Beneficial effect of TRAIL on HIV burden, without detectable immune consequences. - Wikidata - awgldk - TriplyDB

Beneficial effect of TRAIL on HIV burden, without detectable immune consequences.

Beneficial effect of TRAIL on HIV burden, without detectable immune consequences.

http://www.wikidata.org/entity/Q33366417

about

Inability of plasmacytoid dendritic cells to directly lyse HIV-infected autologous CD4+ T cells despite induction of tumor necrosis factor-related apoptosis-inducing ligand.Mechanisms of HIV-associated lymphocyte apoptosis: 2010 Type I interferon upregulates Bak and contributes to T cell loss during human immunodeficiency virus (HIV) infection.Isolation of a TRAIL antagonist from the serum of HIV-infected patients.Increasing procaspase 8 expression using repurposed drugs to induce HIV infected cell death in ex vivo patient cells.Colony-Stimulating Factor 1 Receptor Antagonists Sensitize Human Immunodeficiency Virus Type 1-Infected Macrophages to TRAIL-Mediated Killing.CD8+ T cells use TRAIL to restrict West Nile virus pathogenesis by controlling infection in neurons.

P2860

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P2860

Beneficial effect of TRAIL on HIV burden, without detectable immune consequences.

http://www.wikidata.org/entity/Q33366417

description

2008 nî lūn-bûn

@nan

2008 թուականի Օգոստոսին հրատարակուած գիտական յօդուած

@hyw

2008 թվականի օգոստոսին հրատարակված գիտական հոդված

@hy

2008年の論文

@ja

2008年論文

@yue

2008年論文

@zh-hant

2008年論文

@zh-hk

2008年論文

@zh-mo

2008年論文

@zh-tw

2008年论文

@wuu

name

Beneficial effect of TRAIL on HIV burden, without detectable immune consequences.

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Beneficial effect of TRAIL on HIV burden, without detectable immune consequences.

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type

label

Beneficial effect of TRAIL on HIV burden, without detectable immune consequences.

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Beneficial effect of TRAIL on HIV burden, without detectable immune consequences.

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Beneficial effect of TRAIL on HIV burden, without detectable immune consequences.

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Beneficial effect of TRAIL on HIV burden, without detectable immune consequences.

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JOURNAL.PONE.0003096

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Beneficial effect of TRAIL on HIV burden, without detectable immune consequences.

@en

P2093

Andrea Foli

http://www.w3.org/2001/XMLSchema#string

Brett D Shepard

http://www.w3.org/2001/XMLSchema#string

David R McNamara

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Davide De Forni

http://www.w3.org/2001/XMLSchema#string

Franco Lori

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Keith Knutson

http://www.w3.org/2001/XMLSchema#string

Peter J Wettstein

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Roshini S Abraham

http://www.w3.org/2001/XMLSchema#string

Stacey A Rizza

http://www.w3.org/2001/XMLSchema#string

P2860

Identification of a reservoir for HIV-1 in patients on highly active antiretroviral therapy

Apoptotic killing of HIV-1-infected macrophages is subverted by the viral envelope glycoprotein

Quantification of latent tissue reservoirs and total body viral load in HIV-1 infection

Induction of cell death in human immunodeficiency virus-infected macrophages and resting memory CD4 T cells by TRAIL/Apo2l

Differential effects of interleukin-7 and interleukin-15 on NK cell anti-human immunodeficiency virus activity.

Identification of a potential HIV-induced source of bystander-mediated apoptosis in T cells: upregulation of trail in primary human macrophages by HIV-1 tat.

Differential expression of IFN-alpha and TRAIL/DR5 in lymphoid tissue of progressor versus nonprogressor HIV-1-infected patients.

Upregulation of Fas ligand expression by human immunodeficiency virus in human macrophages mediates apoptosis of uninfected T lymphocytes

Early establishment of a pool of latently infected, resting CD4(+) T cells during primary HIV-1 infection.

Macrophage-dependent apoptosis of CD4+ T lymphocytes from HIV-infected individuals is mediated by FasL and tumor necrosis factor

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e3096

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