Segmental duplications arise from Pol32-dependent repair of broken forks through two alternative replication-based mechanisms.
about
Additions, losses, and rearrangements on the evolutionary route from a reconstructed ancestor to the modern Saccharomyces cerevisiae genomeComparative genomics of protoploid SaccharomycetaceaeMechanisms of change in gene copy numberComparative genomics and molecular dynamics of DNA repeats in eukaryotesMechanisms underlying structural variant formation in genomic disordersMechanisms and regulation of mitotic recombination in Saccharomyces cerevisiaeStructural variation mutagenesis of the human genome: Impact on disease and evolution.Differences in the DNA replication of unicellular eukaryotes and metazoans: known unknownsBreak-induced replication requires all essential DNA replication factors except those specific for pre-RC assembly.Regulation of ribosomal DNA amplification by the TOR pathway.Migrating bubble during break-induced replication drives conservative DNA synthesis.Increased mutagenesis and unique mutation signature associated with mitotic gene conversionNoncanonical views of homology-directed DNA repairFrequent Somatic Mutation in Adult Intestinal Stem Cells Drives Neoplasia and Genetic Mosaicism during AgingAsexual populations of the human malaria parasite, Plasmodium falciparum, use a two-step genomic strategy to acquire accurate, beneficial DNA amplificationsThe functional basis of adaptive evolution in chemostatsProperties and rates of germline mutations in humansTwo routes to senescence revealed by real-time analysis of telomerase-negative single lineages.The repertoire and dynamics of evolutionary adaptations to controlled nutrient-limited environments in yeastA microhomology-mediated break-induced replication model for the origin of human copy number variation.Whole-genome sequencing of a laboratory-evolved yeast strain.Origin and fate of pseudogenes in Hemiascomycetes: a comparative analysis.Fusion of nearby inverted repeats by a replication-based mechanism leads to formation of dicentric and acentric chromosomes that cause genome instability in budding yeast.Role of double-stranded DNA translocase activity of human HLTF in replication of damaged DNAFragile genomic sites are associated with origins of replication.Formation of complex and unstable chromosomal translocations in yeastIdentification of uncommon recurrent Potocki-Lupski syndrome-associated duplications and the distribution of rearrangement types and mechanisms in PTLSBreak-induced replication repair of damaged forks induces genomic duplications in human cells.An Sp185/333 gene cluster from the purple sea urchin and putative microsatellite-mediated gene diversification.The origins and impact of primate segmental duplications.Alterations in DNA replication and histone levels promote histone gene amplification in Saccharomyces cerevisiae.Origin-dependent inverted-repeat amplification: a replication-based model for generating palindromic amplicons.Pathways and Mechanisms that Prevent Genome Instability in Saccharomyces cerevisiaeGlobal chromosomal structural instability in a subpopulation of starving Escherichia coli cellsBreak-induced DNA replicationChromosome catastrophes involve replication mechanisms generating complex genomic rearrangementsThe role of replication bypass pathways in dicentric chromosome formation in budding yeastChromosome rearrangements via template switching between diverged repeated sequences.Recovery of arrested replication forks by homologous recombination is error-prone.Mus81 and Yen1 promote reciprocal exchange during mitotic recombination to maintain genome integrity in budding yeast.
P2860
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P2860
Segmental duplications arise from Pol32-dependent repair of broken forks through two alternative replication-based mechanisms.
description
2008 nî lūn-bûn
@nan
2008 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած
@hyw
2008 թվականի սեպտեմբերին հրատարակված գիտական հոդված
@hy
2008年の論文
@ja
2008年論文
@yue
2008年論文
@zh-hant
2008年論文
@zh-hk
2008年論文
@zh-mo
2008年論文
@zh-tw
2008年论文
@wuu
name
Segmental duplications arise f ...... replication-based mechanisms.
@ast
Segmental duplications arise f ...... replication-based mechanisms.
@en
type
label
Segmental duplications arise f ...... replication-based mechanisms.
@ast
Segmental duplications arise f ...... replication-based mechanisms.
@en
prefLabel
Segmental duplications arise f ...... replication-based mechanisms.
@ast
Segmental duplications arise f ...... replication-based mechanisms.
@en
P2860
P1433
P1476
Segmental duplications arise f ...... replication-based mechanisms.
@en
P2093
Bernard Dujon
Celia Payen
P2860
P304
P356
10.1371/JOURNAL.PGEN.1000175
P577
2008-09-05T00:00:00Z