Microglia processes block the spread of damage in the brain and require functional chloride channels.
about
Receptors, ion channels, and signaling mechanisms underlying microglial dynamicsFunction of microglia and macrophages in secondary damage after spinal cord injuryAltered morphological dynamics of activated microglia after induction of status epilepticus.LRRK2 G2019S mutation attenuates microglial motility by inhibiting focal adhesion kinase.Prevention of LPS-induced microglia activation, cytokine production and sickness behavior with TLR4 receptor interfering peptidesContributions of Mouse and Human Hematopoietic Cells to Remodeling of the Adult Auditory Nerve After Neuron Loss.Integrin-Kindlin3 requirements for microglial motility in vivo are distinct from those for macrophages.Adenosine A2A receptor antagonism reverses inflammation-induced impairment of microglial process extension in a model of Parkinson's diseaseCapillary blood flow around microglial somata determines dynamics of microglial processes in ischemic conditions.Neuronal hyperactivity recruits microglial processes via neuronal NMDA receptors and microglial P2Y12 receptors after status epilepticus.Volume-regulated anion channel--a frenemy within the brain.DIDS (4,4-diisothiocyanatostilbenedisulphonic acid) induces apoptotic cell death in a hippocampal neuronal cell line and is not neuroprotective against ischemic stress.Toll-like receptor 2-mediated alternative activation of microglia is protective after spinal cord injury.Macrophages in spinal cord injury: phenotypic and functional change from exposure to myelin debris.Chloride Cotransporters as a Molecular Mechanism underlying Spreading Depolarization-Induced Dendritic BeadingTime lapse in vivo microscopy reveals distinct dynamics of microglia-tumor environment interactions-a new role for the tumor perivascular space as highway for trafficking microglia.The adenosine generating enzymes CD39/CD73 control microglial processes ramification in the mouse brain.Interferon regulatory factor 8/interferon consensus sequence binding protein is a critical transcription factor for the physiological phenotype of microglia.Microglia dynamics and function in the CNS.Chondroitin sulfate proteoglycans potently inhibit invasion and serve as a central organizer of the brain tumor microenvironment.LRRK2 Inhibits FAK Activity by Promoting FERM-mediated Autoinhibition of FAK and Recruiting the Tyrosine Phosphatase, SHP-2In vivo direct reprogramming of reactive glial cells into functional neurons after brain injury and in an Alzheimer's disease model.Glial dysfunction in the pathogenesis of α-synucleinopathies: emerging concepts.Dynamic motility of microglia: purinergic modulation of microglial movement in the normal and pathological brain.Purinergic receptors in microglia: functional modal shifts of microglia mediated by P2 and P1 receptors.Big effects from tiny vessels: imaging the impact of microvascular clots and hemorrhages on the brain.Microglial dynamics and role in the healthy and diseased brain: a paradigm of functional plasticityRethinking regenerative medicine: a macrophage-centered approach.Central nervous system regenerative failure: role of oligodendrocytes, astrocytes, and microglia.Deletion of the Fractalkine Receptor, CX3CR1, Improves Endogenous Repair, Axon Sprouting, and Synaptogenesis after Spinal Cord Injury in Mice.Cerebellar compartmentation of prion pathogenesis.Macrophage polarization: a key event in the secondary phase of acute spinal cord injury.Tamoxifen induces apoptosis of mouse microglia cell line BV-2 cells via both mitochondrial and death receptor pathways.Decreased connexin 43 in astrocytes inhibits the neuroinflammatory reaction in an acute mouse model of neonatal sepsis.Novel Influences of IL-10 on CNS Inflammation Revealed by Integrated Analyses of Cytokine Networks and Microglial Morphology.HDAC3 inhibition ameliorates spinal cord injury by immunomodulation.Neuroadhesive L1 coating attenuates acute microglial attachment to neural electrodes as revealed by live two-photon microscopy.A novel Drosophila injury model reveals severed axons are cleared through a Draper/MMP-1 signaling cascade.In Vivo Imaging of Microglial Calcium Signaling in Brain Inflammation and Injury.TSPO regulation in reactive gliotic diseases.
P2860
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P2860
Microglia processes block the spread of damage in the brain and require functional chloride channels.
description
2009 nî lūn-bûn
@nan
2009 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած
@hyw
2009 թվականի նոյեմբերին հրատարակված գիտական հոդված
@hy
2009年の論文
@ja
2009年論文
@yue
2009年論文
@zh-hant
2009年論文
@zh-hk
2009年論文
@zh-mo
2009年論文
@zh-tw
2009年论文
@wuu
name
Microglia processes block the ...... functional chloride channels.
@ast
Microglia processes block the ...... functional chloride channels.
@en
type
label
Microglia processes block the ...... functional chloride channels.
@ast
Microglia processes block the ...... functional chloride channels.
@en
prefLabel
Microglia processes block the ...... functional chloride channels.
@ast
Microglia processes block the ...... functional chloride channels.
@en
P356
P1433
P1476
Microglia processes block the ...... functional chloride channels.
@en
P2093
Dustin J Hines
Sean J Mulligan
P2860
P304
P356
10.1002/GLIA.20874
P577
2009-11-01T00:00:00Z