VEGFR1 activity modulates myeloid cell infiltration in growing lung metastases but is not required for spontaneous metastasis formation.
about
Macrophage diversity enhances tumor progression and metastasisC-X-C receptor type 4 promotes metastasis by activating p38 mitogen-activated protein kinase in myeloid differentiation antigen (Gr-1)-positive cellsEndothelial focal adhesion kinase mediates cancer cell homing to discrete regions of the lungs via E-selectin up-regulation.Direct evidence for lineage-dependent effects of bone marrow stromal cells on tumor progressionPowerful anti-tumor and anti-angiogenic activity of a new anti-vascular endothelial growth factor receptor 1 peptide in colorectal cancer modelsThe pro-metastatic role of bone marrow-derived cells: a focus on MSCs and regulatory T cellsAntiangiogenic therapy: impact on invasion, disease progression, and metastasis.FLT1 signaling in metastasis-associated macrophages activates an inflammatory signature that promotes breast cancer metastasis.TIMP-2 targets tumor-associated myeloid suppressor cells with effects in cancer immune dysfunction and angiogenesisPlGF/VEGFR-1 Signaling Promotes Macrophage Polarization and Accelerated Tumor Progression in Obesity.miR-155-deficient bone marrow promotes tumor metastasis.Cancer and the metastatic substrate.Different but synergistic effects of bone marrow-derived VEGFR2+ and VEGFR2-CD45+ cells during hepatocellular carcinoma progression.Premetastatic lung "niche": is vascular endothelial growth factor receptor 1 activation required?Inhibition of placenta growth factor with TB-403: a novel antiangiogenic cancer therapy.PlGF: a multitasking cytokine with disease-restricted activity.Placental growth factor in cancer.Glioblastoma resistance to anti-VEGF therapy is associated with myeloid cell infiltration, stem cell accumulation, and a mesenchymal phenotype.VEGFR1-activity-independent metastasis formation.Myeloid cells: Prime drivers of tumor progression.Primary tumor regulates the pulmonary microenvironment in melanoma carcinoma model and facilitates lung metastasis.
P2860
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P2860
VEGFR1 activity modulates myeloid cell infiltration in growing lung metastases but is not required for spontaneous metastasis formation.
description
2009 nî lūn-bûn
@nan
2009 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած
@hyw
2009 թվականի սեպտեմբերին հրատարակված գիտական հոդված
@hy
2009年の論文
@ja
2009年論文
@yue
2009年論文
@zh-hant
2009年論文
@zh-hk
2009年論文
@zh-mo
2009年論文
@zh-tw
2009年论文
@wuu
name
VEGFR1 activity modulates myel ...... ntaneous metastasis formation.
@ast
VEGFR1 activity modulates myel ...... ntaneous metastasis formation.
@en
type
label
VEGFR1 activity modulates myel ...... ntaneous metastasis formation.
@ast
VEGFR1 activity modulates myel ...... ntaneous metastasis formation.
@en
prefLabel
VEGFR1 activity modulates myel ...... ntaneous metastasis formation.
@ast
VEGFR1 activity modulates myel ...... ntaneous metastasis formation.
@en
P2093
P2860
P1433
P1476
VEGFR1 activity modulates myel ...... ntaneous metastasis formation.
@en
P2093
Dai Fukumura
Dan G Duda
Rakesh K Jain
Sung-Suk Chae
P2860
P356
10.1371/JOURNAL.PONE.0006525
P407
P577
2009-09-18T00:00:00Z