Hypoxia response and VEGF-A expression in human proximal tubular epithelial cells in stable and progressive renal disease. - Wikidata - awgldk - TriplyDB

Hypoxia response and VEGF-A expression in human proximal tubular epithelial cells in stable and progressive renal disease.

Hypoxia response and VEGF-A expression in human proximal tubular epithelial cells in stable and progressive renal disease.

http://www.wikidata.org/entity/Q33399420

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Tipping the balance from angiogenesis to fibrosis in CKD Linking transcriptomic and proteomic data on the level of protein interaction networks.Integrative analysis of -omics data and histologic scoring in renal disease and transplantation: renal histogenomics.Renal tubular angiogenic dysregulation in anti-Thy1.1 glomerulonephritis.Acute kidney injury: a springboard for progression in chronic kidney disease Hypoxia inducible factor 1-alpha (HIF-1 alpha) is induced during reperfusion after renal ischemia and is critical for proximal tubule cell survival.From molecular signatures to predictive biomarkers: modeling disease pathophysiology and drug mechanism of action.Increased renal versican expression is associated with progression of chronic kidney disease.A systems view of genetics in chronic kidney disease A 3-biomarker-panel predicts renal outcome in patients with proteinuric renal diseases.Activation of hypoxia-inducible factors prevents diabetic nephropathy.Aristolochic acid I and ochratoxin A differentially regulate VEGF expression in porcine kidney epithelial cells--the involvement of SP-1 and HIFs transcription factors Renal oxidative stress, oxygenation, and hypertension Systems biology of kidney diseases.The unfolded protein response regulates an angiogenic response by the kidney epithelium during ischemic stress Reversal of renal dysfunction by targeted administration of VEGF into the stenotic kidney: a novel potential therapeutic approach Addition of endothelial progenitor cells to renal revascularization restores medullary tubular oxygen consumption in swine renal artery stenosis Physiological Capillary Regression is not Dependent on Reducing VEGF Expression.Vascular Endothelial Growth Factor and Podocyte Protection in Chronic Hypoxia: Effects of Endothelin-A Receptor Antagonism.Renal vascular structure and rarefaction.Capillary rarefaction, hypoxia, VEGF and angiogenesis in chronic renal disease.Renal tubulointerstitial hypoxia: cause and consequence of kidney dysfunction.Molecular phenotypes of kidney graft rejection.RNA expression signatures and posttranscriptional regulation in diabetic nephropathy.Expanding roles of the hypoxia-response network in chronic kidney disease.In vivo sodium tungstate treatment prevents E-cadherin loss induced by diabetic serum in HK-2 cell line.SOCS-3 is downregulated in progressive CKD patients and regulates proliferation in human renal proximal tubule cells in a STAT1/3 independent manner.Metabolomics-on-a-chip and metabolic flux analysis for label-free modeling of the internal metabolism of HepG2/C3A cells.Parathyroid hormone-related protein promotes epithelial-mesenchymal transition A molecular classifier for predicting future graft loss in late kidney transplant biopsies Preconditioned suppression of prolyl-hydroxylases attenuates renal injury but increases mortality in septic murine models.Integrative bioinformatics analysis of proteins associated with the cardiorenal syndrome Laser capture microdissection and multiplex-tandem PCR analysis of proximal tubular epithelial cell signaling in human kidney disease.EGF regulation of proximal tubule cell proliferation and VEGF-A secretion.Renovascular disease, microcirculation, and the progression of renal injury: role of angiogenesis.Characterization of connective tissue growth factor expression in primary cultures of human tubular epithelial cells: modulation by hypoxia.Gene polymorphism of vascular endothelial growth factor -1154 G>A is associated with hypertensive nephropathy in a Hispanic population.Mapping of molecular pathways, biomarkers and drug targets for diabetic nephropathy.Endostatin: a promising biomarker in the cardiovascular continuum?Endothelial Epas1 Deficiency Is Sufficient To Promote Parietal Epithelial Cell Activation and FSGS in Experimental Hypertension.

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P2860

Hypoxia response and VEGF-A expression in human proximal tubular epithelial cells in stable and progressive renal disease.

http://www.wikidata.org/entity/Q33399420

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Gene ontology: tool for the unification of biology

PANTHER version 6: protein sequence and function evolution data with expanded representation of biological pathways

A novel bHLH-PAS factor with close sequence similarity to hypoxia-inducible factor 1alpha regulates the VEGF expression and is potentially involved in lung and vascular development

Minimum information about a microarray experiment (MIAME)-toward standards for microarray data

A gene network for navigating the literature

Induction of the plasminogen activator inhibitor-1 gene expression by mild hypoxia via a hypoxia response element binding the hypoxia-inducible factor-1 in rat hepatocytes

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Gene expression profiles of human proximal tubular epithelial cells in proteinuric nephropathies.

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