Activation of the unfolded protein response is required for defenses against bacterial pore-forming toxin in vivo.
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Dysregulated LRRK2 signaling in response to endoplasmic reticulum stress leads to dopaminergic neuron degeneration in C. elegansEvolution of host innate defence: insights from Caenorhabditis elegans and primitive invertebratesSignaling beyond Punching Holes: Modulation of Cellular Responses by Vibrio cholerae CytolysinMultifaceted activity of listeriolysin O, the cholesterol-dependent cytolysin of Listeria monocytogenesMore than a pore: the cellular response to cholesterol-dependent cytolysinsGlobal functional analyses of cellular responses to pore-forming toxinsNeuronal Goα and CAPS regulate behavioral and immune responses to bacterial pore-forming toxinsmir-233 modulates the unfolded protein response in C. elegans during Pseudomonas aeruginosa infectionMonalysin, a novel ß-pore-forming toxin from the Drosophila pathogen Pseudomonas entomophila, contributes to host intestinal damage and lethalityNucleolar proteins suppress Caenorhabditis elegans innate immunity by inhibiting p53/CEP-1Pneumococcal hydrogen peroxide-induced stress signaling regulates inflammatory genesPhysiological IRE-1-XBP-1 and PEK-1 signaling in Caenorhabditis elegans larval development and immunityMechanistic and single-dose in vivo therapeutic studies of Cry5B anthelmintic action against hookwormsBacillus thuringiensis-derived Cry5B has potent anthelmintic activity against Ascaris suumGene-trap mutagenesis identifies mammalian genes contributing to intoxication by Clostridium perfringens ε-toxinProteome changes of Caenorhabditis elegans upon a Staphylococcus aureus infection.Phosphatase-dependent regulation of epithelial mitogen-activated protein kinase responses to toxin-induced membrane pores.Hypoxia and the hypoxic response pathway protect against pore-forming toxins in C. elegans.WWP-1 is a novel modulator of the DAF-2 insulin-like signaling network involved in pore-forming toxin cellular defenses in Caenorhabditis elegans.Bacillus thuringiensis DB27 produces two novel protoxins, Cry21Fa1 and Cry21Ha1, which act synergistically against nematodes.Curating the innate immunity interactome.Constitutive activation of the midgut response to Bacillus thuringiensis in Bt-resistant Spodoptera exigua.An essential role for XBP-1 in host protection against immune activation in C. elegans.Endoplasmic Reticulum Stress Caused by Lipoprotein Accumulation Suppresses Immunity against Bacterial Pathogens and Contributes to ImmunosenescenceHeat-Labile Enterotoxin-Induced PERK-CHOP Pathway Activation Causes Intestinal Epithelial Cell Apoptosis.Reduced levels of membrane-bound alkaline phosphatase are common to lepidopteran strains resistant to Cry toxins from Bacillus thuringiensis.Induction of endoplasmic reticulum stress and unfolded protein response constitutes a pathogenic strategy of group A streptococcusThe early-onset torsion dystonia-associated protein, torsinA, is a homeostatic regulator of endoplasmic reticulum stress response.Protein misfolding induces hypoxic preconditioning via a subset of the unfolded protein response machinery.Transcriptome profiling of the intoxication response of Tenebrio molitor larvae to Bacillus thuringiensis Cry3Aa protoxinMembrane damage during Listeria monocytogenes infection triggers a caspase-7 dependent cytoprotective responseRole of pore-forming toxins in bacterial infectious diseases.Oligomerization of Clostridium perfringens epsilon toxin is dependent upon caveolins 1 and 2.Proteome response of Tribolium castaneum larvae to Bacillus thuringiensis toxin producing strains.RTCB-1 mediates neuroprotection via XBP-1 mRNA splicing in the unfolded protein response pathway.Regulation of innate immunity by signaling pathways emerging from the endoplasmic reticulum.RAB-5- and RAB-11-dependent vesicle-trafficking pathways are required for plasma membrane repair after attack by bacterial pore-forming toxin.Regulation of anoxic death in Caenorhabditis elegans by mammalian apoptosis signal-regulating kinase (ASK) family proteins.Role of the nervous system in the control of proteostasis during innate immune activation: insights from C. elegansTLR activation of the transcription factor XBP1 regulates innate immune responses in macrophages.
P2860
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P2860
Activation of the unfolded protein response is required for defenses against bacterial pore-forming toxin in vivo.
description
2008 nî lūn-bûn
@nan
2008 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2008 թվականի հոտեմբերին հրատարակված գիտական հոդված
@hy
2008年の論文
@ja
2008年論文
@yue
2008年論文
@zh-hant
2008年論文
@zh-hk
2008年論文
@zh-mo
2008年論文
@zh-tw
2008年论文
@wuu
name
Activation of the unfolded pro ...... al pore-forming toxin in vivo.
@ast
Activation of the unfolded pro ...... al pore-forming toxin in vivo.
@en
type
label
Activation of the unfolded pro ...... al pore-forming toxin in vivo.
@ast
Activation of the unfolded pro ...... al pore-forming toxin in vivo.
@en
prefLabel
Activation of the unfolded pro ...... al pore-forming toxin in vivo.
@ast
Activation of the unfolded pro ...... al pore-forming toxin in vivo.
@en
P2093
P2860
P1433
P1476
Activation of the unfolded pro ...... ial pore-forming toxin in vivo
@en
P2093
Ferdinand C O Los
Larry J Bischof
Manuel R Gonzalez
Raffi V Aroian
Steven P Briggs
Zhouxin Shen
P2860
P304
P356
10.1371/JOURNAL.PPAT.1000176
P577
2008-10-10T00:00:00Z