Factor XI-deficient mice display reduced inflammation, coagulopathy, and bacterial growth during listeriosis - Wikidata - awgldk - TriplyDB

Factor XI-deficient mice display reduced inflammation, coagulopathy, and bacterial growth during listeriosis

Factor XI-deficient mice display reduced inflammation, coagulopathy, and bacterial growth during listeriosis

http://www.wikidata.org/entity/Q33397684

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Pharmacological targeting of the thrombomodulin-activated protein C pathway mitigates radiation toxicity Causes and consequences of coagulation activation in sepsis: an evolutionary medicine perspective Factor XI and contact activation as targets for antithrombotic therapy Genetic sequence analysis of inherited bleeding diseases.Factor XI Deficiency Protects Against Atherogenesis in Apolipoprotein E/Factor XI Double Knockout Mice.Factor XIa inhibitors: A review of the patent literature.Coagulation factor XI vaccination: an alternative strategy to prevent thrombosis.Prochemerin cleavage by factor XIa links coagulation and inflammation.

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Factor XI-deficient mice display reduced inflammation, coagulopathy, and bacterial growth during listeriosis

http://www.wikidata.org/entity/Q33397684

description

2011 nî lūn-bûn

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2011 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած

@hyw

2011 թվականի հոտեմբերին հրատարակված գիտական հոդված

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2011年の論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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name

Factor XI-deficient mice displ ...... rial growth during listeriosis

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Factor XI-deficient mice displ ...... rial growth during listeriosis

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type

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Factor XI-deficient mice displ ...... rial growth during listeriosis

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Factor XI-deficient mice displ ...... rial growth during listeriosis

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Factor XI-deficient mice displ ...... rial growth during listeriosis

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Factor XI-deficient mice displ ...... rial growth during listeriosis

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Infection and Immunity

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Factor XI-deficient mice displ ...... rial growth during listeriosis

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David Gailani

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Deyan Luo

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Erik I Tucker

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Frank M Szaba

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Lawrence L Johnson

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Lawrence W Kummer

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Stephen T Smiley

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P2860

Definitions for sepsis and organ failure and guidelines for the use of innovative therapies in sepsis. The ACCP/SCCM Consensus Conference Committee. American College of Chest Physicians/Society of Critical Care Medicine

Human recombinant activated protein C for severe sepsis

Activation of endothelial cell protease activated receptor 1 by the protein C pathway

Universal changes in biomarkers of coagulation and inflammation occur in patients with severe sepsis, regardless of causative micro-organism [ISRCTN74215569]

Coagulation in sepsis: all bugs bite equally

Epidemiology of severe sepsis in the United States: analysis of incidence, outcome, and associated costs of care

The molecular basis of blood coagulation

Activation of human blood coagulation factor XI independent of factor XII. Factor XI is activated by thrombin and factor XIa in the presence of negatively charged surfaces

A role for factor XIIa-mediated factor XI activation in thrombus formation in vivo

Efficacy and safety of recombinant human activated protein C for severe sepsis

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10.1128/IAI.05568-11

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