a mouse model of ehrlichiosis.Influence of Genetic Background on Hematologic and Histopathologic Alterations during Acute Granulocytic Anaplasmosis in 129/SvEv and C57BL/6J Mice Lacking Type I and Type II Interferon Signaling.Development of Structure-Based Vaccines for Ehrlichiosis.Type I interferons promote severe disease in a mouse model of lethal ehrlichiosis.Ixodes ovatus Ehrlichia exhibits unique ultrastructural characteristics in mammalian endothelial and tick-derived cells.CD11c+ T-bet+ memory B cells: Immune maintenance during chronic infection and inflammation?Essential role for humoral immunity during Ehrlichia infection in immunocompetent mice.Anaplasma marginale major surface protein 2 CD4+-T-cell epitopes are evenly distributed in conserved and hypervariable regions (HVR), whereas linear B-cell epitopes are predominantly located in the HVR.CD11c expression identifies a population of extrafollicular antigen-specific splenic plasmablasts responsible for CD4 T-independent antibody responses during intracellular bacterial infection.CD4 T-cell epitopes associated with protective immunity induced following vaccination of mice with an ehrlichial variable outer membrane protein.MyD88-dependent inflammasome activation and autophagy inhibition contributes to Ehrlichia-induced liver injury and toxic shock.The CD4+ T cell immunodominant Anaplasma marginale major surface protein 2 stimulates gammadelta T cell clones that express unique T cell receptors.Temporal changes in macrophage phenotype after peripheral nerve injury.
P2860
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P2860
Production of IFN-gamma by CD4 T cells is essential for resolving ehrlichia infection.
description
2004 nî lūn-bûn
@nan
2004 թուականի Յունիսին հրատարակուած գիտական յօդուած
@hyw
2004 թվականի հունիսին հրատարակված գիտական հոդված
@hy
2004年の論文
@ja
2004年論文
@yue
2004年論文
@zh-hant
2004年論文
@zh-hk
2004年論文
@zh-mo
2004年論文
@zh-tw
2004年论文
@wuu
name
Production of IFN-gamma by CD4 T cells is essential for resolving ehrlichia infection.
@ast
Production of IFN-gamma by CD4 T cells is essential for resolving ehrlichia infection.
@en
type
label
Production of IFN-gamma by CD4 T cells is essential for resolving ehrlichia infection.
@ast
Production of IFN-gamma by CD4 T cells is essential for resolving ehrlichia infection.
@en
prefLabel
Production of IFN-gamma by CD4 T cells is essential for resolving ehrlichia infection.
@ast
Production of IFN-gamma by CD4 T cells is essential for resolving ehrlichia infection.
@en
P1476
Production of IFN-gamma by CD4 T cells is essential for resolving ehrlichia infection.
@en
P2093
Constantine Bitsaktsis
Gary Winslow
P304
P356
10.4049/JIMMUNOL.172.11.6894
P407
P577
2004-06-01T00:00:00Z