Induction of the CLOCK gene by E2-ERα signaling promotes the proliferation of breast cancer cells. - Wikidata - awgldk - TriplyDB

Induction of the CLOCK gene by E2-ERα signaling promotes the proliferation of breast cancer cells.

Induction of the CLOCK gene by E2-ERα signaling promotes the proliferation of breast cancer cells.

http://www.wikidata.org/entity/Q33559921

about

Circadian disruption and breast cancer: an epigenetic link?DACH1 inhibits SNAI1-mediated epithelial-mesenchymal transition and represses breast carcinoma metastasis.Circadian Clock, Cell Division, and Cancer: From Molecules to Organism.FOXK2 transcription factor suppresses ERα-positive breast cancer cell growth through down-regulating the stability of ERα via mechanism involving BRCA1/BARD1.Circadian Rhythms and Breast Cancer: The Role of Per2 in Doxorubicin-Induced Cell Death DEC1 regulates breast cancer cell proliferation by stabilizing cyclin E protein and delays the progression of cell cycle S phase.Circadian clocks, epigenetics, and cancer.Core Circadian Clock Genes Regulate Leukemia Stem Cells in AML Sumoylation of TCF21 downregulates the transcriptional activity of estrogen receptor-alpha SUMOylation of PES1 upregulates its stability and function via inhibiting its ubiquitination.Circadian systems biology in Metazoa.The Pathophysiologic Role of Disrupted Circadian and Neuroendocrine Rhythms in Breast Carcinogenesis.The Circadian NAD+ Metabolism: Impact on Chromatin Remodeling and Aging.SYNJ2BP promotes the degradation of PTEN through the lysosome-pathway and enhances breast tumor metastasis via PI3K/AKT/SNAI1 signaling.Clock genes associate with white matter integrity in depressed bipolar patients.Circadian clock and oral cancer.The basic helix-loop-helix transcription factor SHARP1 is an oncogenic driver in MLL-AF6 acute myelogenous leukemia.Altered Circadian Rhythms and Breast Cancer: From the Human to the Molecular Level Checkpoint suppressor 1 suppresses transcriptional activity of ERα and breast cancer cell proliferation via deacetylase SIRT1.The in vitro estrogenic activity of the crude drugs found in Japanese herbal medicines prescribed for menopausal syndrome was enhanced by combining them.Altered circadian genes expression in breast cancer tissue according to the clinical characteristics.

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Induction of the CLOCK gene by E2-ERα signaling promotes the proliferation of breast cancer cells.

http://www.wikidata.org/entity/Q33559921

description

2014 nî lūn-bûn

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2014 թուականի Մայիսին հրատարակուած գիտական յօդուած

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2014 թվականի մայիսին հրատարակված գիտական հոդված

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2014年の論文

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2014年論文

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2014年論文

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2014年論文

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Induction of the CLOCK gene by ...... ration of breast cancer cells.

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Induction of the CLOCK gene by ...... ration of breast cancer cells.

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Alan K Chang

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Hailian Bi

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Huijian Wu

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Liyun Xiao

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Miao Wang

http://www.w3.org/2001/XMLSchema#string

Ming-Xi Zang

http://www.w3.org/2001/XMLSchema#string

Shujing Li

http://www.w3.org/2001/XMLSchema#string

Xinrong Xing

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P2860

The molecular mechanism governing the oncogenic potential of SOX2 in breast cancer

PTEN suppresses the oncogenic function of AIB1 through decreasing its protein stability via mechanism involving Fbw7 alpha

Characterization of the core mammalian clock component, NPAS2, as a REV-ERBalpha/RORalpha target gene

Direct regulation of CLOCK expression by REV-ERB.

Increased breast cancer risk among women who work predominantly at night

Rotating night shifts and risk of breast cancer in women participating in the nurses' health study

Coordinated transcription of key pathways in the mouse by the circadian clock

GAPDH as a housekeeping gene: analysis of GAPDH mRNA expression in a panel of 72 human tissues

Interacting molecular loops in the mammalian circadian clock

Estrogen directly modulates circadian rhythms of PER2 expression in the uterus.

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