Characterization of the interferon-producing cell in mice infected with Listeria monocytogenes - Wikidata - awgldk - TriplyDB

Characterization of the interferon-producing cell in mice infected with Listeria monocytogenes

Characterization of the interferon-producing cell in mice infected with Listeria monocytogenes

http://www.wikidata.org/entity/Q33422966

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IFN-γ Priming Effects on the Maintenance of Effector Memory CD4(+) T Cells and on Phagocyte Function: Evidences from Infectious Diseases TLR7/9 versus TLR3/MDA5 signaling during virus infections and diabetes Splenic red pulp macrophages produce type I interferons as early sentinels of malaria infection but are dispensable for control Conditional Stat1 ablation reveals the importance of interferon signaling for immunity to Listeria monocytogenes infection The bacterial pathogen Listeria monocytogenes and the interferon family: type I, type II and type III interferons Bifidobacterium bifidum actively changes the gene expression profile induced by Lactobacillus acidophilus in murine dendritic cells.Type I IFN signaling in CD8- DCs impairs Th1-dependent malaria immunity.Listeria monocytogenes induces an interferon-enhanced activation of the integrated stress response that is detrimental for resolution of infection in mice A fluorescence reporter model defines "Tip-DCs" as the cellular source of interferon β in murine listeriosis Production of IFN-β during Listeria monocytogenes infection is restricted to monocyte/macrophage lineage Type I interferon production induced by Streptococcus pyogenes-derived nucleic acids is required for host protection The N-ethyl-N-nitrosourea-induced Goldenticket mouse mutant reveals an essential function of Sting in the in vivo interferon response to Listeria monocytogenes and cyclic dinucleotides.Both TLR2 and TRIF contribute to interferon-β production during Listeria infection Type I IFN induces IL-10 production in an IL-27-independent manner and blocks responsiveness to IFN-γ for production of IL-12 and bacterial killing in Mycobacterium tuberculosis-infected macrophages.Cell depletion in mice that express diphtheria toxin receptor under the control of SiglecH encompasses more than plasmacytoid dendritic cells Listeria monocytogenes induces IFNβ expression through an IFI16-, cGAS- and STING-dependent pathway.STING/MPYS mediates host defense against Listeria monocytogenes infection by regulating Ly6C(hi) monocyte migration.The timing of IFNβ production affects early innate responses to Listeria monocytogenes and determines the overall outcome of lethal infection.Defining the transcriptional and cellular landscape of type 1 diabetes in the NOD mouse Influence of internalin A murinisation on host resistance to orally acquired listeriosis in mice.Toll-like receptor 7-mediated type I interferon signaling prevents cholestasis- and hepatotoxin-induced liver fibrosis.Route of Infection Determines the Impact of Type I Interferons on Innate Immunity to Listeria monocytogenes The C5a anaphylatoxin receptor (C5aR1) protects against Listeria monocytogenes infection by inhibiting type 1 IFN expression LipA, a tyrosine and lipid phosphatase involved in the virulence of Listeria monocytogenes.Plasmacytoid dendritic cells mediate the regulation of inflammatory type T cell response for optimal immunity against respiratory Chlamydia pneumoniae infection.STING-dependent type I IFN production inhibits cell-mediated immunity to Listeria monocytogenes.MPYS is required for IFN response factor 3 activation and type I IFN production in the response of cultured phagocytes to bacterial second messengers cyclic-di-AMP and cyclic-di-GMP.Herpes virus entry mediator licenses Listeria infection induced immunopathology through control of type I interferon.Analysis of Transcriptional Signatures in Response to Listeria monocytogenes Infection Reveals Temporal Changes That Result from Type I Interferon Signaling.Unraveling the functions of plasmacytoid dendritic cells during viral infections, autoimmunity, and tolerance.Receptor-Interacting Protein Kinase-2 Inhibition by CYLD Impairs Antibacterial Immune Responses in Macrophages.Type I interferons: diversity of sources, production pathways and effects on immune responses.Response to interferons and antibacterial innate immunity in the absence of tyrosine-phosphorylated STAT1 I kappa B kinase alpha (IKKα) activity is required for functional maturation of dendritic cells and acquired immunity to infection.The Tumor Suppressor Hace1 Is a Critical Regulator of TNFR1-Mediated Cell Fate.TPL-2-ERK1/2 signaling promotes host resistance against intracellular bacterial infection by negative regulation of type I IFN production Confounding roles for type I interferons during bacterial and viral pathogenesis.Type I interferons regulate susceptibility to inflammation-induced preterm birth.Type I IFN exhaustion is a host defence protecting against secondary bacterial infections.Lactobacillus acidophilus induces virus immune defence genes in murine dendritic cells by a Toll-like receptor-2-dependent mechanism.

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Characterization of the interferon-producing cell in mice infected with Listeria monocytogenes

http://www.wikidata.org/entity/Q33422966

description

2009 nî lūn-bûn

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2009 թուականի Մարտին հրատարակուած գիտական յօդուած

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2009 թվականի մարտին հրատարակված գիտական հոդված

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2009年の論文

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2009年論文

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2009年論文

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2009年論文

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2009年論文

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2009年論文

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2009年论文

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Characterization of the interf ...... ed with Listeria monocytogenes

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Characterization of the interf ...... ed with Listeria monocytogenes

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Characterization of the interf ...... ed with Listeria monocytogenes

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Characterization of the interf ...... ed with Listeria monocytogenes

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JOURNAL.PPAT.1000355

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Characterization of the interf ...... ed with Listeria monocytogenes

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Andreas Pilz

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Benjamin Reutterer

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Claus Vogl

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Elisabeth Kernbauer

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Gabriele Stengl

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Renate Kastner

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Sandra Westermayer

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Tadatsugu Taniguchi

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Theresa Frenz

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Thomas Decker

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P2860

DAI (DLM-1/ZBP1) is a cytosolic DNA sensor and an activator of innate immune response

Toll-like receptor signalling

Evidence for licensing of IFN-gamma-induced IFN regulatory factor 1 transcription factor by MyD88 in Toll-like receptor-dependent gene induction program

Key role of splenic myeloid DCs in the IFN-alphabeta response to adenoviruses in vivo

IRFs: master regulators of signalling by Toll-like receptors and cytosolic pattern-recognition receptors

Interferons, interferon-like cytokines, and their receptors

Integral role of IRF-5 in the gene induction programme activated by Toll-like receptors

The interferon regulatory factor, IRF5, is a central mediator of toll-like receptor 7 signaling

IRF-7 is the master regulator of type-I interferon-dependent immune responses

Stats: transcriptional control and biological impact

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e1000355

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10.1371/JOURNAL.PPAT.1000355

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3

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5

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Mathias Müller

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2009-03-27T00:00:00Z

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24238474

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19325882

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Listeria monocytogenes

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2654726

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