Sialylation-independent mechanism involved in the amelioration of murine immune thrombocytopenia using intravenous gammaglobulin.
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Induction of Regulatory T Cells by Intravenous Immunoglobulin: A Bridge between Adaptive and Innate ImmunityRapid and reversible responses to IVIG in autoimmune neuromuscular diseases suggest mechanisms of action involving competition with functionally important autoantibodiesImmune recruitment or suppression by glycan engineering of endogenous and therapeutic antibodiesAnalysis and functional consequences of increased Fab-sialylation of intravenous immunoglobulin (IVIG) after lectin fractionationDeveloping the IVIG biomimetic, hexa-Fc, for drug and vaccine applicationsImmunoglobulin G dimers and immune complexes are dispensable for the therapeutic efficacy of intravenous immune globulin in murine immune thrombocytopenia.Cellular immune dysfunction in immune thrombocytopenia (ITP).Cytokine profiles in mouse models of experimental immune thrombocytopenia reveal a lack of inflammation and differences in response to intravenous immunoglobulin depending on the mouse strain.CD44 antibody-mediated amelioration of murine immune thrombocytopenia (ITP): mouse background determines the effect of FcγRIIb genetic disruption.Controlled tetra-Fc sialylation of IVIg results in a drug candidate with consistent enhanced anti-inflammatory activity.Contributions of T lymphocyte abnormalities to therapeutic outcomes in newly diagnosed patients with immune thrombocytopenia.IVIG regulates the survival of human but not mouse neutrophils.Optimal attenuation of experimental autoimmune encephalomyelitis by intravenous immunoglobulin requires an intact interleukin-11 receptor.Protection in antibody- and T cell-mediated autoimmune diseases by antiinflammatory IgG Fcs requires type II FcRs.Production of α2,6-sialylated IgG1 in CHO cellsEfficacy of Intravenous Immunoglobulin in Neurological Diseases.Sialylation converts arthritogenic IgG into inhibitors of collagen-induced arthritis.Identification of novel plasma glycosylation-associated markers of aging.Intravenous immunoglobulin-induced IL-33 is insufficient to mediate basophil expansion in autoimmune patientsThe carbohydrate switch between pathogenic and immunosuppressive antigen-specific antibodies.Mechanisms and potential therapeutic targets in allergic inflammation: recent insights.Review: intravenous immunoglobulin and B cells: when the product regulates the producer.Immunomodulation by IVIg and the Role of Fc-Gamma Receptors: Classic Mechanisms of Action after all?Differences in Anti-Inflammatory Actions of Intravenous Immunoglobulin between Mice and Men: More than Meets the Eye.IVIG-mediated effector functions in autoimmune and inflammatory diseases.The Emerging Importance of IgG Fab Glycosylation in Immunity.Clinical Use and Therapeutic Potential of IVIG/SCIG, Plasma-Derived IgA or IgM, and Other Alternative Immunoglobulin Preparations.Rapid and improved characterization of therapeutic antibodies and antibody related products using IdeS digestion and subunit analysis.Modulators of IgG penetration through the blood-brain barrier: Implications for Alzheimer's disease immunotherapy.Immunoglobulins: current understanding and future directions.Will sialylation change intravenous immunoglobulin therapy in the future?Glycosylation engineering of therapeutic IgG antibodies: challenges for the safety, functionality and efficacy.Therapeutic effect of IVIG on inflammatory arthritis in mice is dependent on the Fc portion and independent of sialylation or basophils.IgG subclass and vaccination stimulus determine changes in antigen specific antibody glycosylation in mice.Massive immune response against IVIg interferes with response against other antigens in mice: A new mode of action?Sialylation may be dispensable for reciprocal modulation of helper T cells by intravenous immunoglobulin.Effects of Acyclovir and IVIG on Behavioral Outcomes after HSV1 CNS Infection.Differential antibody glycosylation in autoimmunity: sweet biomarker or modulator of disease activity?Sweet SIGNs: IgG glycosylation leads the way in IVIG-mediated resolution of inflammation.rIgG1 Fc Hexamer Inhibits Antibody-Mediated Autoimmune Disease via Effects on Complement and FcγRs.
P2860
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P2860
Sialylation-independent mechanism involved in the amelioration of murine immune thrombocytopenia using intravenous gammaglobulin.
description
2012 nî lūn-bûn
@nan
2012 թուականի Յունուարին հրատարակուած գիտական յօդուած
@hyw
2012 թվականի հունվարին հրատարակված գիտական հոդված
@hy
2012年の論文
@ja
2012年論文
@yue
2012年論文
@zh-hant
2012年論文
@zh-hk
2012年論文
@zh-mo
2012年論文
@zh-tw
2012年论文
@wuu
name
Sialylation-independent mechan ...... ing intravenous gammaglobulin.
@ast
Sialylation-independent mechan ...... ing intravenous gammaglobulin.
@en
type
label
Sialylation-independent mechan ...... ing intravenous gammaglobulin.
@ast
Sialylation-independent mechan ...... ing intravenous gammaglobulin.
@en
prefLabel
Sialylation-independent mechan ...... ing intravenous gammaglobulin.
@ast
Sialylation-independent mechan ...... ing intravenous gammaglobulin.
@en
P2093
P2860
P1433
P1476
Sialylation-independent mechan ...... sing intravenous gammaglobulin
@en
P2093
Danila Leontyev
Donald R Branch
Sylvia Miescher
Xue-Zhong Ma
Yulia Katsman
P2860
P304
P356
10.1111/J.1537-2995.2011.03517.X
P50
P577
2012-01-18T00:00:00Z