Monocyte and neutrophil recruitment during oral Salmonella infection is driven by MyD88-derived chemokines. - Wikidata - awgldk - TriplyDB

Monocyte and neutrophil recruitment during oral Salmonella infection is driven by MyD88-derived chemokines.

Monocyte and neutrophil recruitment during oral Salmonella infection is driven by MyD88-derived chemokines.

http://www.wikidata.org/entity/Q33511278

about

Innate immune signaling in defense against intestinal microbes Protective host immune responses to Salmonella infection Immunity to intestinal pathogens: lessons learned from Salmonella Development of protective immunity to Salmonella, a mucosal pathogen with a systemic agenda Intestinal inflammation responds to microbial tissue load independent of pathogen/non-pathogen discrimination.The Gut Microbiota Reduces Colonization of the Mesenteric Lymph Nodes and IL-12-Independent IFN-γ Production During Salmonella Infection Phosphatidylinositol 3-kinase-γ signaling promotes Campylobacter jejuni-induced colitis through neutrophil recruitment in mice.Yersinia enterocolitica YopH-Deficient Strain Activates Neutrophil Recruitment to Peyer's Patches and Promotes Clearance of the Virulent Strain.Novel regulatory functions for Toll-like receptor-activated B cells during intracellular bacterial infection.Innate immune control of Salmonella enterica serovar Typhimurium: mechanisms contributing to combating systemic Salmonella infection.Early MyD88-dependent induction of interleukin-17A expression during Salmonella colitis.Salmonella typhimurium-induced IL-1 release from primary human monocytes requires NLRP3 and can occur in the absence of pyroptosis.Synergy between CD40 and MyD88 Does Not Influence Host Survival to Salmonella Infection Salmonella inhibits monocyte differentiation into CD11c hi MHC-II hi cells in a MyD88-dependent fashion.Comparative study of the protective capacity against Salmonella infection between probiotic and nonprobiotic Lactobacilli.Differential intestinal M-cell gene expression response to gut commensals.Schizandrin B protects LPS-induced sepsis via TLR4/NF-κB/MyD88 signaling pathway.

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P2860

Monocyte and neutrophil recruitment during oral Salmonella infection is driven by MyD88-derived chemokines.

http://www.wikidata.org/entity/Q33511278

description

2009 nî lūn-bûn

@nan

2009 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած

@hyw

2009 թվականի նոյեմբերին հրատարակված գիտական հոդված

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2009年の論文

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2009年論文

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2009年論文

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2009年論文

@zh-hk

2009年論文

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2009年論文

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2009年论文

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name

Monocyte and neutrophil recrui ...... n by MyD88-derived chemokines.

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Monocyte and neutrophil recrui ...... n by MyD88-derived chemokines.

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Monocyte and neutrophil recrui ...... n by MyD88-derived chemokines.

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type

label

Monocyte and neutrophil recrui ...... n by MyD88-derived chemokines.

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Monocyte and neutrophil recrui ...... n by MyD88-derived chemokines.

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Monocyte and neutrophil recrui ...... n by MyD88-derived chemokines.

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prefLabel

Monocyte and neutrophil recrui ...... n by MyD88-derived chemokines.

@ast

Monocyte and neutrophil recrui ...... n by MyD88-derived chemokines.

@en

Monocyte and neutrophil recrui ...... n by MyD88-derived chemokines.

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European Journal of Immunology

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Monocyte and neutrophil recrui ...... n by MyD88-derived chemokines.

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P2093

Anna Rydström

http://www.w3.org/2001/XMLSchema#string

Mary Jo Wick

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P2860

CCR2 mediates homeostatic and inflammatory release of Gr1(high) monocytes from the bone marrow, but is dispensable for bladder infiltration in bacterial urinary tract infection

The P2X7 receptor: a key player in IL-1 processing and release.

Additive roles for MCP-1 and MCP-3 in CCR2-mediated recruitment of inflammatory monocytes during Listeria monocytogenes infection.

TLR adaptor MyD88 is essential for pathogen control during oral toxoplasma gondii infection but not adaptive immunity induced by a vaccine strain of the parasite.

Polymorphonuclear neutrophils improve replication of Chlamydia pneumoniae in vivo upon MyD88-dependent attraction.

CCR2-dependent trafficking of F4/80dim macrophages and CD11cdim/intermediate dendritic cells is crucial for T cell recruitment to lungs infected with Mycobacterium tuberculosis.

Toll-like receptor 4 dependence of innate and adaptive immunity to Salmonella: importance of the Kupffer cell network.

Toll-like receptors are temporally involved in host defense.

CXCR2 deficiency confers impaired neutrophil recruitment and increased susceptibility during Toxoplasma gondii infection.

Monocyte recruitment, activation, and function in the gut-associated lymphoid tissue during oral Salmonella infection.

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3019-3030

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scholarly article

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10.1002/EJI.200939483

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11

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39

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2009-11-01T00:00:00Z

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38020147

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19839009

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