Isolation of a cellular factor that can reactivate latent HIV-1 without T cell activation.
about
CpG methylation controls reactivation of HIV from latency.Transcriptional target-based expression cloning of immunoregulatory moleculesMolecular mechanisms of HIV-1 persistence in the monocyte-macrophage lineageQuantitative phosphoproteomics reveals extensive cellular reprogramming during HIV-1 entry.Establishment of HIV-1 latency in resting CD4+ T cells depends on chemokine-induced changes in the actin cytoskeleton.Calcium/calcineurin synergizes with prostratin to promote NF-κB dependent activation of latent HIVAn enhanced chimeric firefly luciferase-inspired enzyme for ATP detection and bioluminescence reporter and imaging applications.Human immunodeficiency virus (HIV) latency: the major hurdle in HIV eradicationNF-κB/Rel: agonist and antagonist roles in HIV-1 latency.HIV reservoirs and latency models.HIV-1 transcription and latency: an update.Deployment of the human immunodeficiency virus type 1 protein arsenal: combating the host to enhance viral transcription and providing targets for therapeutic developmentHIV-1 regulation of latency in the monocyte-macrophage lineage and in CD4+ T lymphocytes.Molecular control of HIV-1 postintegration latency: implications for the development of new therapeutic strategies.Studies of HIV-1 latency in an ex vivo model that uses primary central memory T cells.HIV reservoirs and the possibility of a cure for HIV infection.Antibody-based candidate therapeutics against HIV-1: implications for virus eradication and vaccine design.The two sides of Tat.Current views on HIV-1 latency, persistence, and cure.Molecular Understanding of HIV-1 Latency.Heat Shock Protein 90 Facilitates Latent HIV Reactivation through Maintaining the Function of Positive Transcriptional Elongation Factor b (p-TEFb) under Proteasome Inhibition.MARCH8 inhibits HIV-1 infection by reducing virion incorporation of envelope glycoproteins.Replication-competent noninduced proviruses in the latent reservoir increase barrier to HIV-1 cure.Cyclin T1 and CDK9 T-loop phosphorylation are downregulated during establishment of HIV-1 latency in primary resting memory CD4+ T cells.HIV-1 and the macrophage
P2860
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P2860
Isolation of a cellular factor that can reactivate latent HIV-1 without T cell activation.
description
2009 nî lūn-bûn
@nan
2009 թուականի Մարտին հրատարակուած գիտական յօդուած
@hyw
2009 թվականի մարտին հրատարակված գիտական հոդված
@hy
2009年の論文
@ja
2009年論文
@yue
2009年論文
@zh-hant
2009年論文
@zh-hk
2009年論文
@zh-mo
2009年論文
@zh-tw
2009年论文
@wuu
name
Isolation of a cellular factor ...... V-1 without T cell activation.
@ast
Isolation of a cellular factor ...... V-1 without T cell activation.
@en
type
label
Isolation of a cellular factor ...... V-1 without T cell activation.
@ast
Isolation of a cellular factor ...... V-1 without T cell activation.
@en
prefLabel
Isolation of a cellular factor ...... V-1 without T cell activation.
@ast
Isolation of a cellular factor ...... V-1 without T cell activation.
@en
P2860
P50
P356
P1476
Isolation of a cellular factor ...... V-1 without T cell activation.
@en
P2093
P2860
P304
P356
10.1073/PNAS.0809536106
P407
P577
2009-03-31T00:00:00Z