Tropomyosin3 overexpression and a potential link to epithelial-mesenchymal transition in human hepatocellular carcinoma. - Wikidata - awgldk - TriplyDB

Tropomyosin3 overexpression and a potential link to epithelial-mesenchymal transition in human hepatocellular carcinoma.

Tropomyosin3 overexpression and a potential link to epithelial-mesenchymal transition in human hepatocellular carcinoma.

http://www.wikidata.org/entity/Q33548174

about

Silencing of KIF14 interferes with cell cycle progression and cytokinesis by blocking the p27(Kip1) ubiquitination pathway in hepatocellular carcinoma Identification and characterization of tropomyosin 3 associated with granulin-epithelin precursor in human hepatocellular carcinoma Targeted proteomics of isolated glomeruli from the kidneys of diabetic rats: sorbin and SH3 domain containing 2 is a novel protein associated with diabetic nephropathy.Proteomic analysis of the hepatic tissue of Long-Evans Cinnamon (LEC) rats according to the natural course of Wilson disease.Proteomic analysis indicates the importance of TPM3 in esophageal squamous cell carcinoma invasion and metastasis Stage-specific analysis of plasma protein profiles in ovarian cancer: Difference in-gel electrophoresis analysis of pooled clinical samples.MTBP suppresses cell migration and filopodia formation by inhibiting ACTN4 Comparative analysis of Notch1 and Notch2 binding sites in the genome of BxPC3 pancreatic cancer cells.MDM2 binding protein, a novel metastasis suppressor.Ras Transformation Overrides a Proliferation Defect Induced by Tpm3.1 Knockout.MicroRNA-191, by promoting the EMT and increasing CSC-like properties, is involved in neoplastic and metastatic properties of transformed human bronchial epithelial cells.TPM3, a strong prognosis predictor, is involved in malignant progression through MMP family members and EMT-like activators in gliomas.Inhibitions of epithelial to mesenchymal transition and cancer stem cells-like properties are involved in miR-148a-mediated anti-metastasis of hepatocellular carcinoma.Silencing of hHS6ST2 inhibits progression of pancreatic cancer through inhibition of Notch signalling.Label-free quantitative mass spectrometry reveals a panel of differentially expressed proteins in colorectal cancer.MicroRNA-491 is involved in metastasis of hepatocellular carcinoma by inhibitions of matrix metalloproteinase and epithelial to mesenchymal transition.

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Tropomyosin3 overexpression and a potential link to epithelial-mesenchymal transition in human hepatocellular carcinoma.

http://www.wikidata.org/entity/Q33548174

description

2010 nî lūn-bûn

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2010 թուականի Ապրիլին հրատարակուած գիտական յօդուած

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2010 թվականի ապրիլին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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name

Tropomyosin3 overexpression an ...... uman hepatocellular carcinoma.

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Tropomyosin3 overexpression an ...... uman hepatocellular carcinoma.

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Tropomyosin3 overexpression an ...... uman hepatocellular carcinoma.

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Hae-Jin Hu

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Hai-Dong Xu

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Hye-Sun Choi

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Jong Young Choi

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Seon-Hee Yim

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Seung-Hun Shin

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Seung-Hyun Jung

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Yeun-Jun Chung

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P2860

Snail and SIP1 increase cancer invasion by upregulating MMP family in hepatocellular carcinoma cells

TPM3-ALK and TPM4-ALK oncogenes in inflammatory myofibroblastic tumors

Analysis of Relative Gene Expression Data Using Real-Time Quantitative PCR and the 2−ΔΔCT Method

Global cancer statistics, 2002

A sequence analysis of the genomic regions involved in the rearrangements between TPM3 and NTRK1 genes producing TRK oncogenes in papillary thyroid carcinomas

Hepatocellular carcinoma: epidemiology and molecular carcinogenesis

Epithelial-mesenchymal transitions in development and pathologies

Molecular requirements for epithelial-mesenchymal transition during tumor progression

Clinical implication of recurrent copy number alterations in hepatocellular carcinoma and putative oncogenes in recurrent gains on 1q.

Decreased expression of a single tropomyosin isoform, TM5/TM30nm, results in reduction in motility of highly metastatic B16-F10 mouse melanoma cells.

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