Interactions between Shiga toxins and human polymorphonuclear leukocytes.
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Shiga Toxins as Multi-Functional Proteins: Induction of Host Cellular Stress Responses, Role in Pathogenesis and Therapeutic ApplicationsEndothelial damage induced by Shiga toxins delivered by neutrophils during transmigration.Change in conformation with reduction of alpha-helix content causes loss of neutrophil binding activity in fully cytotoxic Shiga toxin 1.Shiga toxin pathogenesis: kidney complications and renal failure.Microparticle generation and leucocyte death in Shiga toxin-mediated HUS.[Evolution and infection biology of hemolytic-uremic syndrome (HUS) associated E. coli (HUSEC)].A rapid and sensitive method to measure the functional activity of Shiga toxins in human serumThe Antibiotic Polymyxin B Impairs the Interactions between Shiga Toxins and Human Neutrophils.Induction of apoptosis by Shiga toxins.Shiga toxin glycosphingolipid receptors in microvascular and macrovascular endothelial cells: differential association with membrane lipid raft microdomainsNeutral glycosphingolipids in human blood: a precise mass spectrometry analysis with special reference to lipoprotein-associated Shiga toxin receptorsShiga toxins: intracellular trafficking to the ER leading to activation of host cell stress responses.Differential response of the human renal proximal tubular epithelial cell line HK-2 to Shiga toxin types 1 and 2.Shiga toxins induce autophagy leading to differential signalling pathways in toxin-sensitive and toxin-resistant human cells.Role of neutrophils in innate immunity: a systems biology-level approachAssociation of Shiga toxin glycosphingolipid receptors with membrane microdomains of toxin-sensitive lymphoid and myeloid cells.The interactions of human neutrophils with shiga toxins and related plant toxins: danger or safety?Facing glycosphingolipid-Shiga toxin interaction: dire straits for endothelial cells of the human vasculature.Shiga toxin 1 and ricin A chain bind to human polymorphonuclear leucocytes through a common receptor.Shiga Toxin Glycosphingolipid Receptors in Human Caco-2 and HCT-8 Colon Epithelial Cell Lines.LPS-primed CD11b+ leukocytes serve as an effective carrier of Shiga toxin 2 to cause hemolytic uremic syndrome in mice.Management of hemolytic uremic syndromeSoluble Toll-Like Receptor 4 Impairs the Interaction of Shiga Toxin 2a with Human Serum Amyloid P Component
P2860
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P2860
Interactions between Shiga toxins and human polymorphonuclear leukocytes.
description
2008 nî lūn-bûn
@nan
2008 թուականի Յուլիսին հրատարակուած գիտական յօդուած
@hyw
2008 թվականի հուլիսին հրատարակված գիտական հոդված
@hy
2008年の論文
@ja
2008年論文
@yue
2008年論文
@zh-hant
2008年論文
@zh-hk
2008年論文
@zh-mo
2008年論文
@zh-tw
2008年论文
@wuu
name
Interactions between Shiga toxins and human polymorphonuclear leukocytes.
@ast
Interactions between Shiga toxins and human polymorphonuclear leukocytes.
@en
type
label
Interactions between Shiga toxins and human polymorphonuclear leukocytes.
@ast
Interactions between Shiga toxins and human polymorphonuclear leukocytes.
@en
prefLabel
Interactions between Shiga toxins and human polymorphonuclear leukocytes.
@ast
Interactions between Shiga toxins and human polymorphonuclear leukocytes.
@en
P2093
P2860
P356
P1476
Interactions between Shiga toxins and human polymorphonuclear leukocytes.
@en
P2093
Alfredo Caprioli
Andrea Bontadini
Domenica Carnicelli
Elisa Ravanelli
Francesca Ricci
Gaia Scavia
Maurizio Brigotti
Pier Luigi Tazzari
Stefania Barbieri
P2860
P304
P356
10.1189/JLB.0308157
P577
2008-07-14T00:00:00Z