Pain modulation by release of the endogenous cannabinoid anandamide. - Wikidata - awgldk - TriplyDB

Pain modulation by release of the endogenous cannabinoid anandamide.

Pain modulation by release of the endogenous cannabinoid anandamide.

http://www.wikidata.org/entity/Q33421146

about

Development of highly sensitive fluorescent assays for fatty acid amide hydrolase Supersensitivity to anandamide and enhanced endogenous cannabinoid signaling in mice lacking fatty acid amide hydrolase The endocannabinoid system as an emerging target of pharmacotherapy The elmiric acids: biologically active anandamide analogs Inactivation of N-acyl phosphatidylethanolamine phospholipase D reveals multiple mechanisms for the biosynthesis of endocannabinoids Treatment of trigeminal ganglion neurons in vitro with NGF, GDNF or BDNF: effects on neuronal survival, neurochemical properties and TRPV1-mediated neuropeptide secretion Endocannabinoids control platelet activation and limit aggregate formation under flow Discovery and molecular basis of potent noncovalent inhibitors of fatty acid amide hydrolase (FAAH)Interaction between Cannabinoid System and Toll-Like Receptors Controls Inflammation Medical cannabis - the Canadian perspective CB(1) receptor antagonist SR141716A increases capsaicin-evoked release of Substance P from the adult mouse spinal cord The endogenous cannabinoid agonist, anandamide stimulates sensory nerves in guinea-pig airways Genetic dissection of behavioural and autonomic effects of Delta(9)-tetrahydrocannabinol in mice.The endocannabinoid system in the rat dorsolateral periaqueductal grey mediates fear-conditioned analgesia and controls fear expression in the presence of nociceptive tone.Monoamines and neuropeptides interact to inhibit aversive behaviour in Caenorhabditis elegans.Cannabinoid receptor ligands: clinical and neuropharmacological considerations, relevant to future drug discovery and development.Potential anti-inflammatory actions of the elmiric (lipoamino) acids.Food for thought: endocannabinoid modulation of lipogenesis.Quantification of brain endocannabinoid levels: methods, interpretations and pitfalls How might cannabinoids influence sexual behavior?Fatty acid amide hydrolase and monoacylglycerol lipase inhibitors produce anti-allodynic effects in mice through distinct cannabinoid receptor mechanisms.Molecular basis for the perception of pain.Functional disassociation of the central and peripheral fatty acid amide signaling systems.Endocannabinoids and exercise.An endocannabinoid mechanism for stress-induced analgesia.A Cannabinoid CB1 Receptor-Positive Allosteric Modulator Reduces Neuropathic Pain in the Mouse with No Psychoactive Effects Role of endocannabinoids in alcohol consumption and intoxication: studies of mice lacking fatty acid amide hydrolase.The endocannabinoid system and pain Arthritis and pain. Future targets to control osteoarthritis pain Enhanced human tissue microdialysis using hydroxypropyl-ß-cyclodextrin as molecular carrier.Endocannabinoid modulation by FAAH and monoacylglycerol lipase within the analgesic circuitry of the periaqueductal grey Nitric oxide modulates the hyperalgesic response to mechanical noxious stimuli in sleep-deprived rats.Role of FAAH-like anandamide transporter in anandamide inactivation.The general anesthetic propofol increases brain N-arachidonylethanolamine (anandamide) content and inhibits fatty acid amide hydrolase Inhibition of fatty acid amide hydrolase unmasks CB1 receptor and TRPV1 channel-mediated modulation of glutamatergic synaptic transmission in midbrain periaqueductal grey.Effects of endocannabinoid system modulation on cognitive and emotional behavior Actions of the FAAH inhibitor URB597 in neuropathic and inflammatory chronic pain models.Cannabinoid receptors and endocannabinoids: evidence for new players.Site-specific increases in peripheral cannabinoid receptors and their endogenous ligands in a model of neuropathic pain.The role of cannabinoids in prostate cancer: Basic science perspective and potential clinical applications

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P2860

Pain modulation by release of the endogenous cannabinoid anandamide.

http://www.wikidata.org/entity/Q33421146

description

1999 nî lūn-bûn

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1999 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած

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1999 թվականի հոտեմբերին հրատարակված գիտական հոդված

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1999年の論文

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1999年論文

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1999年論文

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1999年論文

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1999年論文

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1999年論文

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1999年论文

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name

Pain modulation by release of the endogenous cannabinoid anandamide.

@ast

Pain modulation by release of the endogenous cannabinoid anandamide.

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type

label

Pain modulation by release of the endogenous cannabinoid anandamide.

@ast

Pain modulation by release of the endogenous cannabinoid anandamide.

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prefLabel

Pain modulation by release of the endogenous cannabinoid anandamide.

@ast

Pain modulation by release of the endogenous cannabinoid anandamide.

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PNAS.96.21.12198

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Proceedings of the National Academy of Sciences of the United States of America

P1476

Pain modulation by release of the endogenous cannabinoid anandamide.

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P2093

J M Walker

http://www.w3.org/2001/XMLSchema#string

K Tsou

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M C Sañudo-Peña

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N M Strangman

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S M Huang

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P2860

Enzymatic synthesis and degradation of anandamide, a cannabinoid receptor agonist

Isolation and structure of a brain constituent that binds to the cannabinoid receptor

Characterization and localization of cannabinoid receptors in rat brain: a quantitative in vitro autoradiographic study

Liquid chromatographic-atmospheric pressure chemical ionization mass spectrometric determination of anandamide and its analogs in rat brain and peripheral tissues.

Antagonism of stimulation-produced analgesia by naloxone, a narcotic antagonist.

Analgesia from electrical stimulation in the brainstem of the rat.

SR141716A, a potent and selective antagonist of the brain cannabinoid receptor.

Functional role of high-affinity anandamide transport, as revealed by selective inhibition.

The perceived effects of smoked cannabis on patients with multiple sclerosis.

Control of pain initiation by endogenous cannabinoids.

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