Pathogenic hantaviruses direct the adherence of quiescent platelets to infected endothelial cells.
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Hemorrhagic Fever with Renal Syndrome: Pathogenesis and Clinical PictureHantavirus protein interactions regulate cellular functions and signaling responses.Hantavirus infection: a neglected diagnosis in thrombocytopenia and fever?Hantavirus infection--hemorrhagic fever with renal syndrome: the first case series reported in Romania and review of the literature.Hantavirus infections for the clinician: from case presentation to diagnosis and treatment.Pathogenesis of emerging severe fever with thrombocytopenia syndrome virus in C57/BL6 mouse model.Hypoxia induces permeability and giant cell responses of Andes virus-infected pulmonary endothelial cells by activating the mTOR-S6K signaling pathway.Platelets and infection - an emerging role of platelets in viral infection.Endothelial cell dysfunction in viral hemorrhage and edema.Increased Thrombopoiesis and Platelet Activation in Hantavirus-Infected Patients.Thrombocytopenia associates with the severity of inflammation and variables reflecting capillary leakage in Puumala Hantavirus infection, an analysis of 546 Finnish patients.Platelets and infections in the resource-limited countries with a focus on malaria and viral haemorrhagic fevers.Hantavirus interferon regulation and virulence determinantsPathogenic hantaviruses Andes virus and Hantaan virus induce adherens junction disassembly by directing vascular endothelial cadherin internalization in human endothelial cellsHantaviruses in the americas and their role as emerging pathogensDifferential lymphocyte and antibody responses in deer mice infected with Sin Nombre hantavirus or Andes hantavirus.Andes virus regulation of cellular microRNAs contributes to hantavirus-induced endothelial cell permeability.Virus interactions with endothelial cell receptors: implications for viral pathogenesis.VEGFR2 and Src kinase inhibitors suppress Andes virus-induced endothelial cell permeabilityThe C-terminal 42 residues of the Tula virus Gn protein regulate interferon induction.The role of platelets in the pathogenesis of viral hemorrhagic feversT cells are not required for pathogenesis in the Syrian hamster model of hantavirus pulmonary syndrome.Effect of Puumala hantavirus infection on human umbilical vein endothelial cell hemostatic function: platelet interactions, increased tissue factor expression and fibrinolysis regulator release.Andes virus infection of lymphatic endothelial cells causes giant cell and enhanced permeability responses that are rapamycin and vascular endothelial growth factor C sensitive.Hantavirus regulation of type I interferon responses.Elevated VEGF Levels in Pulmonary Edema Fluid and PBMCs from Patients with Acute Hantavirus Pulmonary Syndrome.Sustained high level of serum VEGF at convalescent stage contributes to the renal recovery after HTNV infection in patients with hemorrhagic fever with renal syndrome.Seoul virus-infected rat lung endothelial cells and alveolar macrophages differ in their ability to support virus replication and induce regulatory T cell phenotypes.The Andes Virus Nucleocapsid Protein Directs Basal Endothelial Cell Permeability by Activating RhoA.An innate immunity-regulating virulence determinant is uniquely encoded by the Andes virus nucleocapsid protein.The role of endothelial activation in dengue hemorrhagic fever and hantavirus pulmonary syndrome.Review: Viral infections and mechanisms of thrombosis and bleeding.Epidemiology of Hantavirus infections in humans: a comprehensive, global overview.The fundamental role of endothelial cells in hantavirus pathogenesis.Cytokine response to Hantaan virus infection in patients with hemorrhagic fever with renal syndrome.Intestinal lymphatic vasculature: structure, mechanisms and functions.Slit2-Robo4 receptor responses inhibit ANDV directed permeability of human lung microvascular endothelial cells.Can hantavirus infections be predicted on admission to hospital?Arterial thrombosis in the context of HCV-associated vascular disease can be prevented by protein C.Role of vascular and lymphatic endothelial cells in hantavirus pulmonary syndrome suggests targeted therapeutic approaches.
P2860
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P2860
Pathogenic hantaviruses direct the adherence of quiescent platelets to infected endothelial cells.
description
2010 nî lūn-bûn
@nan
2010 թուականի Փետրուարին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի փետրվարին հրատարակված գիտական հոդված
@hy
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
name
Pathogenic hantaviruses direct ...... to infected endothelial cells.
@ast
Pathogenic hantaviruses direct ...... to infected endothelial cells.
@en
type
label
Pathogenic hantaviruses direct ...... to infected endothelial cells.
@ast
Pathogenic hantaviruses direct ...... to infected endothelial cells.
@en
prefLabel
Pathogenic hantaviruses direct ...... to infected endothelial cells.
@ast
Pathogenic hantaviruses direct ...... to infected endothelial cells.
@en
P2093
P2860
P356
P1433
P1476
Pathogenic hantaviruses direct ...... to infected endothelial cells.
@en
P2093
Elena E Gorbunova
Erich R Mackow
Irina N Gavrilovskaya
P2860
P304
P356
10.1128/JVI.02405-09
P577
2010-02-24T00:00:00Z