Control of stochastic gene expression by host factors at the HIV promoter. - Wikidata - awgldk - TriplyDB

Control of stochastic gene expression by host factors at the HIV promoter.

Control of stochastic gene expression by host factors at the HIV promoter.

http://www.wikidata.org/entity/Q33398516

about

Spatial epigenetic control of mono- and bistable gene expression Retroviral integration: Site matters: Mechanisms and consequences of retroviral integration site selection HIV promoter integration site primarily modulates transcriptional burst size rather than frequency Adjusting phenotypes by noise control Cellular heterogeneity and live cell arrays.Negative elongation factor is required for the maintenance of proviral latency but does not induce promoter-proximal pausing of RNA polymerase II on the HIV long terminal repeat.Opinion: Inhibition of Blood-Brain Barrier Repair as a Mechanism in HIV-1 Disease.Functional properties of the HIV-1 long terminal repeat containing single-nucleotide polymorphisms in Sp site III and CCAAT/enhancer binding protein site I.Combinatorial latency reactivation for HIV-1 subtypes and variants Transcriptional and posttranscriptional regulation of HIV-1 gene expression Regulation of Mycobacterium tuberculosis-dependent HIV-1 transcription reveals a new role for NFAT5 in the toll-like receptor pathway.The molecular biology of HIV latency: breaking and restoring the Tat-dependent transcriptional circuit.Innate and adaptive factors regulating human immunodeficiency virus type 1 genomic activation.Genetic selection for context-dependent stochastic phenotypes: Sp1 and TATA mutations increase phenotypic noise in HIV-1 gene expression.Epigenetic heterogeneity in HIV-1 latency establishment.Viral-host interactions that control HIV-1 transcriptional elongation Calcium/calcineurin synergizes with prostratin to promote NF-κB dependent activation of latent HIV A hardwired HIV latency program.HIV Latency Is Established Directly and Early in Both Resting and Activated Primary CD4 T Cells.Orthogonal control of expression mean and variance by epigenetic features at different genomic loci An evolutionary role for HIV latency in enhancing viral transmission.Modeling the Effects of Vorinostat In Vivo Reveals both Transient and Delayed HIV Transcriptional Activation and Minimal Killing of Latently Infected Cells.Quantitative evaluation and optimization of co-drugging to improve anti-HIV latency therapy Mutual information analysis reveals coevolving residues in Tat that compensate for two distinct functions in HIV-1 gene expression.HIV develops indirect cross-resistance to combinatorial RNAi targeting two distinct and spatially distant sites.Chromatin accessibility at the HIV LTR promoter sets a threshold for NF-κB mediated viral gene expression A passive-flow microfluidic device for imaging latent HIV activation dynamics in single T cells Human immunodeficiency virus (HIV) latency: the major hurdle in HIV eradication Distinct promoter activation mechanisms modulate noise-driven HIV gene expression.NF-κB/Rel: agonist and antagonist roles in HIV-1 latency.Multiple NF-κB sites in HIV-1 subtype C long terminal repeat confer superior magnitude of transcription and thereby the enhanced viral predominance.CRISPR-mediated Activation of Latent HIV-1 Expression CNS-specific regulatory elements in brain-derived HIV-1 strains affect responses to latency-reversing agents with implications for cure strategies.Contribution of myocyte enhancer factor 2 family transcription factors to BZLF1 expression in Epstein-Barr virus reactivation from latency.Functional Incompatibility between the Generic NF-κB Motif and a Subtype-Specific Sp1III Element Drives the Formation of the HIV-1 Subtype C Viral Promoter A dynamic G-quadruplex region regulates the HIV-1 long terminal repeat promoter HIV interactions with monocytes and dendritic cells: viral latency and reservoirs.In vitro dynamics of HIV-1 BF intersubtype recombinants genomic regions involved in the regulation of gene expression Stochastic gene expression as a molecular switch for viral latency.Control of HIV latency by epigenetic and non-epigenetic mechanisms.

P2860

Q21090179-5E7D196A-6315-4F27-B9F0-CDACCEF53460 Q28088514-E900D415-D3ED-4F80-80C5-47470E3A944E Q28475647-2D2BEEA5-355F-4A7D-8D79-963B8DD57222 Q28478888-A6B7C743-D979-463A-9273-C6FDB6354CE5 Q30401785-DEDC345E-987F-4D90-ADBC-AB388285AAA5 Q33602605-B637C6E1-69FB-4A00-A73B-00DBDA8A4D2C Q33603457-DAD9A712-ECAB-4FB7-8A31-F6A4993D7675 Q33716931-97423FF5-514B-496F-9C13-48344A0DDAA3 Q33877492-3FE0E5CA-46A7-4233-A62E-AD47ACE368DF Q34030804-0E8B6580-70B0-45FD-AFF9-01D319F0038D Q34229621-AFA13CF1-8F7F-495C-B2FB-E6CE4C3DC09A Q34544409-F3B4FA3F-F4CA-4581-A133-D2B552A21B4E Q34671609-8CD55175-4657-4140-9FAC-DD9294598A83 Q34845153-2A21E6F0-490F-4C2E-B73D-56FA8EFFF62C Q34877762-689F4BD8-BF85-4AE2-B6A6-4A2A942F0B4B Q35026881-BF417F08-2AB3-4EAE-9539-C69DE0A13C7B Q35035572-1B949E8E-7B23-49B9-A242-57F3E8CE07AB Q35400385-68E6825E-BE1D-4E06-8426-610E8469CF50 Q35661660-B5739A8F-ED97-4650-B488-3269677608CC Q35705533-3A2E3B03-9052-4DB9-B166-CE3F90AC8137 Q35807225-B4FD05E8-D4F4-43CC-B3E0-728D33BBADF4 Q35819184-E1A48C27-C7C9-466C-85D9-DC99679CD9E8 Q35848096-262BC56A-B4EE-4A38-9D9E-3B96F88D0129 Q35868299-8D4A62EC-9385-46EB-8AAB-8A9F7FA99D0A Q35876190-FC06BF17-F289-4E6A-A24A-530F368836B6 Q35996134-7B926536-59F9-4D8F-87C3-FE2EFBD95F3B Q36021924-7C93FBDA-E51E-42F9-9E32-7031B8B6CB43 Q36330062-AE07873F-8D47-418F-96A7-64FAC9D267BE Q36369858-E4AE4DB7-9AB4-4B8A-AFEB-74288EC57A45 Q36383606-B8079430-62EA-4C39-A8F1-C9DB027DC4E2 Q36492916-FAB14AE7-D185-447E-AEA0-C8D313E0B931 Q36675297-66C48450-A328-43F2-B760-BDEF47971F19 Q36719187-C3CE414D-9ECA-4766-ABD5-459CABE23F3A Q37123207-3ADEB532-7F59-41D4-923A-1D125A65D969 Q37174431-101FA842-1D4D-4666-865B-D19882E6811E Q37216613-6B260748-7EF1-41DD-84BA-8CFF224C410B Q37229398-E1A1BEBD-B949-467D-9C6B-60CD97582A61 Q37281360-C4FE15D8-1901-41F1-82BF-0B043B17B4FB Q37549111-B1FB3D70-E7B4-4569-ADBB-D38EDC4CE4DF Q37972643-1B02C0DD-F51E-474B-87C1-16267E2097D5

P2860

Control of stochastic gene expression by host factors at the HIV promoter.

http://www.wikidata.org/entity/Q33398516

description

2009 nî lūn-bûn

@nan

2009 թուականի Յունուարին հրատարակուած գիտական յօդուած

@hyw

2009 թվականի հունվարին հրատարակված գիտական հոդված

@hy

2009年の論文

@ja

2009年論文

@yue

2009年論文

@zh-hant

2009年論文

@zh-hk

2009年論文

@zh-mo

2009年論文

@zh-tw

2009年论文

@wuu

name

Control of stochastic gene expression by host factors at the HIV promoter.

@ast

Control of stochastic gene expression by host factors at the HIV promoter.

@en

type

label

Control of stochastic gene expression by host factors at the HIV promoter.

@ast

Control of stochastic gene expression by host factors at the HIV promoter.

@en

prefLabel

Control of stochastic gene expression by host factors at the HIV promoter.

@ast

Control of stochastic gene expression by host factors at the HIV promoter.

@en

P1433

Q33398516-5252196D-0FC8-4D8B-B4FD-D61C44CCBC15

P1476

Q33398516-86AA291E-7001-4BAE-AD20-D646439E9EFD

P2093

Q33398516-79ADA635-9DDE-4580-9280-11F6BB8937AB

Q33398516-B795F65D-F53C-4B9B-952D-6BA0C9547D83

Q33398516-D01D15D4-3053-4AFA-BC2A-C489B291822A

P2860

Q33398516-04FE1DC4-29CD-47A9-B286-2904833FEEFC

Q33398516-059F3149-AB53-4CD5-8C34-433D4F8E8B58

Q33398516-0FBC3959-E0D0-49C0-920B-F4B822551FEA

Q33398516-0FD0169E-A91A-4F31-9CC0-CDAB649A7A83

Q33398516-131ABF46-AD0C-42C2-A9FC-586C5D2A2D4A

Q33398516-1578134C-58C0-4526-A4C6-1182891EC73A

Q33398516-162B5D4A-71F5-4A85-B589-6500FE385B8E

Q33398516-1F2B2DB9-8F62-4CEF-A572-24C3A1D886BC

Q33398516-2281A2EC-870A-4EC1-AA68-234D8A4776A4

Q33398516-236B358D-7003-489A-AD47-4473F449769D

P304

Q33398516-1300DB22-128F-4086-9919-B9C9C732F18A

P31

Q33398516-C6AD8EA7-84F8-4A94-A674-6C0AA6CC66A9

P356

Q33398516-DA5A8FA8-C9C8-42BA-834D-4C8AC1AD3AE0

P433

Q33398516-D995629F-E4A6-4151-A450-BEF0FC1E855B

P478

Q33398516-4FACB2AE-C4DF-4D1C-AB7C-BDC167A643AE

P50

Q33398516-652E68B4-B1CD-4078-AB0F-2EC6A6235954

Q33398516-F72136F2-0A30-4214-8722-A09B78603814

P577

Q33398516-A19A6724-C325-4274-B73D-DB51A8962647

P5875

Q33398516-03872319-0B90-44C7-A49C-C68471B7C559

P698

Q33398516-3BCA662E-9496-4F21-A436-26E0CE1320AB

P932

Q33398516-933606B3-CA50-495D-95B7-0D70D411005A

P356

JOURNAL.PPAT.1000260

P1433

P1476

Control of stochastic gene expression by host factors at the HIV promoter.

@en

P2093

David V Schaffer

http://www.w3.org/2001/XMLSchema#string

Kathryn Miller-Jensen

http://www.w3.org/2001/XMLSchema#string

Priya S Shah

http://www.w3.org/2001/XMLSchema#string

P2860

Stochastic mRNA synthesis in mammalian cells

Recruitment of chromatin-modifying enzymes by CTIP2 promotes HIV-1 transcriptional silencing

The SWI/SNF chromatin-remodeling complex is a cofactor for Tat transactivation of the HIV promoter

The human factors YY1 and LSF repress the human immunodeficiency virus type 1 long terminal repeat via recruitment of histone deacetylase 1.

Counterregulation of chromatin deacetylation and histone deacetylase occupancy at the integrated promoter of human immunodeficiency virus type 1 (HIV-1) by the HIV-1 repressor YY1 and HIV-1 activator Tat

Stochastic kinetic analysis of developmental pathway bifurcation in phage lambda-infected Escherichia coli cells

The p65 (RelA) subunit of NF-kappaB interacts with the histone deacetylase (HDAC) corepressors HDAC1 and HDAC2 to negatively regulate gene expression

CREB-binding protein/p300 are transcriptional coactivators of p65

Regulation of HIV-1 gene expression by histone acetylation and factor recruitment at the LTR promoter

Repression of human immunodeficiency virus type 1 through the novel cooperation of human factors YY1 and LSF

P304

e1000260

http://www.w3.org/2001/XMLSchema#string

P31

scholarly article

P356

10.1371/JOURNAL.PPAT.1000260

http://www.w3.org/2001/XMLSchema#string

P433

1

http://www.w3.org/2001/XMLSchema#string

P478

5

http://www.w3.org/2001/XMLSchema#string

P50

John C. Burnett

P577

2009-01-09T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P5875

23766860

http://www.w3.org/2001/XMLSchema#string

P698

19132086

http://www.w3.org/2001/XMLSchema#string

P932

2607019

http://www.w3.org/2001/XMLSchema#string