Mycobacterium leprae actively modulates the cytokine response in naive human monocytes. - Wikidata - awgldk - TriplyDB

Mycobacterium leprae actively modulates the cytokine response in naive human monocytes.

Mycobacterium leprae actively modulates the cytokine response in naive human monocytes.

http://www.wikidata.org/entity/Q33557846

about

NFκB activation in cutaneous lesions of leprosy is associated with development of multibacillary infection PARK2 mediates interleukin 6 and monocyte chemoattractant protein 1 production by human macrophages.Gene expression profiling specifies chemokine, mitochondrial and lipid metabolism signatures in leprosy Host targeted activity of pyrazinamide in Mycobacterium tuberculosis infection Mycobacterium leprae-host-cell interactions and genetic determinants in leprosy: an overview.MicroRNA-21 targets the vitamin D-dependent antimicrobial pathway in leprosy Genetic Variation in Toll-Interacting Protein Is Associated With Leprosy Susceptibility and Cutaneous Expression of Interleukin 1 Receptor Antagonist.Mycobacterium leprae alters classical activation of human monocytes in vitro.Mycobacterium leprae-induced Insulin-like Growth Factor I attenuates antimicrobial mechanisms, promoting bacterial survival in macrophages.Virulent Type A Francisella tularensis actively suppresses cytokine responses in human monocytes Mycobacterial phenolic glycolipids with a simplified lipid aglycone modulate cytokine levels through Toll-like receptor 2.Modulation of the cytokine response in human monocytes by mycobacterium leprae phenolic glycolipid-1.Assessment of serum level of interleukin-1b and interleukin-12 in leprosy: impact of previous Bacillus Calmitte Guerin vaccination.Nutrigenomics and inflammatory bowel diseases.Leprosy As a Complex Infection: Breakdown of the Th1 and Th2 Immune Paradigm in the Immunopathogenesis of the Disease.Immune Checkpoints in Leprosy: Immunotherapy As a Feasible Approach to Control Disease Progression.Serum Th1/Th2 and macrophage lineage cytokines in leprosy; correlation with circulating CD4(+) CD25(high) FoxP3(+) T-regs cells.Type I Interferons, Autophagy and Host Metabolism in Leprosy.CD163 favors Mycobacterium leprae survival and persistence by promoting anti-inflammatory pathways in lepromatous macrophages.

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P2860

Mycobacterium leprae actively modulates the cytokine response in naive human monocytes.

http://www.wikidata.org/entity/Q33557846

description

2009 nî lūn-bûn

@nan

2009 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած

@hyw

2009 թվականի հոտեմբերին հրատարակված գիտական հոդված

@hy

2009年の論文

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2009年学术文章

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2009年学术文章

@zh-cn

2009年学术文章

@zh-hans

2009年学术文章

@zh-my

2009年学术文章

@zh-sg

2009年學術文章

@yue

name

Mycobacterium leprae actively modulates the cytokine response in naive human monocytes.

@ast

Mycobacterium leprae actively modulates the cytokine response in naive human monocytes.

@en

Mycobacterium leprae actively modulates the cytokine response in naive human monocytes.

@nl

type

label

Mycobacterium leprae actively modulates the cytokine response in naive human monocytes.

@ast

Mycobacterium leprae actively modulates the cytokine response in naive human monocytes.

@en

Mycobacterium leprae actively modulates the cytokine response in naive human monocytes.

@nl

prefLabel

Mycobacterium leprae actively modulates the cytokine response in naive human monocytes.

@ast

Mycobacterium leprae actively modulates the cytokine response in naive human monocytes.

@en

Mycobacterium leprae actively modulates the cytokine response in naive human monocytes.

@nl

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Infection and Immunity

P1476

Mycobacterium leprae actively modulates the cytokine response in naive human monocytes.

@en

P2093

Blas Peixoto

http://www.w3.org/2001/XMLSchema#string

Claudia Manca

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Daniel Sinsimer

http://www.w3.org/2001/XMLSchema#string

Dorothy Fallows

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James Krahenbuhl

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P2860

Interleukin-1 receptor antagonist activity of a human interleukin-1 inhibitor

The continuing challenges of leprosy

Human TLR1 deficiency is associated with impaired mycobacterial signaling and protection from leprosy reversal reaction.

LIRs/ILTs/MIRs, inhibitory and stimulatory Ig-superfamily receptors expressed in myeloid and lymphoid cells.

A functional promoter polymorphism in monocyte chemoattractant protein-1 is associated with increased susceptibility to pulmonary tuberculosis.

Th1-Th2 paradigm: insights from leprosy.

Mycobacterium leprae infection in monocyte-derived dendritic cells and its influence on antigen-presenting function.

Classification of leprosy according to immunity. A five-group system.

Polymorphisms in Toll-like receptor 4 (TLR4) are associated with protection against leprosy

Toll-like receptor 2 (TLR2) polymorphisms are associated with reversal reaction in leprosy

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293-300

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10.1128/IAI.00816-09

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1

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78

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19841079

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Mycobacterium leprae

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2798203

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