Phagocytosis of Staphylococcus aureus by macrophages exerts cytoprotective effects manifested by the upregulation of antiapoptotic factors.
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Intracellular persisting Staphylococcus aureus is the major pathogen in recurrent tonsillitisAutosomal dominant and sporadic monocytopenia with susceptibility to mycobacteria, fungi, papillomaviruses, and myelodysplasiaNatural mutations in a Staphylococcus aureus virulence regulator attenuate cytotoxicity but permit bacteremia and abscess formationStaphylococcal PknB as the first prokaryotic representative of the proline-directed kinases.Subinhibitory concentrations of thymol reduce enterotoxins A and B and alpha-hemolysin production in Staphylococcus aureus isolates.Citrullination alters immunomodulatory function of LL-37 essential for prevention of endotoxin-induced sepsis.A cardinal role for cathepsin d in co-ordinating the host-mediated apoptosis of macrophages and killing of pneumococci.Innate host defense requires TFEB-mediated transcription of cytoprotective and antimicrobial genesTranslocation of Porphyromonas gingivalis gingipain adhesin peptide A44 to host mitochondria prevents apoptosis.Animal and human antibodies to distinct Staphylococcus aureus antigens mutually neutralize opsonic killing and protection in mice.Proteolytic inactivation of LL-37 by karilysin, a novel virulence mechanism of Tannerella forsythiaEnterococcus faecalis infection activates phosphatidylinositol 3-kinase signaling to block apoptotic cell death in macrophages.The KdpD/KdpE two-component system: integrating K⁺ homeostasis and virulence.The early phase transcriptome of bovine monocyte-derived macrophages infected with Staphylococcus aureus in vitro.The Janus face of α-toxin: a potent mediator of cytoprotection in staphylococci-infected macrophagesCelecoxib sensitizes Staphylococcus aureus to antibiotics in macrophages by modulating SIRT1.NsaRS is a cell-envelope-stress-sensing two-component system of Staphylococcus aureus.Structural mechanism of sensing long dsRNA via a noncatalytic domain in human oligoadenylate synthetase 3Nanoparticulate drug delivery platforms for advancing bone infection therapies.Rapid neutrophil destruction following phagocytosis of Staphylococcus aureusStaphylococcus aureus Strain USA300 Perturbs Acquisition of Lysosomal Enzymes and Requires Phagosomal Acidification for Survival inside Macrophages.Role of JAK-STAT signaling in maturation of phagosomes containing Staphylococcus aureus.Identification of an intracellular M17 family leucine aminopeptidase that is required for virulence in Staphylococcus aureus.Francisella tularensis alters human neutrophil gene expression: insights into the molecular basis of delayed neutrophil apoptosis.Identification and treatment of the Staphylococcus aureus reservoir in vivoThe impact of α-toxin on host cell plasma membrane permeability and cytokine expression during human blood infection by CA-MRSA USA300Omics Approaches for the Study of Adaptive Immunity to Staphylococcus aureus and the Selection of Vaccine Candidates.Structural and functional role of Staphylococcus aureus surface components recognizing adhesive matrix molecules of the host.Staphylococcus aureus adaptation to the host and persistence: role of loss of capsular polysaccharide expression.Intracellular Staphylococcus aureus: live-in and let die.Alveolar macrophages in pulmonary host defence the unrecognized role of apoptosis as a mechanism of intracellular bacterial killing.The role of programmed death ligand 1 pathway in persistent biomaterial-associated infections.Staphylococcal Esx proteins modulate apoptosis and release of intracellular Staphylococcus aureus during infection in epithelial cells.Simultaneous bactericidal and osteogenic effect of nanoparticulate calcium phosphate powders loaded with clindamycin on osteoblasts infected with Staphylococcus aureus.Thymus vulgaris L. and thymol assist murine macrophages (RAW 264.7) in the control of in vitro infections by Staphylococcus aureus, Pseudomonas aeruginosa, and Candida albicans.Apoptosis, Toll-like, RIG-I-like and NOD-like Receptors Are Pathways Jointly Induced by Diverse Respiratory Bacterial and Viral Pathogens.Riboflavin along with antibiotics balances reactive oxygen species and inflammatory cytokines and controls Staphylococcus aureus infection by boosting murine macrophage function and regulates inflammation.MCPIP-1, Alias Regnase-1, Controls Epithelial Inflammation by Posttranscriptional Regulation of IL-8 Production.Targeting Multidrug-resistant Staphylococci with an anti-rpoA Peptide Nucleic Acid Conjugated to the HIV-1 TAT Cell Penetrating Peptide.Influence of Sae-regulated and Agr-regulated factors on the escape of Staphylococcus aureus from human macrophages.
P2860
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P2860
Phagocytosis of Staphylococcus aureus by macrophages exerts cytoprotective effects manifested by the upregulation of antiapoptotic factors.
description
2009 nî lūn-bûn
@nan
2009 թուականի Ապրիլին հրատարակուած գիտական յօդուած
@hyw
2009 թվականի ապրիլին հրատարակված գիտական հոդված
@hy
2009年の論文
@ja
2009年論文
@yue
2009年論文
@zh-hant
2009年論文
@zh-hk
2009年論文
@zh-mo
2009年論文
@zh-tw
2009年论文
@wuu
name
Phagocytosis of Staphylococcus ...... tion of antiapoptotic factors.
@ast
Phagocytosis of Staphylococcus ...... tion of antiapoptotic factors.
@en
type
label
Phagocytosis of Staphylococcus ...... tion of antiapoptotic factors.
@ast
Phagocytosis of Staphylococcus ...... tion of antiapoptotic factors.
@en
prefLabel
Phagocytosis of Staphylococcus ...... tion of antiapoptotic factors.
@ast
Phagocytosis of Staphylococcus ...... tion of antiapoptotic factors.
@en
P2093
P2860
P50
P1433
P1476
Phagocytosis of Staphylococcus ...... tion of antiapoptotic factors.
@en
P2093
Adeline R Whitney
Daniel E Sturdevant
Joanna Koziel
Tomasz Mikolajczyk
P2860
P356
10.1371/JOURNAL.PONE.0005210
P407
P577
2009-04-21T00:00:00Z