Altered expression of glutamate signaling, growth factor, and glia genes in the locus coeruleus of patients with major depression. - Wikidata - awgldk - TriplyDB

Altered expression of glutamate signaling, growth factor, and glia genes in the locus coeruleus of patients with major depression.

Altered expression of glutamate signaling, growth factor, and glia genes in the locus coeruleus of patients with major depression.

http://www.wikidata.org/entity/Q33553152

about

Towards a glutamate hypothesis of depression: an emerging frontier of neuropsychopharmacology for mood disorders Comorbidity Factors and Brain Mechanisms Linking Chronic Stress and Systemic Illness Functional alterations of astrocytes in mental disorders: pharmacological significance as a drug target Astrocyte pathology in major depressive disorder: insights from human postmortem brain tissue Glial abnormalities in substance use disorders and depression: does shared glutamatergic dysfunction contribute to comorbidity?The stressed synapse: the impact of stress and glucocorticoids on glutamate transmission A brief history of the development of antidepressant drugs: from monoamines to glutamate Astrocyte heterogeneity in the brain: from development to disease Altered functional protein networks in the prefrontal cortex and amygdala of victims of suicide Fluoxetine affects GluK2 editing, glutamate-evoked Ca(2+) influx and extracellular signal-regulated kinase phosphorylation in mouse astrocytes.Convergent functional genomic studies of ω-3 fatty acids in stress reactivity, bipolar disorder and alcoholism.Glutamate and its receptors in the pathophysiology and treatment of major depressive disorder Inflammation Effects on Brain Glutamate in Depression: Mechanistic Considerations and Treatment Implications.NMDA receptor hypofunction induces dysfunctions of energy metabolism and semaphorin signaling in rats: a synaptic proteome study.Reduced connexin 43 immunolabeling in the orbitofrontal cortex in alcohol dependence and depression.Altered miRNA expression network in locus coeruleus of depressed suicide subjects Region-specific in situ hybridization-guided laser-capture microdissection on postmortem human brain tissue coupled with gene expression quantification.Construction and analysis of the protein-protein interaction networks for schizophrenia, bipolar disorder, and major depression Astroglial connexin 43 sustains glutamatergic synaptic efficacy.Altered expression of synapse and glutamate related genes in post-mortem hippocampus of depressed subjects Epigenetic mechanisms of depression and antidepressant action Metabotropic glutamate receptor mGluR2/3 and mGluR5 binding in the anterior cingulate cortex in psychotic and nonpsychotic depression, bipolar disorder and schizophrenia: implications for novel mGluR-based therapeutics.The molecular bases of the suicidal brain.Evidence for transcriptional factor dysregulation in the dorsal raphe nucleus of patients with major depressive disorder.Suicide and suicidal behaviour Linking molecules to mood: new insight into the biology of depression.Amitriptyline up-regulates connexin43-gap junction in rat cultured cortical astrocytes via activation of the p38 and c-Fos/AP-1 signalling pathway Gene expression deficits in pontine locus coeruleus astrocytes in men with major depressive disorder Systematic review of genome-wide gene expression studies of bipolar disorder Association of rare variation in the glutamate receptor gene SLC1A2 with susceptibility to bipolar disorder and schizophrenia.Altered choroid plexus gene expression in major depressive disorder Inflammatory Factors Mediate Vulnerability to a Social Stress-Induced Depressive-like Phenotype in Passive Coping Rats.Expression profiling in neuropsychiatric disorders: emphasis on glutamate receptors in bipolar disorder Evidence for alterations of the glial syncytial function in major depressive disorder.Developmental underpinnings of differences in rodent novelty-seeking and emotional reactivity.An investigation of amino-acid neurotransmitters as potential predictors of clinical improvement to ketamine in depression Fibroblast growth factor 9 is a novel modulator of negative affect.Astrogliopathology in neurological, neurodevelopmental and psychiatric disorders.Synaptic potentiation is critical for rapid antidepressant response to ketamine in treatment-resistant major depression The fibroblast growth factor family: neuromodulation of affective behavior.

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Altered expression of glutamate signaling, growth factor, and glia genes in the locus coeruleus of patients with major depression.

http://www.wikidata.org/entity/Q33553152

description

2010 nî lūn-bûn

@nan

2010 թուականի Ապրիլին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի ապրիլին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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name

Altered expression of glutamat ...... atients with major depression.

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Altered expression of glutamat ...... atients with major depression.

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Altered expression of glutamat ...... atients with major depression.

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Altered expression of glutamat ...... atients with major depression.

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Altered expression of glutamat ...... atients with major depression.

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Altered expression of glutamat ...... atients with major depression.

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Altered expression of glutamat ...... atients with major depression.

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Molecular Psychiatry

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P2860

Lifetime Prevalence and Age-of-Onset Distributions of DSM-IV Disorders in the National Comorbidity Survey Replication

New roles for astrocytes: gap junction hemichannels have something to communicate

Connexin30 in rodent, cat and human brain: selective expression in gray matter astrocytes, co-localization with connexin43 at gap junctions and late developmental appearance

Epilepsy and exacerbation of brain injury in mice lacking the glutamate transporter GLT-1

Essential role of brain-derived neurotrophic factor in adult hippocampal function.

Acute and longer-term outcomes in depressed outpatients requiring one or several treatment steps: a STAR*D report

Knockout of glutamate transporters reveals a major role for astroglial transport in excitotoxicity and clearance of glutamate

Impairments of neuroplasticity and cellular resilience in severe mood disorders: implications for the development of novel therapeutics.

Clinical studies implementing glutamate neurotransmission in mood disorders.

The implications of using an inappropriate reference gene for real-time reverse transcription PCR data normalization.

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