Learning from lesions: patterns of tissue inflammation in leprosy. - Wikidata - awgldk - TriplyDB

Learning from lesions: patterns of tissue inflammation in leprosy.

Learning from lesions: patterns of tissue inflammation in leprosy.

http://www.wikidata.org/entity/Q33554190

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The continuing challenges of leprosy Leprosy: review of the epidemiological, clinical, and etiopathogenic aspects - part 1 High-resolution sonography: a new technique to detect nerve damage in leprosy A major T cell antigen of Mycobacterium leprae is a 10-kD heat-shock cognate protein.Potential plasma markers of Type 1 and Type 2 leprosy reactions: a preliminary report Immunological significance of Mycobacterium leprae cell walls.High matrix metalloproteinase production correlates with immune activation and leukocyte migration in leprosy reactional lesions.Changes in expression of signal transduction proteins in T lymphocytes of patients with leprosy.Characterization of Haemophilus ducreyi-specific T-cell lines from lesions of experimentally infected human subjects Immunoregulation in onchocerciasis. Functional and phenotypic abnormalities of lymphocyte subsets and changes with therapy Immunostimulatory activity of major membrane protein II from Mycobacterium tuberculosis Target organ localization of memory CD4(+) T cells in patients with chronic beryllium disease.CD4+ Th17 cells discriminate clinical types and constitute a third subset of non Th1, Non Th2 T cells in human leprosy Reduced suppressor cell response to Mycobacterium leprae in lepromatous leprosy Fibronectin-binding antigen 85 and the 10-kilodalton GroES-related heat shock protein are the predominant TH-1 response inducers in leprosy contacts.T cells in the lesion of experimental autoimmune encephalomyelitis. Enrichment for reactivities to myelin basic protein and to heat shock proteins.Interleukin-17: the missing link between T-cell accumulation and effector cell actions in rheumatoid arthritis?Divergent T-cell cytokine patterns in inflammatory arthritis Longitudinal immune profiles in type 1 leprosy reactions in Bangladesh, Brazil, Ethiopia and Nepal.Role of the HLA-DP Glu 69 and the TNF-alpha TNF-alpha 2 gene markers in susceptibility to beryllium hypersensitivity.Antigen-specific cellular and humoral responses are induced by intradermal Mycobacterium leprae infection of the mouse ear.Analysis of naturally occurring delayed-type hypersensitivity reactions in leprosy by in situ hybridization.Differences in host susceptibility to disease progression in the human challenge model of Haemophilus ducreyi infection.Multiple mechanisms support oligoclonal T cell expansion in rheumatoid synovitis.T-cell-mediated cytotoxicity against Mycobacterium antigen-pulsed autologous macrophages in leprosy patients.Effect of adoptive transfer of cloned Actinobacillus actinomycetemcomitans-specific T helper cells on periodontal disease.LILRA2 activation inhibits dendritic cell differentiation and antigen presentation to T cells.Conserved mycobacterial lipoglycoproteins activate TLR2 but also require glycosylation for MHC class II-restricted T cell activation.Antigens of Corynebacterium pseudotuberculosis and prospects for vaccine development.Serial measurement of the circulating levels of tumour necrosis factor and its soluble receptors 1 and 2 for monitoring leprosy patients during multidrug treatment.Challenges in immunodiagnostic tests for leprosy.Understanding the type 1 reactional state for early diagnosis and treatment: a way to avoid disability in leprosy.New strategies for leprosy and tuberculosis and for development of bacillus Calmette-Guérin into a multivaccine vehicle.Leprosy as a model to understand cancer immunosurveillance and T cell anergy.The mycobacterial secreted antigen 85 complex possesses epitopes that are differentially expressed in human leprosy lesions and Mycobacterium leprae-infected armadillo tissues.Do leprosy and tuberculosis generate a systemic inflammatory shift? Setting the ground for a new dialogue between experimental immunology and bioarchaeology.Characterization of the culture filtrate-specific cytotoxic T lymphocyte response induced by Bacillus Calmette-Guérin vaccination in H-2b mice.ML1419c peptide immunization induces Mycobacterium leprae-specific HLA-A*0201-restricted CTL in vivo with potential to kill live mycobacteria.Detection of transforming growth factor-beta 1 in dermal lesions of different clinical forms of leprosy.Leprosy and human immunodeficiency virus infection. A closer look at the lesions.

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P2860

Learning from lesions: patterns of tissue inflammation in leprosy.

http://www.wikidata.org/entity/Q33554190

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1988 nî lūn-bûn

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1988 թուականի Փետրուարին հրատարակուած գիտական յօդուած

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1988 թվականի փետրվարին հրատարակված գիտական հոդված

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1988年の論文

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1988年論文

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1988年論文

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1988年論文

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1988年論文

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1988年論文

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1988年论文

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Learning from lesions: patterns of tissue inflammation in leprosy.

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Learning from lesions: patterns of tissue inflammation in leprosy.

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Learning from lesions: patterns of tissue inflammation in leprosy.

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Learning from lesions: patterns of tissue inflammation in leprosy.

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Learning from lesions: patterns of tissue inflammation in leprosy.

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Learning from lesions: patterns of tissue inflammation in leprosy.

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Proceedings of the National Academy of Sciences of the United States of America

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Learning from lesions: patterns of tissue inflammation in leprosy.

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B R Bloom

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C Pirmez

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H Kino

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J Convit

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J Melancon-Kaplan

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P2860

Classification of leprosy according to immunity. A five-group system.

Limiting dilution assays for the determination of immunocompetent cell frequencies. I. Data analysis.

Homing receptors and the control of lymphocyte migration.

Quantification of antigen-reactive cells among human T lymphocytes.

Monoclonal antibody 9.3 and anti-CD11 antibodies define reciprocal subsets of lymphocytes.

Phenotypic identification of suppressor-effector, suppressor-amplifier and suppressor-inducer T cells of B cell differentiation in man.

Cloned suppressor T cells from a lepromatous leprosy patient suppress Mycobacterium leprae reactive helper T cells.

Immunotherapy with a mixture of Mycobacterium leprae and BCG in different forms of leprosy and in Mitsuda-negative contacts.

Genetically restricted suppressor T-cell clones derived from lepromatous leprosy lesions

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